Chapter 6 Flashcards

1
Q

What are the Preoccupation and Obsession Disorders?

A

->Somatic Symptom and Related Disorders

->Previously called Somatoform Disorders (DSM-IV-TR)

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2
Q

What’s the difference Between DSM-IV-TR & DSM-5 TR definition of somatic symptom disorder?

A

->DSM-IV-TR: Emphasized that bodily symptoms are medically unexplained

->DSM-5 TR: Focuses on distress related to bodily concerns

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3
Q

What is Somatic Symptom Disorder (SSD)?

A

->Pierre Briquet (1859) – Briquet’s syndrome: Patients always feel sick or experience symptoms

->Definition: Preoccupation with health or the body (“soma” = body)

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4
Q

What are the key clinical features of SSD?

A

->Constantly feeling weak & ill

->Life revolves around symptoms

->Ex. Severe pain worsened by psychological factors → Leads to anxiety & distress

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5
Q

How is SSD diagnosed?

A

-> At least one symptom is present with intense rxn to symptoms

->Specifiers for severity & dominant symptom type

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6
Q

How does Somatic Symptom Disorder differ from Illness Anxiety Disorder?

A

Somatic Symptom Disorder:
->Physical symptoms due to stress & anxiety
->Symptoms affect daily life

Illness Anxiety Disorder:
->Minimal or no physical symptoms
->Fear of developing a serious illness (e.g., mild rash = skin cancer)

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7
Q

What is Illness Anxiety Disorder (IAD)?

A

->Persistent fear of serious illness despite few/no symptoms

->Categorical vs Dimensional approach → IAD is dimensional

->Physical symptoms are mild or absent

->Concern is about the idea of being sick

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8
Q

How do patients with IAD respond to medical reassurance?

A

->Doctor’s reassurance is not helpful → Patients go from doctor to doctor

->“Disease Conviction”: Firm belief that they have an illness despite medical evidence

->Focus on long-term process of illness and disease EX. cancer, autoimmune disease

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9
Q

What are the two types of Illness Anxiety Disorder?

A
  1. Care-seeking type: Frequently visits doctors/tests (more common)
  2. Care-avoidant type: Avoids doctors due to overwhelming anxiety
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10
Q

What are the key statistics for SSD & IAD? (prevelance, comorbidtiy, onset)

A

-> Lifetime prevalence: 1-5%

->Severe Illness Anxiety: Late onset (increases with age)

->SSD Onset: Adolescence
—>More common in unmarried women, low SES, low education
—>Culture-specific symptoms (e.g., burning sensations in hands & feet in Pakistan-India)

->Comorbidities: Anxiety & Mood Disorders

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11
Q

What are the causes of Somatic Symptom and Illness Anxiety Disorder?

A

->Cognitive factors are central (sometimes called Disorders of Cognition)

->”Catastrophic” misinterpretations of bodily sensations

->Strong beliefs that unexplained bodily changes = serious illness

->Dysfunctional mind-set leads to worry about health

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12
Q

What is the Cognitive Model of Health Anxiety?

A

->A model that explains how health anxiety develops

->Involves four contributing factors

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13
Q

What are the four contributing factors to health anxiety?

A
  1. Critical precipitating incident
  2. Previous experience of illness (e.g., “Mom had heart palpitations → cancer; I have it → cancer”)
  3. Inflexible or negative cognitive assumptions
  4. Severity of anxiety (depends on factors that increase or decrease health anxiety)
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14
Q

What factors influence the severity of health anxiety?

A

Increase health anxiety:
->Perceived likelihood of illness
->Perceived costs & burden of illness

Reduce health anxiety:
->Perceived ability to cope
->Presence of “rescue” factors (e.g., availability of medical help)

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15
Q

What is Enhanced Somatic Sensitivity? Possible reasons?

A

->Interpreting ambiguous stimuli as threatening

->Possible reasons:
—>Genetic causes (modest, nonspecific tendency to overreact to stress)
—>Stressful life events

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16
Q

How do people with illness anxiety disorder focus on physical symptoms?

A

->Disproportionate incidence of disease in the family

->Social & interpersonal factors:
—>Some may unconsciously seek attention through illness

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17
Q

What is the illness anxiety disorder cycle?

A
  1. Faulty interpretation of physical symptoms
  2. Additional physical symptoms appear
  3. Increased anxiety
  4. Intensified focus on symptoms
  5. Cycle repeats
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18
Q

Why is Somatic Symptom and Illness Anxiety Disorder difficult to treat?

A

->Patients firmly believe they are ill despite reassurance

->Medical tests don’t relieve their concerns

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19
Q

What are common treatments for SSD & IAD disorders?

A
  1. Explanatory Therapy

-> Education & reassurance (explain the disorder, show test results)

  1. Cognitive-Behavioral Therapy (CBT)

->Reduce stress
-> Minimize help-seeking behaviors
-> Broaden social relationships beyond symptoms

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20
Q

How does Exposure-Based Therapy work?

A

->Expose patients to their fears without allowing safety behaviors

Ex. Show them a documentary on skin cancer → Don’t let them check their body for signs

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21
Q

What are Psychological Factors Affecting Medical Condition?

A

-> A diagnosed medical condition (e.g., asthma, diabetes, severe pain)

->Negatively affected by psychological/behavioral factors (e.g., anxiety, denial)

Ex. A person denies high blood pressure and refuses medication, worsening their condition

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22
Q

What is Functional Neurological Symptom Disorder (Conversion Disorder)?

A

->Previously called Hysteria (originally used to describe what are now known as conversion disorders)

->Origin of term: Believed to be caused by a wandering uterus (presumed to symbolize the longing to produce a kid)

->Freud’s theory: Repressed conflict converts into physical symptoms

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23
Q

How did Freud explain Conversion Disorder?

A

->Energy from repressed instincts is diverted into sensory-motor channels

->Physical dysfunction occurs without an organic cause

->Symptoms are a way for the unconscious mind to express conflict

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24
Q

What are common symptoms of Conversion Disorder? Examples?

A

->People appear neurologically impaired, but tests show no organic cause

Examples:

  1. Globus hystericus – Lump in throat, difficulty swallowing
  2. Astasia-abasia – Inability to stand/walk despite normal motor function
  3. Psychogenic seizures – Resemble epilepsy but EEG is normal
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25
Q

What are the two main symptom categories of Conversion Disorder?

A
  1. Motor Symptoms/Deficits (most common):
    ->Impaired coordination/balance
    ->Paralysis
    ->Abnormal limb posturing
    ->Muscle weakness (most frequent)
  2. Anesthesia (Loss of Sensation) (less common):
    ->Sudden blindness or tunnel vision
    ->Aphonia (loss of voice, can only whisper)
    ->Anosmia (loss of smell)
    ->Psychogenic seizures (appear suddenly in stressful situations)
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26
Q

How do Conversion Disorder symptoms progress?

A

->Acute episode: Symptoms last less than 6 months, often disappear with stress relief

->Persistent: Symptoms last more than 6 month

27
Q

What are two closely related disorders?

A
  1. Malingering (faking symptoms for external gain)

-> La belle indifférence: Emotional indifference to symptoms (e.g., someone paralyzed but unconcerned)

  1. Factitious Disorder (faking illness for attention, not external reward)

->two types:

  1. Factitious disorder imposed on self (fake own illness)
  2. Factitious disorder imposed on another (e.g., often a mom making a child sick → previously called “Munchausen by Proxy”)
28
Q

What are the key psychological processes in Conversion Disorder?

A

->Unconscious mental processes

->Dissociation of experiences from awareness

->Some may fake symptoms (malingering or factitious disorder)

29
Q

What are the statistics on Conversion Disorder?

A

->Rare in mental health settings

->More common in neurological settings (30% prevalence)

->Primarily affects women, usually develops in adolescence

30
Q

What are the biological causes of Conversion Disorder?

A

-> Weak evidence for biological causes

->Brain scans show:

—>Left side of body more affected

—>Failure to activate right inferior frontal cortex

—>Increased amygdala-motor connectivity (linked to stress processing)

31
Q

How does behavioral theory explain Conversion Disorder?

A

-> Symptoms persist due to attention and benefits received

-> Can be a way to justify poor performance or avoid stress

->Illness behaviors learned from parents (e.g., exposure to parental illness)

32
Q

What were Freud’s four stages of Conversion Disorder?

A
  1. Trauma/conflict → Anxiety
  2. Repression of conflict (unconscious)
  3. Conversion to physical symptoms → Anxiety reduced (primary gain)
  4. Social attention/support received (secondary gain)
33
Q

What social and cultural factors contribute to Conversion Disorder?

A

-> Substantial stress (e.g., abuse, divorce)

->More common in less educated, lower socioeconomic groups

->Limited medical knowledge influences symptom selection

34
Q

What treatments are available for Conversion Disorder?

A

->Difficult to treat

->Identify and reduce stress

->Minimize help-seeking behaviors

->Cognitive-Behavioral Therapy (CBT)
—>Shifts focus away from symptoms
—>Cognitive restructuring (changing how they interpret symptoms)

->Symptom-focused CBT:
—>Teaching coping strategies
—>Emphasizing psychological/social factors

35
Q

What is Obsessive-Compulsive Disorder (OCD)?

A

Chronic disorder characterized by:

  1. Unwanted intrusive thoughts (obsessions)
  2. Repetitive behaviors or mental acts (compulsions) to reduce distress

->Causes significant distress & interference with daily life

->Not always logically connected to its purpose

36
Q

What mental health conditions commonly co-occur with OCD?

A

->Generalized anxiety

->Recurrent panic attacks

->Debilitating avoidance

->Major depression

->Suicidal ideation & attempts

->Severe obsessions predict suicide risk more than compulsions

37
Q

What does it mean that obsessions in OCD are ego-dystonic?

A

->Thoughts feel “out of line” with one’s identity and values

->Causes distress and discomfort because they contradict personal beliefs

38
Q

How severe is OCD in terms of impairment?

A

->One of the top ten most impairing conditions (World Health Organization)

->More severe obsessions → poorer quality of life

39
Q

What are the three main subtypes of OCD (include obsession and compulsion of each)?

A
  1. Symmetry/Exactness (“Just Right”)
    ->Obsession: Needing things to be perfectly symmetrical or aligned
    ->Compulsion: Ordering, arranging, repeating rituals
  2. Forbidden Thoughts/Actions (Aggressive/Sexual/Religious)
    ->Obsession: Fears of harming self/others, offending God
    ->Compulsion: Checking, avoidance, seeking reassurance
  3. Cleaning/Contamination
    ->Obsession: Fear of germs/contamination
    ->Compulsion: Excessive washing, wearing gloves/masks
40
Q

What is the comorbidity of OCD?

A

Highly comorbid with:
->Anxiety disorders
->Mood disorders
->Impulse-control disorders
->Substance use disorders

->Leads to high impairment and difficulty in treatment

41
Q

How are Tic Disorder and OCD related?

A

->Involuntary movements can co-occur with OCD

->Tourette’s syndrome is commonly linked

->Tic-related OCD: Obsessions almost always about symmetry

->movements may not be tics but compulsions

42
Q

What are the statistics on OCD? (prevalence, onset, common in)

A

->Lifetime prevalence: 1%–3%

->More common in boys during childhood, but equalizes in adolescence

->Onset: Early adolescence to mid-20s

->Rarely develops after early 30s

->Chronic once it develops

43
Q

How does culture influence OCD?

A

->The content of obsessions and type of compulsions may vary

Ex. Middle Eastern cultures → obsessions related to cleanliness & religion

44
Q

What are examples of intrusive harming thoughts in OCD?

A

Impulses to:
->Jump out of a window
->Jump in front of a car
->Push someone in front of a train
->Drop a baby
->Wish someone would die

45
Q

What brain structures are linked to OCD?

A
  1. Frontal lobes → Increased activation (overconcern with thoughts)
  2. Basal ganglia → Linked to motor behavior & tics (a set of subcortical structures caudate, putamen (smaller in people with OCD), globus pallidus, and amygdala)
    —>Smaller putamen in people with OCD
    —>PET scans show increased activation
    —>Connection to Tourette’s syndrome
46
Q

What is Freud’s psychoanalytic explanation for OCD?

A

->Fixation at anal stage due to harsh toilet training
Reaction formation:

->Resisting the urge to soil → compulsively neat and clean

47
Q

What is Rachman & Shafran’s theory of obsessions?

A

->Inflated sense of personal responsibility for preventing harm

Ex: Seeing a sharp object on the road and feeling responsible if someone crashes

->Thought-action fusion:

  1. Thinking about something makes it more likely to happen
  2. Thinking about something is as bad as doing it
48
Q

What are the most effective treatments for OCD?

A

->SSRIs (help 40-60% but relapse is common)

->Exposure & Response Prevention (ERP) → Most effective
—>Expose person to fear without allowing compulsion
—>High dropout rate
Cognitive Behavioral Therapy (CBT)

->Psychosurgery (Cingulotomy) → Last resort

->Deep Brain Stimulation → Reversible but invasive

49
Q

What is Body Dysmorphic Disorder (BDD)?

A

->Preoccupation with an imagined defect in appearance
—>“Imagined ugliness” despite looking normal

->Ritualistic/compulsory behaviors:
—>Mirror checking, excessive grooming, skin picking
—>Some avoid mirrors entirely

->Co-occurs with OCD

->High suicide risk

49
Q

What areas of concern are common in BDD?

A

With muscle dysmorphia clarify:

  1. Good/Fair insight
    ->(know your imagining it)= more likely to respond to treatment then poor
    ->(know what your feeling about self is it’s probably true)
  2. Absent insight
    ->100% convinced that body dysmorphia is true
50
Q

What are statistics for BDD? (prevalence, onset, common)

A
  • Prevalence- Difficult to estimate- tends to be kept secret
  • Affects 1.7-2.4% people worldwide
  • Strong interest in art and design
  • Onset in early adolescence through the 20s
  • High degree of stress, reduced quality of life, and impairment common
51
Q

What are the causes of BDD? (bilogical, cognitive, & psychoanalytic)

A
  1. Biological:
    ->Reduced brain volume in:
    —>Right orbitofrontal cortex (self-image processing)
    —>Left anterior cingulate cortex (emotional regulation)
  2. Cognitive factors:
    ->Catastrophic thinking about appearance
    ->Maladaptive coping (ex: mirror checking, avoiding social situations)
  3. Psychoanalytic explanation:
    ->Displacement of anxiety onto physical appearance
52
Q

What are effective treatments for BDD?

A

->SSRIs (Fluvoxamine - specific for OCD/BDD)

->CBT (focuses on stopping compulsive behaviors)

->Exposure & Response Prevention (ERP)

Ex: Prevent mirror checking or encourage social interactions

53
Q

Why is plastic surgery NOT an effective treatment for BDD?

A

->It is avoidance behavior, not a real solution

->Most patients seek repeated surgeries

->8%-25% of plastic surgery patients have BDD

->Plastic surgeons should screen for BDD before operating

54
Q

When does hoarding typically start, and when do people seek treatment?

A

->Starts early in life and worsens over time

->Patients usually seek treatment after age 50

55
Q

What are the three major characteristics of hoarding disorder?

A
  1. Excessive acquisition of items
  2. Difficulty discarding anything
  3. Extreme clutter and disorganization
56
Q

What is unique about animal hoarding?

A

-> It’s not about the number of animals but how they are treated

Ex. A person keeps a dead pet’s body because they can’t part with it

57
Q

What are the possible causes of hoarding disorder?

A

->Genetic factors may contribute

->Cognitive factors:

—>Erroneous beliefs about the importance of possessions

—>Emotional attachment to objects due to lack of emotional attachments with people

58
Q

What are the treatments for hoarding disorder?

A

-> SSNRI (venlafaxine)

->Cognitive-Behavioral Therapy (CBT) → Focuses on faulty thoughts about attachment to objects

59
Q

What is Trichotillomania (Hair-Pulling Disorder)?

A

->Compulsive pulling of hair, leading to hair loss

->Common areas: Scalp, eyebrows, eyelids

->Affects 1%–5% of college students, more common in females

->Intense shame → People try to hide it with hats, wigs, etc.

60
Q

What is Excoriation (Skin-Picking Disorder)?

A

->Compulsive picking of the skin → Leads to scabs, scars, and open wounds

->Affects 1%–5% of the population

->Common areas: Face, hands, arms

->Tools used: Fingernails, tweezers, needles

->Must be chronic and cause skin lesions to qualify as a disorder

61
Q

What are the two main causes of Trichotillomania and Excoriation? (trigger and behavior for each model)

A
  1. Emotion Regulation Model

Trigger: Negative emotions (stress)

Behavior: Hair-pulling or skin-picking reduces stress (negative reinforcement)

  1. Frustrated Action Model

Trigger: Boredom or frustration

Behavior: Hair-pulling or skin-picking relieves boredom

62
Q

What is the most effective treatment for Trichotillomania and Excoriation?

A

->Habit Reversal Training (HRT) → Helps replace behavior with a competing response

  1. Self-monitoring: Track frequency/intensity of behavior
  2. Awareness training: Identify triggers
  3. Competing response: Replace behavior with a different action

Ex: Instead of picking skin, sit on hands or use a stress toy