Child Psychiatry Flashcards

1
Q

General considerations in assessment of children

A

Internalising (psychological state) vs Externalising (to outwards env)

  • need various collateral info to get full picture – parents, teachers, child
  • interviewing parents also allows indirect evaluation of their personalities, parenting etc.

Multi-dimensional assessment

  • psychiatric diagnosis – COMORBIDITIES ARE COMMON
  • developmental delays
  • intellectual level
  • medical conditions
  • psychosocial situation
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2
Q

Risk factors and protective factors for child psychiatric problems

A

Interaction of genes and environment

Risks:
Predisposing 
- genetic
- perinatal e.g. substance/alcohol in pregnancy, extreme low weight/prematurity (ADHD, ASD risk), birth complications, physical disabilities 
- insecure attachment 
- temperament 
- chronic illness, CNS trauma

Precipitating/Perpetuating

  • parental mental illness
  • family: parenting problem, conflicts, violence, abuse
  • school: academic failure, bullying, rejection
  • society: poverty, discrimination, isolation

Protective factors:

  • good self esteem, happy temperament
  • good problem solving and social skills
  • appropriately involved and consistent parenting
  • harmonious family, able to resolve conflicts
  • sense of achievement and belonging
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3
Q

General mx in child psychiatry

A
Multidisciplinary 
Physical, psychological, social
Collaboration and liaison with community partners
- family, school
- share understanding and difficulties 
- skills in handling child
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4
Q

Specific developmental disorders - definition, intelligence

A

Disturbed acquisition of specific cognitive or motor function during child’s development e.g. language, reading, spelling, calculation, motor skills

Other areas of cognition are normal
NORMAL INTELLIGENCE

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5
Q

Pervasive developmental disorders - features, disorders included

A

Severe impairments in SOCIAL INTERACTION AND COMMUNICATION SKILLS
RESTRICTED, STEREOTYPED INTERESTS and BEHAVIOUR

Pervades all areas of functioning and usually evident in first few years of life
Diagnosis NOT BASED ON INTELLECTUAL FUNCTIONING

Autism Spectrum Disorder
- includes: autism, Asperger’s, Rett’s, Childhood disintegrative disorder

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6
Q

Autism Spectrum Disorder - epidemiology, M:F, aetiology, syndromal vs non-syndromal, similarities with schiz

A

1-2% of children
M>F 3:1

Aetiology: exact cause not identified - genetic (56-95% heritability), prenatal, peri-natal, immunological factors implicated

Can be non-syndromal or syndromal e.g. tuberous sclerosis, Rett’s, Di George, fragile X

Social withdrawal, communication impairment and poor eye contact similar to negative symptoms of schizophrenia

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7
Q

ASD - main symptoms

A

IMPAIRMENT IN SOCIAL INTERACTION and COMMUNICATION SKILLS

  • verbal: speech delay, difficulty in initiating or sustaining convo
  • non-verbal: poor eye contact, restricted facial expressions, poor use of gestures
  • weak social awareness, reciprocity (e.g. less response to name calling), empathy, emotional recognition (can’t adjust well)
  • failure to develop and share enjoyment of peer relationships (always alone)

RESTRICTED, STEREOTYPED INTERESTS and BEHAVIOURS

  • insistent on sameness, inflexible adherence to routines and rituals
  • unusual intense preoccupations with interests such as dates, numbers, timetables; hard/moving objects e.g. bus, spinning wheels (selective attention)
  • repetitive stereotyped (no function) movements e.g. clapping, rocking, flicking
  • sensory problems – hyper or hypo: may be sensory seeking e.g. massage, rubbing, hitting themselves
  • lack of imaginative play
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8
Q

ASD - onset, intelligence, comorbidities, other behavioural problems

A

Early onset but usually present later

Intelligence: most have normal IQ

High rates of psychiatric comorbidity - MUST SCREEN!
- ADHD (30-40%), anxiety, depression (esp when older), ODD, conduct problems

May exhibit behavioural problems e.g. aggression, impulsivitym, self-harm – RISK

Physical comorbidities
- GI, immunity, 25-30% develop epilepsy

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9
Q

ASD management - initial assessment, main form of treatment, indications for medication

A

Usually child assessment service paediatrician screens development and physical syndromes if parents suspect problem –> then refer to PSY

Psychoeducation
ONGOING SUPPORT
ONGOING TRAINING (OT and CP)
- parents involved (parenting skills, stress)
- social skills (how to handle certain situations)
- rigid behaviour modification
Emotional regulatory skills
Refer Ed Psy for Educational needs and support
Refer PT for gross motor development
Refer ST for speech therapy

Pharmacological treatment only if:

  • comorbid MDD, GAD, ADHD etc
  • high self harm risk – may need SGA to decrease irritability or aggression
  • SSRI may have repetitive behaviours
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10
Q

ASD prognosis, good prognostic factors

A

Generally poor – only 1-2% achieve full independence but 50% have sufficient social skills

Good prognostic factor
- IQ>70, good language development, home environment supportive

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11
Q

Asperger’s syndrome - M:F, main features, IQ, onset age, comparison with autism

A

Outdated, now part of ASD
M>F

Same core features of ASD but NO IMPAIRMENT in LANGUAGE ACQUISITION AND ABILITY (still have impairment in reciprocal social interaction) or in cognitive development and intelligence

IQ and language may be superior in some cases (high functioning)

Onset usually >5 (may be later in teenage yrs when more complex interactions develop - due to emotional problem, temper outburst, self harm)
Mild social deficits, active but odd interactions (whereas autism is passive), pedantic speech, more sophisticated interests, high verbal IQ

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12
Q

Rett’s syndrome - cause, onset, features

A

Mutation in gene MECP2 on X chromosome
Apparently normal development in first 5 months after birth

6 months -2 yrs –> PROGRESSIVE AND DESTRUCTIVE ENCEPHALOPATHY

  • deceleration of head growth
  • loss of language development
  • loss of purposeful hand movements and fine motor skills with subsequent development of stereotyped hand movements

Prognosis: wheelchair bound, incontinence, muscle wasting, rigidity, no language (after 10 yrs)

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13
Q

Childhood disintegrative disorder - main feature

A

2 years of normal development followed by LOSS OF PREVIOUSLY ACQUIRED SKILLS
- language, social, adaptive, play, bowel/bladder control, motor skills before age 10

A/w autism like impairment of social interaction, repetitive behaviour

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14
Q

Acquired disorders with onset usually in childhood or adolescence

A
ADHD
Conduct disorder, ODD
Separation Anxiety 
Phobic Anxiety
Social Anxiety
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15
Q

Attention Deficit Hyperactivity Disorder - M:F, prevalence, risk factors, age of onset

A

5-8% school-aged children
M>F 3:1

Aetiology/ Risk factors:

  • highly heritable (80%; underdevelopment of frontal lobe with NE/DA dysfunction)
  • environment: fetal alcohol syndrome, maternal smoking, lead poisoning, birth complications, hypoxia, brain injury

Onset: toddler years but presentation at school age
- hx of kindergarten complaints

Things to ask in Hx include daily routine, behaviours in different settings etc

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16
Q

ADHD main features

A

IMPAIRED ATTENTION

  • difficulty sustaining attention in tasks
  • not listening when being spoken to, highly distractible
  • reluctance to engage in activities that require sustained mental effort
  • forgetful, lose things regularly

HYPERACTIVITY

  • restlessness, running around and jumping in inappropriate situations
  • incessant fidgeting
  • excessive talkativeness or noisiness
  • difficulty engaging in quiet activities

IMPULSIVITY

  • difficult awaiting turn, interrupting others’ conversations or games
  • prematurely blurting out answers to questions
  • short tempered

All symptoms are DISPROPORTIONATE TO AGE (clinical diagnosis)
Can be combined type, IA type or HI type

17
Q

ADHD comorbidities

A

Dysfunction to family, education, social, self-esteem hence HIGHLY COMORBID!!

Dyslexia, motor clumsiness, sleep problem, behaviour problem (school refusal, drop out, change jobs frequently), relationship problems
ODD (30-40%), anxiety, mood disorder, conduct disorder, substance abuse, teenage pregnancy
ASD, tics

18
Q

ADHD management - initial assessment, mild-moderate tx, severe tx

A

Thorough assessment of

  • comorbidities (and treat)
  • family relationship and parenting
  • psychosocial stresses
  • functional impairment

For mild to moderate cases: 1st line is PSYCHOSOCIAL

  • parental education or training
  • CBT
  • CP for social skills training, behavioural strategies for impulsivity and functional impairments e.g. organisation
  • educational needs and support (EP)
  • self-esteem building

For severe cases: pharmacological (should be >5 yrs old)

  • Methylphenidate (stimulant) –> S/E: CVS problems (monitor bp, HR, ECG), suppress appetite and growth, insomnia, epilepsy (but no sig long term effects); avoid during asthmatic attack
  • Atomoxetine (non-stimulant)
  • Other options if ineffective: bupropion, clonidine, modafinil, amphetamine based, imipramine
19
Q

ADHD prognosis - response to treatment, future problems, poor prognostic factors

A

60-70% response to decrease symptoms and adverse comorbidities

  • hyperactivity improves
  • inattention and impulsivity may continue
  • doesn’t change trajectory of illness

1/3 outgrow it, 2/3 need lifelong meds

Risk of developing conduct problem, antisocial PD, interpersonal/occupational/forsenic problems

Poor prognostic factors: unstable family dynamics, coexisting conduct disorder

20
Q

Conduct disorder and ODD – prevalence, age of onset, aetiology, features, management

A

1-10%
M>F 4:1
Onset before 18

Aetiology: genetic, parental problems, child abuse/neglect, educational impairment, poor SE status

Repetitive persistent pattern of:

  • aggression to people and animals
  • destruction of property
  • deceitfulness or theft
  • major violations of societal expectations or rules

Oppositional defiant disorder = persistent negativistic defiant and hostile behaviour in the ABSENCE OF BEHAVIOUR THAT VIOLATES THE LAW
e.g. angry, resentful, deliberately defy rules or requests, annoy others

Management: CBT, family therapy

21
Q

Anxiety disorders in childhood/pre-adolescents

A

Separation anxiety

  • normal is 6 months-2/4 years
  • developmentally inappropriate and excessive anxiety when separation from home – life threatening feeling that harm will befall on attachment figure (repeated phoning, can’t sleep, refuse school)

Phobic anxiety

  • minor phobic symptoms common in childhood and varies through development
  • Dx when AGE INAPPROPRIATE or clinically abnormal levels

Social anxiety

  • normal stranger anxiety is 8 months-1 year
  • persistent or recurrent fear/avoidance of strangers with abnormal anxiety
22
Q

Management of depression in adolescence

A

Symptoms may be misinterpreted

Mx:

  • SSRI (fluoxetine, sertraline, citalopram) ONLY WHEN CLEARLY INDICATED – AVOID IF <12 yrs old
  • -> monitor S/E, dosage, suicidal risk when start med
  • PSYCHOLOGICAL THERAPY
    • mild: 2 wks FU, self-help + short CBT
    • mod-severe: CBT, IPT, family therapy for 12 wks

Social: counselling, play therapy (for younger kids)

23
Q

Selective mutism - must r/o, features, onset, may develop ? later

A

MUST R/O AUTISM

Child is mute in many social situations but can speak freely to familiar people (need evidence)
- adequately developed language skills

Onset <5, F>M

Often a/w anxiety symptoms, may develop social phobia later

24
Q

Tic disorders - characteristics, types of tics

A

Sudden repetitive non-rhythmic motor movements or vocalisations
INVOLUNTARY
Often prominent during stress

Simple motor tics: eye blinking, neck jerking, facial grimacing
Simple vocal tics: grunting, coughing, barking
Complex motor tics: jumping, touching self, copropraxia
Complex vocal tics: repeating words, coprolalia

25
Q

Gilles de la Tourette’s syndrome - defintion, associated disorders, aetiology/neurotransmitter dysfunction, management

A

Presence of both multiple motor tics (face, neck, LIMBS) and >1 vocal tics for >1 YEAR
- symptoms come and go

Motor tics usually present by 7

A/w OCD, ADHD

Aetiology: social, env, genetic – disturbance in CSTC circuit, GABAergic impairment, overactive DA system

Management:

  • treat OCD, ADHD
  • CBT and other psychosocial for anxiety and stress
  • DOPAMINE ANTAGONISTS – haloperidol, sulpiride, CLONIDINE FIRST LINE (alpha-2 agonist, less S/E)