chapter 15 control and co-ordination Flashcards
1
Q
dendrites
A
carry impulse towards cell body
2
Q
axon
A
carries impulse from cell body
3
Q
what are impulses
A
brief changes to the distribution of electrical charges across the membrane
4
Q
roles of sensory receptor cells
A
- detects stimuli
- acts as transducer which converts stimulus energy
into electrical energy , produces a receptor potential
that is strong enough to pass an impulse along the
sensory neurone
5
Q
taste chemoreceptor cells
A
- Na+ ions diffuse into cells via microvilli and increases
the positive charge in the cell. - membrane is depolarised and an receptor potential
is produced - volted-gated Ca+ channel opens and Ca+ ions
enters cells - trigger the movement of vesicles containing
neurotransmitters and neurotransmitters are released - neurotransmitters stimulates an action potential in
the sensory neurone and the impulse is send to the
taste centre in the brain
6
Q
resting potential
A
- Na+/K+ pump, 3 Na+ ions out and 2 K+ ions in. An
electrochemical gradient is set up, K+ will diffuse out
as Na+ diffuses in via channel proteins - more K+ channels open than Na+ channels and the
membrane is more permeable to K+ than Na+. More
K+ leaves than Na+ enters, leaking of K+ is
responsible for resting potential
7
Q
depolarisation
A
- voltage-gated K+ channels remains closed
- voltage-gated Na+ channels opens, Na+ ions enters cell and membrane becomes less negative generating an action potential
- the size of the action potential is at +30mV, the higher the intensity of the stimulus the higher the frequency of the action potentials
8
Q
repolarisation
A
- voltage-gated Na+ channel closes
2. voltage-gated K+ channel opens and K+ ions moves out of the cell
9
Q
refractory period
A
- voltage-gated Na+ channels remains closed
- voltage-gated K+ channels closes but there is a
delay hence excess K+ ions are leaked out - when in refractory period, no action potential can be
generated hence ensure one-way transmission of
action potential
10
Q
saltatory conduction
A
.faster transmission
.action potential only at nodes of Ranvier
.local circuit lengthens
.action potential ‘jumps’ from nodes to nodes
11
Q
cholinergic synapse
A
- action potential reaches the presynaptic membrane
- voltage-gated Ca2+ channel is open, membrane
becomes more permeable to Ca2+ and Ca2+ enters
the presynaptic membrane - vesicles containing ACh moves towards the
presynaptic membrane and fuses with the membrane.
ACh is released into the synaptic cleft - ACh diffuses across the synaptic cleft
- ACh binds with the receptor porteins on the
postsynaptic membrane
6.receptor proteins changes shape and Na+ channel
opens. Na+ enters the postsynaptic neurone causing it
to be depolarised, an action potential is generated
7.ACh is broken down by acetylcholinesterase in the
synaptic cleft into acetate and choline and ACh is
recycled to be used in the process of ATP
8.depolarisation stops at the postsynaptic membrane
so there is no continuous production of action
potential which may lead to paralysis
12
Q
muscle contraction
A
- cholinergic synapse (whole process)
- Ca2+ in sarcoplasm binds to troponin, troponin changes shape and moves away from tropomyosin. Exposes the myosin-binding site on actin and the myosin head will attach to the actin forming a cross bridge
13
Q
sliding filament model
A
- myosin head with ADP and pi forms a cross bridge with actin and pi is released
- myosin head tilts and pulls on actin, power strokes moves the actin towards the M line and the sarcomere shortens. ADP is released.
- ATP binds to the myosin head and ATPase hydrolyses ATP to ADP and pi. myosin head lets go of actin and returns to original position.