Chap 54 Chronic Kidney Disease Flashcards

1
Q

what are the only reasons in which a catheter should be used

A
  1. acute urinary retention or bladder outlet obstruction
  2. hemodynamic instability to use as an indirect measurement of CO
  3. perioperative use: (urologic surgery)
  4. open sacral or perineal wounds
  5. prolonged immobilization
  6. comfort at end of life
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2
Q

what are the four top causes of CKD

A
  1. HTN, 2. DM 3. glomerulonephritis 4. pyelonephritis
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3
Q

how does pyelonephritis present and what is the RX

A

S/S: nausea/vomiting, flank pain, fever, chills, pyuria, elevated WBC, shift to the left 6% bands.
RX: 2weeks of Abx., liberalize fluids, follow up in 2weeks for f/u culture.

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4
Q

who is at risk for pyelonephritis?

A

weakened immune system, damaged nerves in bladder, obtruction of urinary tract, prolonged use of urinary catheter, or conditions in which urine flows the opposite direction

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5
Q

what is acute glomerulonephritis and its presenting s/s

A

acute glomerulonephritis also known as acute nephritic syndrome. Manifestation is often post strep.
S/S: hematuria, edema, azotemia, RBCs in UA
cadiomegaly, pulm. edema, and neuro symptoms may be present. increases in BUN and Cr, anorexia, hypertension

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6
Q

what are the potential complications and treatment of acute glomerulonephritis/acute NEPHRITIC syndrome?

A

complications: kidney failure, hypertensive encephalopathy pulmonary edema and heart failure.

Rx: plasmapheresis, high dose steroids, and cytoxic agents

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7
Q

what are the nursing considerations in treating patients with glomerulonephritis

A
  1. mon. vs. I&O, urine characteristics
  2. daily weights at the same time
  3. limit activity
  4. Na restriction, possible K restriction
  5. monitor for complications
  6. admin diuretics
  7. initiate seizure precautions
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8
Q

how does NephROTIC syndrome present? S/S

A

NephrOTIC syndrome is a disorder that involves that podocytes and basement membrane it causes the body to excrete massive amounts of protein via the urine. This has insidious onset,
S/S: insidious onset, frothy urine, 3.5g of protein/day, increases of lipids in blood, no active sedimentation, edematous, AT-III, dereased albumin, decrease in UO,

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9
Q

what is the Rx for NephROTIC syndrome

A

fluid restriction, lasix, steriods,

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10
Q

what are the stages of CKD and their corresponding GFRs?

A

Stage Rx:
Stage 1: GFR >90
Manage risk, A1c:<7, BS<170, BP 130/90
Stage 2: GFR 60-89 (susceptible)
Protein restriction, ACE-I
Stage 3: GFR 30-59 (insufficiency)
Diuretics, phosphate binders, , calcium, bicarb, insulin, D5W, kayexelate, diet restrictions
Stage 4: GFR 15-29 (severe decreases)
Manage fluids, restrict fluids, bicarb, diuretics, insulin, d5w, kayexalate, calcium, diet restrictions, blood
ESRD: <15 Dialysis, transplant

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11
Q

what are the electrolyte, acid base, and hematologic abnormalities

A

high K, high phos, high Mg, low calcium, low RBCs, acidosis, low bicarb, high na, high water retention,

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12
Q

what are the diet and fluid recommendations before and during dialysis? in hemodialysis versus peritoneal?

A
fluid restrictions: 
BEFORE: 500-800mL + UO
During: 700-1100mL + UO
Hemodialysis: 700-1100mL + UO
Peritoneal: A little more liberal
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13
Q

how are the electrolyte abnormalities addressed/treated in CKD?

A

hyperkalemia: kayexalate, insulin, D5W, bicarb, diet restrictions
hypernatremia: dietary restrictions
hyperphosphatemia: dietary restrictions
hypocalcemia: diet,
hypermagnesemia: dialysis

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14
Q

how does uremia present?

A
Nausea.
    Vomiting.
    Fatigue.
    Anorexia.
    Weight loss.
    Muscle cramps.
    Pruritus.
    Change in mental status.
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15
Q

which medications are more easily dialyzed

A

water soluble drugs that are NOT bound to protein. drugs with LMW: tylenol, aspirin, captopril, reglan, protamine, theophylline

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16
Q

which drugs are poorly dialyzed?

A

Protein bound drugs: lasix, heparin, iron, nifedipine, verapamil, quinidine, propanolol, hydralazine

17
Q

what are some potential complications of an AV Graft Fistula

A

Immediately after surgery, hemorrhage, low venous flow or hematoma may occur. At a later stage, there may be complications, such as infections, the development of an aneurysm and/or false aneurysm, fistula vein stenosis, congestive heart failure, steal syndrome, ischemic neuropathy and thrombosis

18
Q

what is the cardiac-CKD connection

A

HTN and DM cause stress on both renal and cardiac systems. When the cardiac system is taxed, there is often a decrease in tissue perfusion, which leads to acute kidney disease. When kidney function is impaired, the RAAS is turned on, which in turn increases demand on the cardiac system.