chapter 11-14 - Sheet1 Flashcards

1
Q

What is the primary focus of Passage 1?**

A

*A: Passage 1 focuses on providing an overview of the importance of nutrition.**

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2
Q
  1. Q: Name two macronutrients discussed in Passage 2.
A
  1. A: Carbohydrates and proteins are discussed as macronutrients in Passage 2.
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3
Q
  1. Q: In Passage 3, why is the digestive process considered significant?
A
  1. A: The digestive process is crucial as it breaks down food into absorbable nutrients for the body.
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4
Q
  1. Q: According to Passage 4, what are the factors influencing Total Daily Energy Expenditure (TDEE)?
A
  1. A: TDEE is influenced by Basal Metabolic Rate (BMR), Physical Activity, and the Thermic Effect of Food, as mentioned in Passage 4.
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5
Q
  1. Q: What key aspect of nutrition does Passage 5 emphasize for athletes?
A
  1. A: Passage 5 emphasizes the importance of adequate hydration for athletes.
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6
Q
  1. Q: How does Passage 2 define macronutrients?
A
  1. A: Macronutrients, according to Passage 2, are nutrients required by the body in relatively large amounts, such as carbohydrates and proteins.
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7
Q
  1. Q: What is the recommended approach to pregame meals, according to Passage 5?
A
  1. A: The pregame meal should include fluids and easily digestible carbohydrate-rich foods, providing 300 to 800 kcalories, ending 1 to 4 hours before competition.
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8
Q
  1. Q: In Passage 3, what role does the digestive process play in nutrient absorption?
A
  1. A: The digestive process breaks down food into absorbable nutrients, facilitating their absorption in the body.
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9
Q
  1. Q: According to Passage 4, what is the Thermic Effect of Food?
A
  1. A: The Thermic Effect of Food, mentioned in Passage 4, refers to the energy required for digestion, absorption, and metabolism of nutrients.
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10
Q
  1. Q: Why does Passage 5 mention the significance of maintaining glycogen stores?
A
  1. A: Passage 5 states that full glycogen stores are critical for athletes and highly active individuals.
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11
Q
  1. Q: Name one potentially harmful effect of caffeine, as discussed in Passage 5.
A
  1. A: Passage 5 mentions that excessive caffeine intake can lead to adverse effects such as stomach upset, nervousness, irritability, headaches, and diarrhea.
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12
Q
  1. Q: What is the primary message regarding dietary supplements in Passage 5?
A
  1. A: Passage 5 emphasizes that athletes should be cautious with dietary supplements, as many lack scientific support and may even pose health risks.
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13
Q
  1. Q: According to Passage 3, what is the role of enzymes in digestion?
A
  1. A: Passage 3 highlights that enzymes facilitate the breakdown of complex molecules during digestion.
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14
Q
  1. Q: How does Passage 2 differentiate between carbohydrates and proteins?
A
  1. A: Passage 2 distinguishes carbohydrates as a primary source of energy and proteins as essential for growth, maintenance, and repair.
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15
Q
  1. Q: What is the recommended timing for the pregame meal, as mentioned in Passage 5?
A
  1. A: Passage 5 suggests the pregame meal should end 1 to 4 hours before competition to allow for proper digestion.
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16
Q
  1. Q: Why is nutrient density emphasized in Passage 5?
A
  1. A: Nutrient-dense foods, as discussed in Passage 5, provide essential vitamins and minerals in proportion to the energy they offer.
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17
Q
  1. Q: According to Passage 4, what role does physical activity play in Total Daily Energy Expenditure?
A
  1. A: Physical activity contributes significantly to Total Daily Energy Expenditure, as outlined in Passage 4.
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18
Q
  1. Q: In Passage 5, what are the potential drawbacks of overhydration for athletes?
A
  1. A: Overhydration can lead to hyponatremia, as mentioned in Passage 5.
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19
Q
  1. Q: What is the primary focus of Passage 4?
A
  1. A: Passage 4 discusses the factors influencing Total Daily Energy Expenditure (TDEE) and the concept of energy balance.
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20
Q
  1. Q: According to Passage 5, what are the key considerations for pregame meals?
A
  1. A: Pregame meals, as per Passage 5, should include plenty of fluids, be light and easily digestible, and primarily consist of carbohydrate-rich foods.
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21
Q
  1. describe the process of nutrient absorption?**
A
  1. A: Passage 3 describes nutrient absorption as the uptake of digested nutrients by cells, making them available for various physiological functions.
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22
Q
  1. Q: What role does glycogen play in athletic performance, according to Passage 5?
A
  1. A: Passage 5 emphasizes that full glycogen stores are critical for athletes to support high-intensity activities.
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23
Q
  1. Q: According to Passage 2, why are micronutrients essential for the body?
A

*A: Passage 2 notes that micronutrients areessential for various physiological processes, supporting overall health.**

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24
Q
  1. Q: What is the primary concern mentioned in Passage 5 regarding dietary supplements?
A
  1. A: Passage 5 expresses concern that some supplements may be contaminated with illegal substances, posing health risks to athletes.
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25
Q
  1. Q: In Passage 4, how does Basal Metabolic Rate (BMR) contribute to Total Daily Energy Expenditure?
A
  1. A: BMR, discussed in Passage 4, represents the energy expended at rest and contributes significantly to Total Daily Energy Expenditure.
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26
Q

What is the primary function of iron in the body?

A

Serves as a component of hemoglobin for oxygen transport.

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27
Q

What are the symptoms of iron deficiency anemia?

A

Fatigue, weakness, and pallor.

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28
Q

Why do vegetarians need more iron in their diet compared to non-vegetarians?

A

Lower bioavailability of iron in plant-based diets.

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29
Q

What is the role of vitamin C in iron absorption?

A

Converts insoluble ferric iron to the more soluble ferrous iron, enhancing absorption.

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30
Q

How does iron contamination occur from nonfood sources?

A

Cooking in iron cookware, influenced by acidity and cooking time.

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31
Q

What are the potential symptoms of iron toxicity?

A

GI distress, constipation, nausea, vomiting, and diarrhea.

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32
Q

Why is iron supplementation a common cause of accidental poisoning in young children?

A

Ingestion of supplements leading to symptoms like nausea, vomiting, and potential death.

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33
Q

How does iron fortification contribute to reducing iron deficiency?

A

Effectively reduces iron deficiency despite being nonheme iron.

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34
Q

How can individuals maximize zinc absorption from the diet?

A

Consuming foods high in zinc, such as shellfish, meats, poultry, milk, and cheese.

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35
Q

What are the roles of zinc in the body?

A

Supports metabolic reactions, immune function, cell membranes, DNA, growth, development, hormones, and enzymes.

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36
Q

How does iodine deficiency impact thyroid hormone production?

A

Leads to diminished production, causing goiter (enlarged thyroid gland).

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37
Q

What are some functions of thyroid hormones regulated by iodine?

A

Body temperature, metabolic rate, reproduction, growth, blood cell production, nerve and muscle function, and oxygen use in cells.

38
Q

What can happen in the case of chronic zinc deficiency?

A

Damage to central nervous system, impaired motor development and cognitive performance, disturbed thyroid function, altered taste, loss of appetite, and slow wound healing.

39
Q

What is the UL for zinc intake, and what potential issue does it address?

A

40 milligrams in adults, addressing potential interference with copper metabolism, which can lead to heart muscle degeneration in animals.

40
Q

How can zinc lozenges potentially affect the duration of common cold symptoms?

A

May shorten the duration (not severity) of symptoms, depending on formulation and high doses taken within 24 hours of symptom onset.

41
Q

What are the two major stages critical in the development of osteoporosis?

A

The bone-acquiring stage of childhood and adolescence and the bone-losing decades of late adulthood, especially in women after menopause.

42
Q

How does bone loss exceed bone formation as people age?

A

The cells that build bone become less active, while those that dismantle bone continue working.

43
Q

What is the significance of maximizing bone mass during the first three decades of life?

A

It helps curtail bone loss in later years, giving an advantage to those with denser bones formed during their youth.

44
Q

Why is dietary calcium crucial for bone health in youth?

A

Calcium builds strong bones, and inadequate intake leads the body to depend on bone, causing bone loss.

45
Q

How do hormones like estrogen and testosterone influence bone and calcium metabolism?

A

Changes in these hormones with age accelerate bone loss, contributing to inefficient bone remodeling.

46
Q

What role do sex hormones play in osteoporosis, and why do women generally face greater losses than men?

A

Sex hormones regulate bone turnover, and women experience rapid bone losses, especially after menopause due to diminished estrogen.

47
Q

How does physical activity support bone growth during adolescence?

A

Active adolescents develop stronger bones, as muscle and bone strength are interconnected.

48
Q

Why is weight training recommended for maintaining bone density in adults, even past menopause?

A

It improves bone density, countering the common trend of bone loss in women during this period.

49
Q

What are the effects of smoking on bone density, and can they be reversed?

A

Smokers have less dense bones, but quitting smoking can lead to recovery in bone density over time.

50
Q

How does alcohol contribute to osteoporosis?

A

Excessive alcohol intake leads to fluid excretion, hormonal imbalance, slowed bone formation, increased bone breakdown, and a higher risk of falling.

51
Q

What role does genetics play in osteoporosis, and how can outside factors influence its impact?

A

Genetics influence peak bone mass and later bone loss, with diet and physical activity maximizing potential and factors like alcohol accelerating losses.

52
Q

Why is an adequate calcium intake alone insufficient for protecting against bone fractures in older adults?

A

Bone strength later in life primarily depends on how well bones were built during childhood and adolescence.

53
Q

What nutrients, besides calcium, support bone health?

A

Adequate protein, vitamin D, vitamin K, vitamin C, magnesium, potassium, vitamin A, carotenoids, and omega-3 fatty acids contribute to bone health.

54
Q

How does a well-balanced diet impact bone health, and what dietary elements are associated with bone losses?

A

A well-balanced diet, rich in fruits, vegetables, and whole grains, supports bone health, while excessive salt, sodas, and certain foods are associated with bone losses.

55
Q

What is emphasized regarding the use of calcium supplements for bone health?

A

While calcium from foods is preferable, supplements may help if dietary intake is insufficient, but caution is advised due to potential risks, and consulting a healthcare professional is recommended.

56
Q

What factors should be considered when selecting a calcium supplement?

A

Calcium content, type of supplement (carbonate, citrate, etc.), absorption rate, and the timing of intake, considering interactions with other nutrients.

57
Q

Why are risk factors like age, gender, and genetics beyond a person’s control, and what prevention strategies are effective?

A

These factors are inherent, but ensuring optimal peak bone mass during youth, maintaining healthy habits, and consulting with a physician for tests and appropriate interventions can minimize bone loss.

58
Q

Define the two subgroups of the vitamin E family and their respective members.

A

e two subgroups are tocopherols and tocotrienols, each with four members (alpha, beta, gamma, and delta). Tocopherols have a saturated side chain, while tocotrienols have an unsaturated side chain.

59
Q

Why is alpha-tocopherol the only member of the vitamin E family maintained in the body for meeting its needs?

A

Alpha-tocopherol is the only member maintained because others are not converted to it in the body, nor are they recognized by its transport protein.

60
Q

What role does vitamin E play in preventing erythrocyte hemolysis, and in what condition is this deficiency observed?

A

Vitamin E prevents erythrocyte hemolysis by protecting polyunsaturated fatty acids in red blood cell membranes. Deficiency is observed in conditions of fat malabsorption, such as cystic fibrosis.

61
Q

Aside from cancer prevention, name other health conditions tocotrienols may protect against according to recent studies.

A

Tocotrienols may also protect against osteoporosis, diabetes, heart disease, and neurological disorders.

62
Q

What is the primary role of vitamin E in the body, and why has most research focused on alpha-tocopherol?

A

Vitamin E acts as an antioxidant. Most research has focused on alpha-tocopherol, as it is the form maintained in the body and recognized in the transport protein.

63
Q

Explain the potential risk associated with extremely high doses of vitamin E supplementation.

A

Extremely high doses may interfere with the blood-clotting action of vitamin K and enhance the effects of anti-blood clotting drugs, leading to hemorrhage.

64
Q

Where is vitamin E found in foods, and why is fresh food preferable as a source?

A

Vitamin E is found in vegetable oils, margarine, salad dressings, and wheat germ oil. Fresh foods are preferable because vitamin E is readily destroyed by heat and oxidation.

65
Q

Why is vitamin E intake influenced by the consumption of polyunsaturated fatty acids?

A

A person consuming large quantities of polyunsaturated fatty acids needs more vitamin E. Fortunately, vitamin E and polyunsaturated fatty acids often occur together in the same foods.

66
Q

Explain why vitamin K is named after the Danish word “koagulation.”

A

Vitamin K is named after “koagulation” because its primary action is blood clotting, a process crucial for maintaining life.

67
Q

Describe the role of vitamin K in blood clotting and its association with proteins like prothrombin.

A

Vitamin K is essential for activating proteins like prothrombin, which is a precursor of thrombin in blood clotting. Lack of these clotting factors can lead to hemorrhagic disease.

68
Q

In what circumstances does a secondary deficiency of vitamin K occur, and how can it lead to excessive bleeding?

A

A secondary deficiency occurs when fat absorption falters, reducing vitamin K absorption. Excessive bleeding happens due to the lack of vitamin K’s role in blood clotting.

69
Q

What unique challenge do newborn infants face regarding vitamin K, and how is it addressed?

A

Newborn infants have a sterile intestinal tract, and vitamin K–producing bacteria take weeks to establish. To prevent hemorrhagic disease, a single dose of vitamin K is given at birth by intramuscular injection.

70
Q

Why hasn’t a UL (Tolerable Upper Intake Level) been established for vitamin K, and what risk is associated with high vitamin K intake for those on anticoagulant drugs?

A

A UL hasn’t been established due to the rarity of toxicity. High vitamin K intake may interfere with anticoagulant drugs, potentially causing irregular blood clotting.

71
Q

What are dietary sources of vitamin K, and what is the primary form found in foods?

A

Dietary sources include leafy green vegetables, fruits like avocado and kiwi, and some vegetable oils like soybean oil. Phylloquinone (vitamin K1) is the primary form found in foods.

72
Q

How does vitamin K contribute to bone health, and what is inconclusive about vitamin K supplements in this regard?

A

Vitamin K helps synthesize bone proteins, particularly osteocalcin. Vitamin K supplements’ effectiveness in bone health is inconclusive, as research results are mixed.

73
Q

Define free radicals and explain their role in the body’s metabolic reactions.

A

Free radicals are highly reactive molecules with unpaired electrons. They are produced in the body’s metabolic reactions, particularly when oxygen reacts with compounds.

74
Q

Describe the chain reaction initiated by free radicals and how antioxidants intervene in this process.

A

Free radicals initiate a chain reaction by stealing electrons from stable compounds. Antioxidants intervene by donating their electrons, breaking the chain and neutralizing free radicals.

75
Q

What are the potential outcomes of free-radical attacks on the body, and why is controlling them essential?

A

Free-radical attacks can damage fatty acids, alter DNA, RNA, and proteins, and contribute to inflammation, diseases, and aging. Controlling them is essential as unrepaired damage accumulates.

76
Q

How do antioxidants, including vitamins and phytochemicals, defend the body against oxidative stress?

A

Antioxidants defend against oxidative stress by limiting free-radical formation, destroying free radicals, stimulating antioxidant enzyme activity, repairing oxidative damage, stimulating repair enzyme activity, and supporting a healthy immune system.

77
Q

Explain the body’s lines of defense against free-radical damage, involving enzymes and antioxidant vitamins.

A

Enzymes dependent on minerals (selenium, copper, manganese, zinc) disarm harmful oxidants. Antioxidant vitamins (E, beta-carotene, C) play roles in stopping free-radical chain reactions and protecting various tissues.

78
Q

Discuss the link between oxidative stress, antioxidants, and diseases such as cancer, arthritis, cataracts, diabetes, hypertension, and heart disease.

A

Oxidative stress may contribute to diseases, and antioxidants may offer protection. Associations exist between oxidative stress and conditions like cancer, arthritis, cataracts, diabetes, hypertension, and heart disease.

79
Q

Examine the potential protective effects of specific antioxidant vitamins against cancer and heart disease.

A

Vitamin C and E may reduce cancer risks by protecting DNA. Evidence suggests correlations between vitamin-rich diets and lower heart disease rates. However, the benefits of supplements are uncertain, and dietary patterns are emphasized.

80
Q

Discuss the importance of obtaining antioxidants from foods compared to supplements, and highlight potential risks associated with supplement use.

A

Foods offer a variety of antioxidants and nutrients. Dietary strategies, including a balanced diet, are effective in preventing diseases. Risks associated with supplement use, including potential harm at pharmacological doses, underscore the importance of obtaining antioxidants from foods.

81
Q

When do symptoms of vitamin A toxicity begin to develop?

A

Symptoms of vitamin A toxicity develop when all binding proteins are loaded, and vitamin A is free to damage cells.

82
Q

Question: Why are children more vulnerable to vitamin A toxicity?

A

Answer: Children are more vulnerable due to needing less vitamin A and being more sensitive to overdoses.

83
Q

Question: What Upper Level (UL) has been set for adults, and to which form of vitamin A does it apply?

A

Answer: The Upper Level (UL) for adults is 3000 micrograms, and it applies only to preformed vitamin A.

84
Q

Question: What are the major roles of vitamin A in the body?

A

Vitamin A regulates the expression of several hundred genes and plays key roles in promoting vision, participating in protein synthesis, cell differentiation, supporting reproduction, and regulating growth.

85
Q

Question: How does retinoic acid differ from other forms of vitamin A in terms of its functions?

A

Answer: Retinoic acid acts as a hormone, regulating cell differentiation, growth, and embryonic development. It differs from other forms in its irreversible conversion and specific function

86
Q

What indispensable roles does vitamin A play in the eye?

A

Vitamin A helps maintain the cornea and participates in the conversion of light energy into nerve impulses at the retina.

87
Q

Question: Where in the body is the majority of vitamin A found, and what functions does it serve in those areas?

A

Answer: The majority of vitamin A is found in the cells lining the body’s surfaces, particularly in epithelial tissues. It participates in protein synthesis, cell differentiation, and helps protect against damage.

88
Q

What dual roles does beta-carotene play in the body?

A

Beta-carotene serves as a vitamin A precursor and acts as an antioxidant capable of protecting the body against disease.

89
Q

How is vitamin A content expressed in foods, and what is the significance of retinol activity equivalents (RAE)?

A

Vitamin A content is expressed as retinol activity equivalents (RAE), acknowledging the less efficient absorption and conversion of beta-carotene compared to retinoids.

90
Q

Question: What are the rich sources of retinoids, and how can individuals ensure a sufficient intake of vitamin A from carotenoids?

A

Answer: Foods derived from animals, such as liver, fish liver oils, milk, and eggs, are rich sources of retinoids. Carotenoids, the precursors found in plants, are abundant in dark leafy greens and deep yellow or orange vegetables and fruits. A diet including these sources helps ensure a sufficient intake of vitamin A.