Chapter 29: High Risk Newborn: Complications Associated with Gestational Age and Development Flashcards

1
Q

late preterm infants (LPIs)

A
  • born between 34 0/7 and 36 6/7 weeks of gestation
  • more stable than preterm infants but are physiologically and metabolically immature and have higher morbidity and mortality than full term infants
  • at risk for: hypothermia, respiratory problems, hypoglycemia, hyperbilirubinemia, feeding difficulties, sepsis, long term neurodevelopmental disorders
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2
Q

what are contributing factors to late preterm birth?

A
  • elective and medically induced inductions and cesarean births
  • preterm labor
  • pROM
  • preeclampsia
  • multifetal pregnancies
  • obesity
  • assisted reproductive technology
  • advanced maternal age
  • inaccurate estimate of gestational age before delivery
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3
Q

thermoregulation with LPIs

A
  • may develop cold stress that is not noticed until signs appear or until a vital signs check (which often occur only once a shift)
    • therefore, nurses should check V/S, esp temp, Q3-4 hours for first 24 hours and then every shift
  • kangaroo care: often used to keep infants warm
    • it is a method of skin to skin b/w infants and parents
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4
Q

feedings with LPIs

A
  • may have immature suck and swallow reflexes, shorter awake periods, and may fall asleep during feedings before being fed enough
  • may have difficulty with latch
  • have an inc caloric need and should be fed every 2-3 hours
  • football and cross cradle holds are helpful to use to help infants feed
  • monitor blood glucose at least 2x daily b/c infant at risk for hypoglycemia
  • must monitor and document breastfeeding evaluations at least 2x daily
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5
Q

discharge of LPIs

A
  • should not be discharged earlier than 48 hours after bith
  • infants should be feeding adequately and have normal V/S for at least 24 hours before discharge
  • bilirubin levels should be checked
  • teach parents to keep infant warm
  • car seat challenge: ensure the infant can sit in car seat w/o bradycardia, apnea, or O2 desats
    • should be monitored for at least 30 min
  • teach parents signs of complications such as jaundice/dehydration
  • f/u w/in 24-72 hours
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6
Q

what is defined as preterm infant?

what are the 3 categories of prematurity?

A
  • preterm: any infant born before beginning of 38 weeks of gestation
    • late preterm: 34-37 weeks
    • moderate preterm: 32-34 weeks
    • very preterm: <32 weeks
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7
Q

moderate preterm

A
  • born between 32-34 weeks
  • higher risk than late preterm infants for hypothermia, respiratory problems, hypoglycemia, hyperbilirubinemia, feeding difficulties, sepsis, and long term neurodevelopmental disorders
  • do not have suck, swallow, breath coordination
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8
Q

early/very preterm

A
  • less than 32 weeks gestation
  • 24 weeks is point of viability
  • 500 grams is indicator of initial survival
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9
Q

low birth weight

A
  • low birth weight most often caused by prematurity
    • LBW: any infant weighing 5 lb 8 oz (2500 g) or less at birth and of any gestational age
    • very low birth weight (VLBW): infants weighing less than 3 lb 5 oz (1500 g) or less at birth
    • extremely low birth weight (ELBW): infants weighing less than 2 lb 3 oz (1000 g) at birth
  • leads to major complications
  • often the cause of: preterm labor, chronic health conditions (DM), infection, placental problems, not gaining weight during pregnancy, hx of LBW infants, cigarette/alcohol abuse
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10
Q

appearance of preterm infants

A
  • frail and weak
  • less developed flexor muscles and muscle tone
  • limp extremities with no resistance
    • often lie in extended position
  • lack subQ tissue or white fat which makes thin skin appear red and translucent
  • vernix and lanugo are abundant
  • plantar creases are absent if less than 32 weeks
  • pinnae of ears are soft, flat, and contain little cartilage
    • they lack the rolled over appearance
  • female: clitoris and labia minora are large and not covered by labia majora
  • male: undescended testes, with smooth scrotal sac
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11
Q

behavior of preterm infants

A
  • depends on gestational age
  • may have poor development of flexion and little excess energy for maintaining muscle tone
  • easily exhausted by noise and activity
  • may respond with low O2 levels and stress related behavior
  • cry may be feeble
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12
Q

problems with respiration in preterm infants

A
  • immature lungs
    • presence of surfactant in adequate amounts is of primary importance b/c it allows the work of breathing to be lowered
      • if lack of surfactant–>RDS
    • ppor cough reflex and narrowed respiratory passageways
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13
Q

assessment of preterm respiratory system

A
  • periodic vs apneic breathing
    • periodic: cessation of breathing for 5-10 sec w/o other changes followed by 10-15 sec of rapid respirations
      • no changes in color or HR
      • normal
    • apneic: absence of breathing lasting more than 20 sec
      • accompanied by cyanosis, pallor, bradycardia, hypotonia
      • may require tactile stimulation and ventilation
  • should assess for WOB and location and severity of retractions
  • grunting is an early sign of RDS
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14
Q

respiratory nursing care of preterm infants: equipment

A
  • respiratory equipment
    • O2 hood: if infant can breathe independently but need extra O2
    • NC: can be used if infant breathes well independently
      • can be used for home O2
      • should be warmed and humidified
    • CPAP: used to keep alveoli open and improve lung expansion
    • ventilation: when respiratory failure, severe apnea, bradycardia
      • can use high frequency ventilation to provide fast respirations w/ less pressure and volume
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15
Q

respiratory nursing care of preterm infants: Positioning

A
  • side or prone positions used to help with drainage of secretions
    • not recommended in normal infants b/c of SIDS risk
    • in preterm, prone position helps inc oxygenation, enhance respiratory control, improve lung mechanics and volume, and reduces energy expenditure
  • start supine sleeping as soon as infant can tolerate
    • can often be used at approx 32 weeks
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16
Q

respiratory nursing care of preterm infants: suctioning and maintaining hydration

A
  • suction mouth then nose
    • only suction as necessary
    • each suction attempt should only be 5-10 seconds long and inc O2 should be provided before and after each attempt
  • adequate hydration is important to keep secretions thin so they can be removed by drainage or suction
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17
Q

problems with thermoregulation in preterm infants

A
  • skin is thin, blood vessels near surface, and little subQ fat so heat loss is rapid
    • preterm so less brown fat was allowed to accumulate
  • also they are in extension rather than flexion, so allow more heat loss
  • temp control center of the brain is immature
  • complications of heat loss: hypoglycemia, respiratory problems, metabolic acidosis, pulmonary vasoconstriction, impaired surfactant production
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18
Q

assessment of thermoregulation in preterm infants

A
  • temp should be recorded Q30-60 min until stable, then Q1-3 hours
  • axillary temp should be between 36.3 deg C and 36.9 deg C
  • low temp may be early sign of infection
  • hypoglycemia and respiratory distress may be first sign of temp instability
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19
Q

signs of inadequate thermoregulation in preterm infants

A
  • axillary temp <36.3 deg C or >36.9 deg C
  • abdominal skin temp <36 deg C or >36.5 deg C
  • poor feeding or feeding intolerance
  • irritability followed by lethargy
  • weak cry or suck
  • dec muscle tone
  • cool skin temp
  • mottled, pale, or acrocyanotic skin
  • signs of hypoglycemia
  • signs of respiratory difficulty
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20
Q

nursing care to maintain a neutral thermal environment

A
  • neutral thermal env prevents the need for inc O2 to maintain the infant’s body temp
  • delivery room should be warm to dec heat loss at birth
    • immediately dry baby (and keep dry) and place on mother or in warmer
    • if less than 29 weeks: wrap in polyethylene bag to prevent evaporative heat loss
  • can use open radiant warmers, but be sure to prevent heat loss through convection from drafts
  • warmed and humidified O2, b/c thermal receptors in face are very sensitive
  • can use heated blankets and hats
  • warm formula, breastmilk
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21
Q

nursing care to wean an infant to an open crib

A
  • prep for this early
    • should keep an infant dressed as much as possible even if in incubator to help infant get used to different temp on face than rest of body
  • can begin gradual weaning from external heat if:
    • weight about 3 lb 5 oz (1500 g)
    • consistent weight for 5 days
    • no medical complications
    • tolerating feedings
  • when move to crib, should double wrap infant to insulate body heat
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22
Q

problems with fluid and electrolyte imbalance in preterm infants

A
  • preterm infants lose fluid easliy and loss inc with degree of prematurity
    • rapid RR and use of O2 inc fluid loss from lungs
    • thin skin and lack of flexion inc water loss
    • heat from radiant warmers/incubators leads to water loss
  • ability of kidneys to conc or dilute urine is poor, so fragile balance between dehydration and overhydration
    • normal urinary output: 1-3 mL/kg/hour during first few days
    • after 24 hours: output less than 0.5 mL/kg/hour is considered oliguria
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23
Q

assessment of fluid and electrolyte balance in preterm infants

A
  • be on high alert for fluid overload or deficit
  • monitor I/O, strictly
  • weigh diapers to determine output (1 g=1 mL)
    • check specific gravity to determine if dilute or concentrated urine (should be b/w 1.002-1.010)
  • daily weights of infant to determine fluid loss or gain
  • monitor for signs of dehydration and overhydraiton
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24
Q

signs of dehydration in the newborn

A
  • urine output <1 mL/kg/hour
  • urine SG >1.010
  • weight loss greater than expected
  • dry skin and mucous membranes
  • sunken anterior fontanel
  • poor tissue turgor
  • blood: elevated Na, protein, HCT
  • hypotension
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25
Q

signs of overhydration in the newborn

A
  • urine output >3 mL/kg/hour
  • urine SG <1.002
  • edema
  • weight gain greater than expected
  • bulging fontanels
  • blood: dec Na, protein, HCT
  • moist breath sounds
  • difficulty breathing
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26
Q

nursing interventions for fluid and electrolyte balance in preterm infants

A
  • regulate IV fluids with a prevision of 0.1 mL/hour
  • IV meds diluted in as little fluid as possible
  • hourly check of IV site for infiltration
  • strict I&O with SG checks
  • weigh infants daily
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27
Q

skin problems in preterm infants

A
  • skin is fragile, permeable, and easliy damaged
    • do not use standard adhesive tape on their skin
    • disinfectants can injure their skin
  • assess skin regularly
  • nursing interventions:
    • avoid adhesives, alcohol, or betadine
    • remove adhesive slowly
    • chlorhexidine gluconate is a common disinfectant that can be used
    • pH balanced cleanser (w/ pH from 5.5-7) should be used for bath (preterm infants should not be bathed every day)
    • humidity in incubators to reduce drying of skin
    • use emollients on skin to help reduce skin fissures
    • frequent position changes to reduce skin breakdown
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28
Q

infection in preterm infants

A
  • high rate of infection due to exposure to maternal infection, lack of IgG transfer from mother, immature immune system
    • prolonged hospital stays and invasive procedures also inc the risk
  • be alert for signs of sepsis
  • nursing interventions:
    • hand washing
    • no jewelry
    • limit exposure to family and staff who have contagious illnesses
    • strict sterile technique for central lines/dressing changes
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29
Q

pain assessment in preterm infants

A
  • pain assessed whenever V/S assessed with NIPS or Premature Infant Pain Profile (PIPP)
    • these assess gestational age and behavior states, HR, O2 sats, brow bulge, eye squeeze, and nasolabial furrow
  • common signs of pain:
    • inc/dec HR and RR
    • inc BP
    • dec O2 sats
    • color changes: red, dusky, pale
    • high pitched, intense, harsh cry
    • whimpering, moaning
    • eyes squeezed shut
    • mouth open grimacing
    • furrowing or bulging of brow
    • tense, rigid muscles or flaccid muscle tone
    • rigidity or flailing of extremities
    • sleep wake pattern changes
30
Q

nursing interventions for pain in preterm infants

A
  • prepare infants for painful precedures by waking them slowly and gently and using containment
    • (simulates the enclosed space of the uterus and prevents excessive and disorganized motor activity–>keep extremities flexed w/ swaddling or w/ hands, position in supine or side lying w/ at least 1 of the infant’s hands near the mouth for sucking)
  • kangaroo care and breastfeeding can help reduce pain
  • nonnutritive sucking on a pacifier
    • put sucrose in infant’s mouth 2-3 min before procedure with pacifier
  • opioids and acetaminophen
31
Q

signs of overstimulation in preterm infants

A
  • O2 changes:
    • BP, pulse, RR instability
    • cyanosis, pallor, mottling
    • flaring nares
    • dec O2 sats
    • sneezing, coughing
  • behavior changes:
    • stiff, extended arms and legs
    • fisting of hands or splaying of fingers
    • arching
    • alert, worried expression
    • turning away from eye contact (gaze aversion)
    • regurgitation, gagging, hiccupping
    • yawning
    • fatigue signs
32
Q

nursing interventions if an infant is overstimulated

A
  • schedule periods of undisturbed rest throughout the day
    • avoid waking infant during quiet sleep phase
    • cluster or group care so more rest allowed w/o interruption
      • but keep clustered care short
      • do not include painful procedures in clustered care
    • provide short rest periods during activities
  • reduce stimuli: light and sound
  • schedule quiet periods that are at least 1 hour long
    • promote flexion which helps to reduce energy loss and promotes quieting and reduces stress
  • massage
  • promoting motor development by keeping infant in an extended, frog leg position
    • reposition every 2-3 hours
  • individualize care
  • communicate an infants’ needs
33
Q

nutrition in a preterm infant

A
  • preterm infants have a lack of fat stores and they use of glucose stores quickly after birth
    • hypoglycemia often develops
  • have small digestive capacity
  • avg healthy preterm infants should gain 15-20 g/kg/day
  • poor absorption in gut of fats b/c of insufficient bile acids and lipase
  • poor coordination of suck and swallow
  • fatigue easily
34
Q

assessment of nutrition in preterm infants

A
  • assess for feeding readiness and tolerance
  • gavage
    • use until about 33-34 weeks gestation
    • check residual volumes if on enteral feedings to determine if tolerating
      • if more than 1/2 of last feeding remains, report it, b/c may be a sign of necrotizing enterocolitis (NEC)
    • vomiting can indicate feedings are too large
    • watch for signs of visible loops of bowel, abdominal distention, ileus, sepsis, obstruction
  • nipple feeds
    • watch for signs of readniess
    • most infants ready to begin around 34-35 weeks of corrected age
    • assess coordination of suck and swallow and breathing
    • assess RR before and during feedings–>if >60, then use gavage feeding to prevent aspiration
35
Q

what are signs that a preterm infant is ready for nipple feeding?

A
  • rooting
  • RR < 60 breaths per min
  • inc ability to tolerate holding and handling
  • intact gag reflex
  • must have enough energy to feed w/o compromised oxygenation
  • coordination of suck and swallow
36
Q

adverse signs during nipple feeding

A
  • tachycardia
  • bradycardia
  • inc or dec RR
  • nasal flaring
  • markedly dec O2 sats
  • cyanosis, pallor
  • apnea
  • choking, coughing
  • gagging, regurgitation
  • drooling, gulping
  • falling asleep early in feeding
  • feeding longer than 20-30 min
37
Q

TPN in preterm infants

A
  • IV infusion of solution containing major nutrients needed for metabolism and growth
  • may be necessary due to respiratory problems, limited gastric capacity, surgery, or reduced peristalsis
38
Q

enteral feedings in preterm infants

A
  • also called trophic feedings
  • stimulate development of the GI tract, enhance gut motility, dec need for TPN, and shorten hospital stay
  • bowel sounds should be present, there should be no abdominal distention, and infants should be stable
  • human breast milk or colostrum can be used and is preferred
    • but preterm infants often need special formulas or fortified breast milk b/c more easily digested and have more calories (at 24 cal/oz instead of 20)
39
Q

how can a nurse help a mother with a premature infant who wants to breast feed?

A
  • offer support and encouragement and tell her her milk is important to help her baby
  • help her to use a breast pump ASAP after birth, then for at least 8 times daily for 10-15 min
  • give mom sterile containers to store milk in
  • if fortifiers will be put in milk, explain the higher needs of the preterm infant so that the mother does not think something is wrong with her milk
  • encourage mom in her efforts
  • provide privacy and relaxation
  • teach mom to use football and cross cradle holds
40
Q

parenting a preterm infant

A
  • emotionally traumatic experience
  • parents cannot hold or feed infant or offer care
  • when infant is not capable of normal newborn behaviors or when the infant’s appearance is not what is expected, attachment may be delayed
  • extended hospitalization causes separation, produces emotional trauma, and disrupts family life
  • guilt and loss of control are common feelings
41
Q

assessment of parents of preterm infants

A
  • assess for signs of parental attachment
    • they may be fearful at first, but they should talk about infant in positive terms, make eye contact, name the infant and call it by name
    • they should ask questions about the infant, should smilke and talk to infant
  • determine if other stressors in parents’ lives that may interfere with ability to visit and form attachment with infant:
    • financial need to work
    • lack of transportation
    • long distances
    • other children
42
Q

signs that bonding may be delayed with a preterm infant

A
  • using negative terms to describe infant
  • discussing infant in impersonal or technical terms
  • failing to give infant a name or use the name
  • visiting or calling infrequently
  • dec number/length of visits
  • showing interest in other infants equal to their own
  • refusing offers to hold and learn to care for infant
  • showing dec in or lack of eye contact
  • spending less time talking to or smiling at infant
43
Q

nursing interventions for parents of preterm infants

A
  • advance preparations and education
  • allow parents to see and touch newborn in delivery room–helps bonding process
  • allow father to watch initial care in NICU to allow him to inc confidence in staff and enables him to give mom a full description
  • take parents to NICU ASAP
    • teach them what to expect, support them as they visit with infant
    • help them hold baby ASAP
  • support fathers and encourage them to participate in hands on newborn care
  • provide accurate info to the parents
    • may often have to repeat things, b/c of emotional stress, they often forget what they heard
  • institute kangaroo care
44
Q

kangaroo care

A
  • KC should begin ASAP and is method of providing skin to skin contact
    • infant wears only a diaper and a hat and is placed upright under the mother’s clothes b/w the breasts (father can also do KC)
  • advantages: opportunity for parents to be involved in care, stabilizes vital signs, inc weight gain, shorter hospital stay, more quiet sleep, less crying, promotes thermoregulation/bonding, helps relieve pain
    • upright position also makes breathing easier
    • containment of extremities dec purposeless movement that uses O2 and calories
    • gentile stimulation is provided
  • should last at least an hour to improve th einfant’s sleep
45
Q

preparing for discharge with a preterm infant

A
  • begin to teach parents early about procedures, treatments, and meds
    • observe the parents and praise their efforts
    • teach them what is normal for their infant and how to respond to abnormal signs
    • help determine any adaptations that need to be made at home
  • infants may require O2, cardiorespiratory monitoring, suctioning, gavage or gastrostomy feedings
    • feedings will need to happen Q3 hours and may be time consuing
  • infants may have a hard time sleeping at first
  • assist parent into determining how to integrate new infant into family
  • car seat challenge should be performed
  • immunizations and hearing screen should be done
46
Q

signs a preterm infant is ready for discharge

A
  • sustained pattern of weight gain, adequate maintenance of body temp in open bed, feeding w/o cardiorespiratory compromise, and stable, cardiorespiratory functioning
  • appropriate immunizations given
  • car seat challenge completed
  • metabolic screening and hearing test performed
  • family and home evaluated
    • family must have at least 2 members who can feed and provide care, perform CPR, give meds, operate equipment
  • f/u care has been scheduled
47
Q

respiratory distress syndrome (RDS)

A
  • condition caused by insufficient production of surfactant in the lungs
    • occurs most often in preterm infants under 28 weeks
    • also occurs in birth asphyxia, C/S, multiple births, male gender, cold stress, and GDM–>all interfere with surfactant production
    • less frequent if antenatal corticosteroids or chronic fetal stress (materal HTN or prolonged ROM) occurs
  • enough surfactant to prevent RDS not usually made until 34-36 weeks
    • if too little surfactant, the lungs become noncompliant or stiff and resist expansion, so WOB inc and retractions result along with seesaw respirations
  • tests of amniotic fluid can detect lecithin, sphingomyelin, phosphatidylglycerol, and phophatidylinositol which are components of surfactant
    • the test can predict whether the fetal lungs are mature enough for survival of the lungs
    • tests are always performed before an induction or C/S of an infant younger than 38 weeks
48
Q

manifestations of RDS

A
  • begin during 1st hours after birth
    • tachypnea
    • tachycardia
    • nasal flaring
    • xiphoid and intercostal retractions
    • cyanosis
    • audible grunting
  • acidosis develops as a result of hypoxemia
    • inc CO2 and dec O2
  • ground glass appearance of lungs of CXR
  • signs become worse and peak w/in 3 days then begin to improve
49
Q

therapeutic management of RDS

A
  • surfactant is instilled into the infant’s trachea shortly after birth or as soon as signs of RDS apparent
  • O2
  • CPAP
  • inhaled nitric oxide
  • correction of acidosis
  • IV fluids
50
Q

bronchopulmonary dysplasia (BPD)

A
  • also known as chronic lung dz
  • chronic condition in which damage to infant’s lungs requires prolonged dependence on supplemental oxygen
    • occurs most often in infants less than 32 weeks and if VLBW
  • patho: results from combo of high levels of O2, oxygen free radicals, and pressure of mechanical ventilation that injure bronchial epithelium and result in inflammation, atelectasis, edema, and airway hyper-reactivity with loss of cilia, thickening of the walls of alveoli, and fibrotic changes
51
Q

BPD: manifestations and management

A
  • manifestations:
    • inc need for or an inability to be weaned for respiratory support
    • tachycardia
    • tachypnea
    • retractions
    • crackles
    • wheezing
    • respiratory acidosis
    • cyanosis
    • inc secretions
    • bronchospasm
    • changes on CXR
  • aim for prevention
    • steroids before birth to reduce prematurity and RDS
    • minimize exposure to O2 and pressure of ventilation
    • avoidance of fluid overload
    • inc nutrition
  • tx: supportive therapy: antibiotics and bronchodilators and gradual dec in O2
    • diuretics, fluid restriction
    • need frequent rehospitalization
52
Q

intraventricular hemorrhage (IVH)

A
  • it is bleeding into and around the ventricles in the brain
    • most common during 1st few days of life in infants less than 1500 grams or less than 32 weeks
    • also occurs in term infants from asphyxia or trauma
  • results from rupture of fragile blood vessels in the germinal matrix around the ventricles
    • assoc with inc or dec BP, asphyxia or respiratory distress, and inc cerebral blood flow
  • hemorrhage graded from 1-4
    • 1: small bleed
    • 2: extends to lateral ventricles
    • 3: causes distention of ventricles
    • 4: causes ventricular dilation and extends to other brain tissue
53
Q

IVH: manifestations, management, and nursing considerations

A
  • manifestations: determined by severity of hemorrhage
    • poor muscle tone
    • deterioration in respiratory status with cyanosis or apnea
    • dec HCT
    • acidosis
    • hyperglycemia
    • dec reflexes
    • tense fontanel
    • seizure
  • management: ultrasounds done to assess
    • tx: supportive and focuses on maintaining respiratory function
    • hydrocephalus may develop so a ventriculoperitoneal shunt may be necessary
  • nursing:
    • avoid situations that may inc the risk of IVH: so keep handling to a minimum, pain and environmental stressors should be reduced
    • daily assessments of hed circumference and changes in neuro status
54
Q

retinopathy of prematurity (ROP)

A
  • condition where injury to the blood vessels in the eye may result in visual impairment or blindness
    • more often in infants less than 1000 g and less than 29 weeks
  • patho: cause is unknown but high O2 is a risk factor
    • prolonged ventilation, acidosis, sepsis, shock, IVH are also risk factors
55
Q

ROP: management and nursing care

A
  • screen infants to detect changes int he eye
  • laser surgery can be used to destroy abnormal blood vessels
    • may also need cryosurgery (reattachment of the retina)
  • nursing:
    • check pulse ox if infant receiving O2
    • teach parents about reasons for eye exams
    • mydriatic eye drops used to dilate eyes may cause HTN, bradycardia, apnea
56
Q

necrotizing enterocolitis (NEC)

A
  • this is a serious inflammatory condition of the intestinal tract that may lead to cellular death of areas of intestinal mucosa
    • ileum and proximal colon are most affected
  • immaturity of the intestine is a major factor
    • previous hypoxia of the intestines may be the causative factor
    • incidence is higher after infants receive feedings
      • when infants are fed, bacteria proliferate, and gas forming organisms invade the intestinal wall which causes necrosis, perforation, and peritonitis
  • breast milk (due to the Igs, leukocytes, and antibacterial agents) may be preventive
57
Q

NEC: manifestations

A
  • inc abdominal girth due to distention
  • inc gastric residuals
  • dec or absent bowel sounds
  • loops of bowel seen thru abdominal wall
  • vomiting
  • bile stained residuals or emesis
  • abdominal tenderness
  • signs of infection
  • occult blood in stools
  • respiratory difficulty
  • apnea
  • bradycardia
  • temp instability
  • lethargy
  • hypoTN
  • presence of air in loops of bowel on radiograph is characteristic of this
58
Q

NEC: therapeutic management and nursing

A
  • supplementing formula with probiotics and breast milk may help prevent NEC
  • tx: abx, discontinuaiton of oral feedings, gastric suction, use of TPN to rest intestines
    • surgery may be needed if perforation or lack of improvement to remove necrosis (which may lead to short bowel syndrome)
  • nursing:
    • encourage moms to breast feed
    • early detection–>withhold next feeding and inform provider
    • measure abdominal girth and maintain IV fluids and TPN
    • measure I&O
    • position infant on their side to prevent pressure on diaphragm
59
Q

short bowel syndrome (SBS)

A
  • caused by a bowel that is shorter than normal due to surgical removal or a congenital condition
  • patho: dec mucosal surface causes inadequate absorption of fluids, electrolytes, and nutrients
  • manifestations:
    • malabsorption
    • diarrhea
    • faliure to thrive
  • management:
    • restore and stabilize fluid and electrolyte balance
    • nutritional support (TPN)
  • nursing:
    • manage TPN and enteral feedings
      • strict aspesis on central venous access device where TPN is infusing
      • advance enteral feedings slowly
    • monitor for signs of electrolyte imbalances
60
Q

problems with post term infants

A
  • those born after 42nd week of gestation
  • problems:
    • placenta begins to function poorly
      • if placental insufficiency–>dec amniotic fluid and compression of umbilical cord may occur–>fetus doesn’t receive appropriate O2 and nutrition–>SGA, hypoxia, malnourishment (postmaturity syndrome)
    • when labor begins, poor O2 reserves may cause fetal compromise
      • fetus may pass meconium due to hypoxia and inc risk for meconium aspiration
61
Q

assessment of postterm infants

A
  • if large, assess for injury and hypoglycemia
  • if postmaturity syndrome, may have apprehensive look associated with hypoxia
  • unusually alert and wide eyed
  • may be thin with loose skin and little subQ fat
  • no vernix or lanugo, but usually has abundant hair on head and long nails
  • may have green staining on cord, skin, and nails
  • umbilical cord is thin with little wharton’s jelly
  • cracked skin
62
Q

therapeutic management and nursing care for post term infants

A
  • if a mother overdue, placenta is tested for functioning, and if deteriorating, then labor is induced
  • apgar scores less than 7 more likely in post-term infants
    • may need respiratory support if asphyxia or meconium aspiration occurred
  • nurse: prevent complications and monitor for changes in status
    • respond to FHR decels, prepare for and assist with emergency delivery
    • if signs of postmaturity, assess for hypoglycemia ASAP
    • temp regulation may be poor b/c of fat stores used up for nourishment in utero
      • provide extra blankets, assess temp
63
Q

SGA: causes

A
  • those below the 10th percentile
  • causes:
    • congenital malformations,
    • chromosomal abnormalities,
    • genetic factors,
    • multiple gestations,
    • fetal infections (rubella, CMV),
    • poor placental functioning or small placental size or malformation,
    • illness in the mother such as pre E or GDM,
    • smoking, drug abuse, alcohol
64
Q

SGA: complications

A
  • low apgar scores
  • meconium aspiration
  • polycythemia
  • hypoglycemia
  • inadequate thermoregulation r/t subQ and brown fat stores being used up to survive in utero
65
Q

symmetric growth restriction

A
  • involves the entire body: measurements of the head, chest, length, and weight are below normal, or below the 10th percentile
  • caused by congenital anomalies, genetic disorders, exposure to infection or drugs early in pregnancy, genetic predisposition
  • body is proportionate and appears normally developed for size
    • total number of cells and cell size is dec
    • small throughout their lives
66
Q

asymmetric growth restriction

A
  • caused by complications like pre eclampsia that begin in 3rd trimester and interfere with uteroplacental function
  • head is normal in size but seems large for rest of body
    • brain growth and heart size are normal
    • length is normal, but weight is below 10th percentile
    • abdominal circumference is dec b/c liver, spleen, and adrenals are small
      • SO, head circumference and length are normal, but abdominal circumference and weight are low
67
Q

characteristics of SGA

A
  • infant appears thin, long, waster
  • dry loose skin with longitudinal thigh creases from loss of subQ fat
  • sunken abdomen
  • sparse hair
  • thin cord
  • facial appearance of being elderly
  • anterior fontanel may be wide or overlapping sutures
    • usually catch up in growth by 2 years if adequately nourished
68
Q

SGA: management and nursing care

A
  • prevent with good prenatal care
  • if suspect SGA is a problem, serial nonstress tests help determine if infant should be born early
  • problems should be treated early and as they occur (include asphyxia, meconium aspiration, hypoglycemia, polycythemia, temp instability)
  • nursing:
    • observe for complication
    • assess for hypoglycemia
    • early and frequent feedings
    • temp regulation and respiratory support
    • observe for jaundice
69
Q

LGA: causes

A
  • those who are above the 90% for gestational age on intrauterine growth charts
    • may have macrosomia: weigh between 8 lb 13 oz-9 lb 15 oz (4000-4500 g)
  • causes:
    • multiparous
    • large parents
    • mothers who are obese
    • ethnic groups known to have large infants
    • GDM
    • erythroblastosis fetalis
70
Q

LGA: complications

A
  • longer labor
  • suffer injury during birth
  • need a cesarean section
  • shoulder dystocia
  • fractures of the clavicle or skull
  • injury to the brachial plexus or facial N
  • cephalohematoma
  • subdural hematoma
  • bruising
  • congenital heart defects
  • mortality
71
Q

LGA: management and nursing care

A
  • management is based on identification of increased size during pregnancy by measurements of fundal height and ultrasound
  • delivery problems may be managed with the use of vacuum extraction, forceps, or cesarean birth
  • nursing:
    • assist with difficult delivery
    • assess for injuries, hypoglycemia, polycythemia
    • daily weights
    • assess for congenital heart defects: pulse ox, ECG, CXR
72
Q

what is the problem with preterm infants?

what is the problem with postterm infants?

A
  • preterm: respiratory problems
  • postterm: metabolic problems