Chest Pain And Acute Coronary Syndrome Flashcards

1
Q

What are the four main systems that could cause chest pain?

A

Cardiac (circulatory)
Respiratory
GI (digestive)
Musculoskeletal

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2
Q

How does pleural/pericardial chest pain compare to cardiac ischaemic chest pain?

A

Pain from the pericardial sac is “somatic”/ specific -it is sharp, well localised and is often worse with inspiration, coughing or positional changes

Pain from the heart itself is “visceral”/ non-specific it is dull and poorly localised and worsens with exertion

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3
Q

What are some differentials for non-cardiac chest pain?

A

Pneumonia, pleurisy, PE
Reflux, peptic ulcer disease
Costochondritis, rib fracture
Aortic dissection

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4
Q

What is stable angina?

A

Angina that occurs as a result of heart tissue ischaemia ONLY when metabolic demands of cardiac muscle are greater than what can be delivered via the coronary arteries (on exertion)

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5
Q

What is the typical patient history of someone with stable angina?

A

Dull, reterosternal chest pain
Triggered by exertion and goes away completely at rest
GTN spray will relieve the pain
Won’t be sweaty/pale

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6
Q

Name all of the acute coronary syndromes

A

Unstable angina
Myocardial infarction
Non-STelevation MI
STelevation MI (STEMI)

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7
Q

What are acute coronary syndromes?

A

Acute myocardial ischaemia caused by atherosclerotic coronary artery disease (atherosclerosis, thrombus formation, occlusion)

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8
Q

Would we see raised troponin levels in a patient with unstable angina? Why?

A

No, the heart tissue is ischaemic, not dead and therefore there is no cardiac enzyme leak

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9
Q

Would we expect to see raised troponin in NSTEMI/STEMI?

Why?

A

Yes

Heart tissue death (infarction) has occurred and therefore cardiac enzymes can leak from the necrosed cardiac myocytes

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10
Q

What is the typical patient history of a patient with unstable angina?
What is there a risk of if patients present with these features?

A
Dull, reterosternal chest pain 
That occurs at REST
GTN spray doesn't work at relieving pain 
May last longer than unstable angina 
RISK OF STEMI/NSTEMI need to be admitted
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11
Q

What is the typical patient history of a patient with MI ?

A
Dull, reterosternal cheat pain 
Much more severe than angina pain 
Radiates to neck/shoulders/lt arm
Pain occurs at rest 
Sweating, pale, nauseous
Nothing helps ease pain 
Pain persists >15 min
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12
Q

What diagnostic tests might be used in suspected acute coronary syndrome?

A

ECG - ST segments, T waves +/- pathological Q waves

Blood tests - troponin

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13
Q

True or false, it is always possible to spot acute coronary syndromes on clinical examination

A

FALSE! Often clinical examination is normal- nothing abnormal to hear with stethoscope

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14
Q

How would the pattern STEMI show on an ECG?

A

ST segment elevation

Hyperacute T waves (big and pointy)

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15
Q

What ECG changes might you see in a patient with unstable angina/ NSTEMI?
How would you distinguish between the two?

A

ST segment depression
T wave flattening or inversion

Test troponin levels

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16
Q

STEMI is usually due to what?

A

Acute, total occlusion of a major coronary artery

17
Q

NSTEMI is usually due to what?

A

Acutely progressive tight stenosis of a coronary artery

18
Q

What are the most common processes that can lead to STEMI/NSTEMI?

A

Atherosclerosis
Coronary spasm
Coronary embolism
Coronary dissection

19
Q

How does a Type I MI differ from a Type II MI?

A

Type I: spontaneous MI (plaque rupture, thrombus, distal platelet emboli)

Type II: MI secondary to ischaemia imbalance (resp.failure, anaemia, tachycardic/bradycardia)