Chapter 7 Hypokinetic Dysarthria Flashcards

1
Q

Hypokinetic Dysarthria is a related to problems with the

A

Basal ganglia control (extrapyramidal problems)

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2
Q

Hypokinetic Dysarthria is most often associated with which degenerative disease?

A

Parkinson’s

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3
Q

Hypokinetic Dysarthria reflects characteristics due to

A
  • rigidity
  • reduced range of motion
  • reduced force of movement
  • may be slow movements
  • may be quick speech movements
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4
Q

Function of the basal ganglia

A
  • facilitates movement
  • regulates muscle tone
  • regulates movements that support goal-directed movements
  • controls postural adjustments in skilled movements
  • Inhibits unnecessary movements
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5
Q

Damage to the basal ganglia control circuit can result in two types of problems:

A

Reduced movement

reduced inhibition of involuntary movements

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6
Q

Neurotransmitter associated with basal ganglia functioning

A

dopamine produced in the substantia nigra then sent to the striatum (caudate nucleus & putamen)

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7
Q

Dopamine

A

is a inhibitory mechanism,

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8
Q

% of Parkinson’s Patients with HD

A

50%

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9
Q

Classic Non Speech signs of PD

A
  • Tremor at rest
  • Pill rolling (finger mvmt)
  • Rigidity (cogwheel)
  • Bradykinesia (false/frozen mvmt)
  • Hypokinesia (reduced mvmt)
  • Posture problems
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10
Q

Non speech Hypokinesia Characteristics

A
  • masked expressions of the face with unblinking eyes
  • reduced arm swing
  • micrographia
  • festination (slow walk fast shuffle)
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11
Q

Hypokinetic Dysarthria Etiologies

A

Anything that can cause damage to the basal ganglia

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12
Q

Degenerative Etiologies

A

Parkinson’s
Alzheimer’s
Pick’s

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13
Q

Vascular Etiologies

A
  • Multiple strokes affecting basal ganglia (vascular parkinsonism)
  • Cerebral hypoxia (lack of O2)
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14
Q

Toxic-Metabolic Etiologies

A

Antipsychotic drugs that block dopamine receptors

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15
Q

Trauma Etiologies

A

TBI/ repeated head trauma to the substantia nigra

stereotactic ventrolateral thalamotomy

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16
Q

Infectious Etiologies

A

Viral Encephalitis (MMRs)

17
Q

Patient Complaints

A
  • Weak/quiet voice
  • Speech rate is too fast
  • words are imprecise and flat emotion tone
  • difficulty starting speech
  • may stutter/repeat
  • fatigue has negative effect on speech
  • swallowing and drooling problems
  • stiff upper lip due to rigidity
18
Q

Non Speech Clinical Findings

A
  • Mask like face, unsmiling, unblinking
  • Reduced mvmt of chest and abdomen while breathing
  • drooling due to saliva accumulation from not swallowing
  • head may not accompany eye movement
  • tremor of jaw/lips at rest and while potruded
  • Flat affect inconsistent with patient’s emotional state
19
Q

Speech Clinical Findings

A
  • monopitch/monoloudness
  • reduced loudness
  • reduced stress
  • short rushes of speech
  • overall increased rate of speech
  • harsh breathy voice quality
  • imprecise consonants
  • difficulty initiating speech
  • rapid blurred AMRs
  • may have tremor on vowel prolongation
  • palilalia
20
Q

Best distinguishing speech characteristics

A
Fast rate
short rushes of speech
reduced stress
monotone/monoloudness
inappropriate silences
breathiness