Chapter 47: Acute and Chronic Kidney Flashcards

1
Q

Risk

A

Serum creatinine increased × 1.5
OR
GFR decreased by 25%

Urine output
<0.5 mL/kg/hr
for 6 hr

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2
Q

Injury

A

Serum creatinine increased × 2
OR
GFR decreased by 50%

Urine output
<0.5 mL/kg/hr
for 12 hr

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3
Q

Failure

A
Serum creatinine increased × 3
OR
GFR decreased by 75%
OR
Serum creatinine >4 mg/dL with
acute rise ≥0.5 mg/dL
Urine output
<0.3 mL/kg/hr
for 24 hr
(oliguria)
OR
Anuria for 12 hr
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4
Q

Loss

A

Persistent acute kidney failure;
complete loss of kidney
function >4 wk

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5
Q

End-stage
kidney
disease

A

Complete loss of kidney function

>3 mo

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6
Q

Nonoliguria AKI indicates a urine output

A

greater than

400 mL/day

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7
Q

a urinalysis may show

A

specific gravity fixed at around 1.010 and urine osmolality at
about 300 mOsm/kg (300 mmol/kg)

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8
Q

Fluid overload

can eventually lead to

A

HF, pulmonary edema, and pericardial

and pleural effusions.

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9
Q

Uncontrolled

hyponatremia or water excess

A

can lead to cerebral edema

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10
Q

The best serum indicator of AKI

A

creatinine

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11
Q

Asterixis

A

(flapping tremor when the wrist is extended)
is most common with liver failure, but has been known to occur
with advanced and severe kidney dysfunction.

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12
Q

During the diuretic phase of AKI, daily

urine output is usually around

A

1 to 3 L, but may reach 5 L or
more.
caused by osmotic diuresis

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13
Q

The recovery phase begins when the

A

GFR
increases, allowing the BUN and serum creatinine levels to
plateau and then decrease

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14
Q

Urine sediment

containing abundant cells, casts, or proteins suggests

A

intrarenal disorders

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15
Q

kidney ultrasound

A

first test done, since it provides
imaging without exposure to potentially nephrotoxic contrast
agents.

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16
Q

A primary nutritional goal in AKI

is to maintain adequate caloric intake

A

(providing 30 to 35 kcal/
kg and 0.8 to 1.0 g of protein per kilogram of desired body
weight) to prevent the further breakdown of body protein for
energy purposes. (diet consists of carbs and fat sources and supplements of essential amino acids.)

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17
Q
Nursing diagnoses and a potential complication for the patient
with AKI (examples-NANDA) p1106
A

• Excess fluid volume related to kidney failure and fluid
retention
• Risk for infection related to invasive lines, uremic toxins,
and altered immune responses secondary to kidney failure
• Fatigue related to anemia, metabolic acidosis, and uremic
toxins
• Anxiety related to disease processes, therapeutic interventions,
and uncertainty of prognosis
• Potential complication: dysrhythmias related to electrolyte
imbalances

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18
Q

infection is the leading cause of death in AKI

A

meticulous

aseptic technique is critical

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19
Q
The Kidney Disease Outcomes
Quality Initiative (KDOQI) of the National Kidney Foundation
defines CKD as either the
A

presence of kidney damage or a
decreased GFR less than 60 mL/min/1.73 m2 for longer than 3
months.

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20
Q

end-stage kidney (renal)

disease (ESKD), occurs when

A

GFR is less than 15 mL/min.
At this point, RRT (dialysis or transplantation) is required to
maintain life

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21
Q

Stage 1

A
Kidney damage
with normal or
↑ GFR
≥90
Diagnosis and treatment
CVD risk reduction
Slow progression
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22
Q

Stage 2

A

Kidney damage
with mild ↓ GFR
60-89
Estimation of progression

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23
Q

Stage 3

A

Moderate ↓ GFR
30-59
Evaluation and treatment of
complications

24
Q

Stage 4

A
Severe ↓ GFR
15-29
Preparation for renal
replacement therapy
(dialysis, kidney transplant
25
Q

Stage 5

A
Kidney failure
<15 (or dialysis)
Renal replacement therapy
(if uremia present and
patient desires treatment)
26
Q

Uremia

A
is a syndrome in which kidney function
declines to the point that symptoms may develop in multiple
body systems
occurs when GFR is
10 mL/min or less
27
Q

ACI….. As the GFR decreases

A

the

BUN and serum creatinine levels increase

28
Q

insulin depends on the kidneys for excretion

A

remains in circulation
longer.

Mild to moderate hyperglycemia and hyperinsulinemia may
occur.

29
Q

The altered lipid

metabolism is related to

A

decreased levels of the enzyme lipoprotein
lipase, which is important in the breakdown of lipoproteins

hyperlipidemia

30
Q

Sodium intake must be

individually determined but is generally

A

restricted to 2 g/day.

31
Q

hormone produced by the kidney

A

erythropoietin

32
Q

two

equations used most frequently to estimate GFR are the

A

Cockcroft-Gault formula and the Modification of Diet in Renal
Disease (MDRD)

33
Q

Cockcroft-Gault GFR =

A

(140 − Age) × (Weight in kilograms) × (0.85 if female)/(72 × Cr).

34
Q

A focus on stages 1 through 4 before the need for dialysis, stage 5

A

includes the control of hypertension,

hyperparathyroid disease, anemia, hyperglycemia, and dyslipidemia.

35
Q

Never give sodium polystyrene sulfonate to a patient

A

with a hypoactive bowel (paralytic ileus) because fluid shifts
could lead to bowel necrosis.

36
Q

recommended

that the target BP be

A

less than 130/80 mm Hg for patients with

CKD and 125/75 mm Hg for patients with significant proteinuria

37
Q

a total parathyroidectomy

A

some parathyroid
tissue is transplanted into the forearm. The transplanted
cells produce PTH as needed.

38
Q

Nursing diagnoses for CKD may include, but are not limited to,
the following:

A

• Risk for electrolyte imbalance related to impaired kidney
function resulting in hyperkalemia, hypocalcemia, hyperphosphatemia,
and altered vitamin D metabolism
• Imbalanced nutrition: less than body requirements related
to restricted intake of nutrients (especially protein),
nausea, vomiting, anorexia, and stomatitis

39
Q

Instruct the patient to avoid certain over-the-counter

drugs such as

A

NSAIDs and aluminum- and magnesium-based

laxatives and antacids.

40
Q

advantages of PD peritoneal dialysis

A
• Immediate initiation in
almost any hospital
• Less complicated than
hemodialysis
• Portable system with CAPD
• Fewer dietary restrictions
• Relatively short training
time
• Usable in patient with
vascular access problems
• Less cardiovascular stress
• Home dialysis possible
• Preferable for diabetic
patient
41
Q

disadvantages of PD

A
• Bacterial or chemical peritonitis
• Protein loss into dialysate
• Exit site and tunnel infections
• Self-image problems with catheter
placement
• Hyperglycemia
• Surgery for catheter placement
• Contraindicated in patient with
multiple abdominal surgeries,
trauma, unrepaired hernia
• Requires completion of education
program
• Catheter can migrate
• Best instituted with willing partner
42
Q

advantages of HD hemodialysis

A
• Rapid fluid removal
• Rapid removal of urea and
creatinine
• Effective potassium
removal
• Less protein loss
• Lowering of serum
triglycerides
• Home dialysis possible
• Temporary access can be
placed at bedside
43
Q

disadvantages of HD

A
• Vascular access problems
• Dietary and fluid restrictions
• Heparinization may be necessary
• Extensive equipment necessary
• Hypotension during dialysis
• Added blood loss that contributes
to anemia
• Specially trained personnel
necessary
• Surgery for permanent access
placement
• Self-image problems with
permanent access
44
Q

To perform HD,

A

a very rapid blood flow is

required, and access to a large blood vessel is essential

45
Q

HD high risk for

A

air ebolus

46
Q

PD more easier done at

A

home than HD

47
Q

PD takes a

A

loner time than HD

48
Q

HD has an increased risk for

A

hep B & C

49
Q

AV GRAFT… description

A

synthetic tubing tunneled beneath the skin connecting an artery and vein

50
Q

AV shunt

A

external loop of synthetic tubing connecting an artery and vein

51
Q

AV fistula

A

internal surgical anastomsis between an artery and a vein

52
Q

thrill

A

buzzing sensation indicates graft is patent

53
Q

When caring for a patient during the oliguric phase of acute kidney injury (AKI), what is an appropriate nursing intervention?

A

Patients in the oliguric phase of acute kidney injury will have fluid volume excess with potassium and sodium retention. Therefore they will need to have dietary sodium, potassium, and fluids restricted. Daily fluid intake is based on the previous 24-hour fluid loss (measured output plus 600 ml for insensible loss). The diet also needs to provide adequate, not low, protein intake to prevent catabolism. The patient should also be weighed daily, not just three times each week.

54
Q
The patient was diagnosed with prerenal AKI. The nurse should know that what is most likely the cause of the patient's diagnosis?
  IV tobramycin (Nebcin)
  Incompatible blood transfusion
  Poststreptococcal glomerulonephritis
  Dissecting abdominal aortic aneurysm
A

A dissecting abdominal aortic aneurysm is a prerenal cause of AKI because it can decrease renal artery perfusion and therefore the glomerular filtrate rate. Aminoglycoside antibiotic administration, a hemolytic blood transfusion reaction, and poststretpcoccal glomerulonephritis are intrarenal causes of AKI.

55
Q

The nurse knows the patient with AKI has entered the diuretic phase when what assessments occur

A

Dehydration, hypokalemia, and hyponatremia occur in the diuretic phase of AKI because the nephrons can excrete wastes but not concentrate urine. Therefore the serum BUN and serum creatinine levels also begin to decrease.

56
Q

Diffusion, osmosis, and ultrafiltration occur in both hemodialysis and peritoneal dialysis. The nurse should know that ultrafiltration in peritoneal dialysis is achieved by which method?

A

Ultrafiltration in peritoneal dialysis is achieved by increasing the osmolality of the dialysate with additional glucose. In hemodialysis the increased pressure gradient from increased pressure in the blood compartment or decreased pressure in the dialysate compartment causes ultrafiltration. Decreasing the concentration of the dialysate in either peritoneal or hemodialysis will decrease the amount of fluid removed from the blood stream.

57
Q

Continuous venovenous hemofiltration (CVVH)

A

CVVH removes large volumes of water and solutes from the patient over a longer period of time by using ultrafiltration and convection