Ch 33 CAD, ACS, MI (Exam 2) Flashcards

1
Q

What is the main cause of coronary artery disease?

A

Atherosclerosis

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2
Q

What does atherosclerosis lead to?

A

Endothelial injury/inflammation

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3
Q

Causes of endothelial injury? (5)

A
  • hypertension
  • smoking
  • infection
  • diabetes
  • hyperlipidemia (high serum lipids)
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4
Q

Diagnostics for coronary artery disease? (2)

A
  • cardiac markers (high troponin)

- echocardiogram

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5
Q

What are the 4 steps of pathophysiology of CAD?

A
  1. chronic endothelial injury
  2. fatty streak and lipid core formation
  3. fibrous plaque covers the lipid core (reduces blood flow, fissures/scars develop)
  4. complicated lesion: plaque ruptures and thrombus forms (causing narrowing/total occlusion)
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6
Q

Complicated lesions lead to thrombus formation due to which mechanism?

A

Platelet accumulation

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7
Q

Prevention of thrombus formation (3)

A
  • avoid vessel injury (smoking cessation, lifestyle changes, exercise)
  • control platelet aggregation (daily antiplatelet therapy like aspirin)
  • control lipid levels (statins, niacin) if diet/exercise is ineffective
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8
Q

The formulation of thrombus is deterred by?

A

Daily administration of baby aspirin

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9
Q

What are the non-modifiable risk factors of coronary artery disease (4)

A
  • age (>60)
  • gender (women tend to live longer)
  • ethnicity
  • genetics, family hx
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10
Q

Major modifiable risk factors of coronary artery disease (3)

A
  • hyperlipidemia (cholesterol >200)
  • hypertension (>140/90)
  • smoking cessation
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11
Q

How does smoking lead to coronary artery disease? (3)

A
  • increases catecholamines (epinephrine) which leads to vasoconstriction and increased HR
  • increases LDL levels, decreases HDL
  • directly injures the epithelium
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12
Q

Contributing modifiable risk factors (3)

A
  • metabolic syndrome (ABD GERD, prediabetes)
  • psychological states: stress/increased epinephrine, anger, depression
  • substance abuse: cocaine increases cardiac demand for oxygen
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13
Q

Ways to increased HDL (good lipids) (5)

A
  • exercise
  • eat healthy fats
  • lose weight
  • moderate alcohol intake
  • quitting smoking
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14
Q

Your patient has been identified as being at high risk for MI. Which lifestyle modification would have the most benefit to the patient?

A

Quit smoking

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15
Q

After teaching about ways to decrease risk factors for CAD, the nurse recognizes that further instruction is needed when the patient says:

A

“I would like to add weight lifting to my exercise program” (strain is contraindicated)

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16
Q

Nitroglycerin treatment for stable vs unstable angina

A
  • unstable: not effective (unstable angina creates life-long changes on ECG)
  • stable: relieved by nitroglycerin (ECG may return to normal)
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17
Q

Describe unstable angina (2)

A
  • chest pain that is new in onset and occurs at rest

- unpredictable and must be treated immediately

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18
Q

What are the effects of nitroglycerin? (3)

A
  • causes vasodilation
  • side effects: severe headaches
  • patient teaching: take with tylenol to relieve headache
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19
Q

Which actions by nitroglycerin help to reduce or eliminate chest pain? (2)

A
  • lowers preload and work of heart

- dilates coronary arteries to increase blood supply to the myocardium

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20
Q

PQRST (to assess for myocardial infarction)

A

P: precipitating events (arguing, exercise)

Q: quality of pain (pressure, dull, ache, tight)

R: region (location) and radiation

S: severity of pain (0-10)

T: time (when did it begin?)

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21
Q

Nutritional therapy (2)

A
  • low sodium diet (prevent fluid retention)

- for CAD: limit saturated fats and cholesterol and more complex carbs (whole grains, fruits, vegetables) and fiber

22
Q

What is a myocardial infarction?

A

abrupt stoppage of blood flow through the coronary artery, causing irreversible cell death

23
Q

S/S of unstable angina

A

severe chest pain that is NOT relieved by rest/position change or nitroglycerin administration

24
Q

S/S of stable angina (2)

A
  • pain during activity/exercise but SUBSIDES AT REST

- pain is described as heavy, pressured, tight/constricted, burning, crushing (“elephant on chest”)

25
What are complications of MI? (2)
- infarction of papillary muscle/rupture | - acute pericarditis
26
Diagnostics for myocardial infarction (stemi vs nstemi)
- STEMI: coronary angiography | - NSTEMI: high troponin levels (contraindicated to do coronary angiography)
27
You patient reports experiencing chest pain during sex that subsides at rest. He is probably experiencing?
Stable angina
28
Your patient's heart is getting less oxygen due to narrowing/blockage of the coronary artery. What symptom will the patient most likely present?
Chest pain or pressure
29
A complication of infarction of papillary muscle from MI can be determined by?
Heart sounds (murmur)
30
Treatment for acute angina
short-acting nitrates are the first-line therapy
31
Describe chronic stable angina (4)
- chest pain that occurs over a long period - patient may deny pain at first - anginal pain lasts only a few mins and subsides when the precipitating factor is relieved - pain at rest is UNUSUAL
32
Medication teatment for chronic stable angina
focused on decreasing oxygen demand or increasing oxygen supply (medications: nitrates, ACE inhibitors, beta-blockers, calcium channel blockers)
33
Describe Prinzmetal's angina, a type of chronic stable angina (4)
- rare form of angina that occurs AT REST in response to a spasm of a major coronary artery - may be seen in patients with a hx of migraine headaches and Raynaud's phenomenon - precipitating factors: smoking, alcohol, amphetamines, cocaine use - pain may be relieved by moderate exercise or spontaneously
34
Treatment for Prinzmetal's angina
calcium channel blockers and/or nitrates (to decrease afterload)
35
What is the goal of interprofessional care for chronic stable angina?
decrease O2 demand or increase O2 supply
36
Function of short-acting nitrates
decreases preload and afterload
37
Nitroglycerin - route - adminsitration - interventions
- sublingually or by spray (faster) - if no relief in 5 mins, call EMS; if some relief after 5 mins, repeat every 3 mins for a max of 3 doses - sit patient down to relax them, elevate HOB to improve airway and decrease O2 demand
38
function of long-acting nitrates
reduces angina incidence
39
ACE inhibitors and angiotensin blocker functions
reduces oxygen demand by lowering HR, afterload, and BP (especially useful after an MI)
40
Purpose of induced hypothermia
might prevent hypoxic brain damage during cardiac arrest
41
What can be delegated to UAP during MI care? (6)
- empty urinary catheter - use of incentive spirometer and deep breathing exercises - observe patient and report to nurse - monitor SCDs for DVT prevention - drain fluid - apply water soluble protectant to nares and lips
42
Delegation to LVN (4)
- give medications - take vitals - basic skills (applying oxygen and inserting urinary catheter) - cannot do initial patient teaching (can reinforce)
43
What is acute coronary syndrome
Deterioration of plaque, leading to rupture and platelet aggregation, which leads to thrombus formation
44
Initial interventions for ACS (5)
- 12-lead ECG - upright position: stop movement, provide comfort, resting position - create IV access - administer statin - MONA **requires doctor's order except oxygen**
45
MONA for ACS stands for?
M: morphine O: oxygen (o2 sat > 92%) N: nitroglycerin (SL) A: aspirin (chewable)
46
Therapy for NSTEMI
Reperfusion therapy
47
Emergency PCI (percutaneous coronary intervention) stent is indicated for? - goal - consists of
- indicated for: confirmed STEMI - goal: get patient to cardiac cath lab within 90 mins of presentation - consists of balloon angioplasty and stents
48
Thrombolytic therapy is only indicated for patients with? - give IV within ______ - contraindications (3)
- indicated for patients with STEMI - given IV within 30 mins of arrival with presenting STEMI - assess contraindications: recent surgeries, peptic ulcer disease, recent bleeds
49
Coronary revascularization with coronary artery bypass graft (CABG) is recommended for? When is it provided? Post op care includes? (6)
- patients who fail medication management - provided in the ICU for the first 24-36 hours (ongoing monitoring of ECG and hemodynamic status) - post-op care focuses on monitoring for dysrhythmias, providing wound care, managing pain, preventing DVTs/bleeding, atelectasis, pneumonia
50
Drug therapy for acute coronary syndrome?
- dual antiplatelet (aspirin) therapy should be continued for 1 year after an MI - stool softeners to avoid straining
51
Sudden cardiac death (2)
- abrupt disruption in cardiac function, leading to loss of CO and cerebral blood flow - most commonly caused by: ventricular dysrhythmias (vfib and vtach)