ch 25 Respiratory assessment Flashcards
is gas exchange
respiratory system
transfer of oxygen (O2) and carbon dioxide (CO2) between the atmosphere and blood.
respiratory system
the upper respiratory tract and the lower respiratory tract
respiratory system is divided into 2 parts:
the nose, mouth, pharynx, epiglottis, larynx, and trachea.
upper respiratory tract includes
the nose
Air enters the respiratory tract through
bone and cartilage
nose is made of
divided into 2 nares by the nasal septum
nose
inside of the nose is shaped into 3 passages by projections called
turbinates
increase the surface area of the nasal mucosa that warms and moistens the air as it enters the nose
turbinates
with the pharynx
nasal cavity connects
the nasopharynx, oropharynx, and laryngopharynx
nasal cavity connects with the pharynx.
- It is a tubular passageway that is subdivided into 3 parts:
to protect the lower airway by warming and humidifying air and filtering small particles before air enters the lungs.
nose functions
, found within the mucosa of the upper part of the nasal cavity, is responsible for the sense of smell.
olfactory nerve
Air moves through the oropharynx to the laryngopharynx. It then travels through the epiglottis to the larynx before moving into the trachea.
Route of air through nose to ody
is a small flap behind the tongue that closes over the larynx during swallowing
epiglottis
prevents solids and liquids from entering the lungs.
epiglottis function
are in the larynx
vocal cords location
(the opening between the vocal cords) and into the trachea.
air passes through the glottis
U-Shaped cartilages keep the trachea open but allow the adjacent esophagus to expand for swallowing.
trachea
into the right and left mainstem bronchi at a point called the carina.
trachea bifurcates
is located at the angle of Louis, which is at the level of the 4th and 5th thoracic vertebrae.
carina
this area during suctioning causes vigorous coughing
Stimulation of carnia
Once air passes the carina, it is in the
considered lower respiratory tract
bronchi, bronchioles, alveolar ducts, and alveoli
lower respiratory tract consists
Except for the right and left mainstem bronchi,
all lower airway structures are found within the lungs.
-upper, middle, and lower
right lung is divided into 3 lobes
-upper and lower
left lung into 2 lobes
mainstem bronchi, pulmonary vessels, and nerves enter the lungs through a slit called the
hilus
is shorter, wider, and straighter than the left mainstem bronchus
right mainstem bronchus
is shorter, wider, and straighter than the left mainstem bronchus.
right mainstem bronchus vs left
right mainstem bronchus is shorter, wider, and straighter than the left mainstem bronchus.
aspiration is more likely to occur in the right lung than in the left lung.
several times to form the lobar, segmental, and subsegmental bronchi.Further divisions form the bronchioles.
mainstem bronchi subdivide
the respiratory bronchioles.
most distant bronchioles are
are encircled by smooth muscles that constrict and dilate in response to variousstimuli.
bronchioles
to a decrease or increase in the diameter of the airways caused by contraction or relaxation of these muscles
bronchoconstriction and bronchodilation refer
act as a pathway to conduct gases to and from the alveoli
trachea and bronchi
volume of air in the trachea and bronchi is called the
-This air does not take part in gas exchange.
anatomic dead space (VD)
is about 500mL (in a 150-lb man). Of each 500 mL inhaled, about 150mL is VD.
adults, normal tidal volume (VT), or volume of air exchanged with each breath,
are small sacs in the lungs that are the primary site of gas exchange for O2 and CO2
Alveoli
They allow movement of air from alveolus to alveolus
alveoli are interconnected by pores of Kohn
air movement through these pores and helps move mucus out of the respiratory bronchioles.
Deep breathing promotes
can also move through these pores, spreading infection to previously uninfected areas.
Bacteria (alveoli)
of about 2500 mL with a surface area for gas exchange that is about the size of a tennis court.
Alveoli have a total volume
, where the alveoli come in contact with pulmonary capillaries
Gases are exchanged across the alveolar-capillary membrane
excess fluid fills the interstitial space and alveoli, markedly reducing gas exchange.
pulmonary edema
is a lipoprotein that lowers the surface tension in the alveoli
-prevents alveoli collapse
Surfactant
reduces the amount of pressure needed to inflate the alveoli and makes them less likely to collapse
Surfactant
each person takes a slightly larger breath, termed a sigh, after every 5 or 6 breaths. This sigh stretches the alveoli and promotes surfactant secretion.
sigh
refers to collapsed, airless alveoli.
atelectasis
because of the effects of anesthesia, decreased mobility, and pain, which can alter breathing and lung expansion.
postoperative patient is at risk for atelectasis
, lack of surfactant contributes to widespread atelectasis and collapse of lung tissue
acute respiratory distress syndrome (ARDS)
pulmonary and bronchial
lungs have 2 different types of circulation:
provides the lungs with blood that takes part in gas exchange
Pulmonary circulation
receives deoxygenated blood from the right ventricle of the heart and delivers it to pulmonary capillaries that lie directly alongside the alveoli.
pathology of pulmonary artery
starts with the bronchial arteries, which arise from the thoracic aorta
Bronchial circulation
but provides O2 to the bronchi and other lung tissues. Deoxygenated blood returns from the bronchial circulation through the azygos vein into the superior vena cava.1
Bronchial circulation does not take part in gas exchange
is shaped, supported, and protected by 24 ribs (12 on each side
chest wall (description)
which consists of the ribs and sternum, protect the lungs and the heart from injury.
thoracic cage function
is the space in the middle of the thoracic cavity.
mediastinum
It contains the major organs of the chest, including the heart, aorta, and esophagus.
mediastinum consists
chest cavity is lined with a membrane called the
parietal pleura
lungs are lined with a membrane called the
visceral pleura
join to form one continuous membrane.
parietal and visceral pleurae join
does not have any sensory (pain) fibers or nerve endings.
visceral pleura consists
This is why irritation or inflammation of the parietal pleura can cause pain with each breath.
parietal pleura has pain fibers.
is the space between the pleural layers
-Normally this space contains 10 to 20 mL of fluid.
intrapleural space
(1) it provides lubrication, allowing the pleural layers to slide over each other during breathing
(2) it increases unity between the pleural layers. This promotes expansion of the pleurae and lungs during inspiration.
intrapleural space (fluid ) serves 2 purposes:
via lymphatic circulation
Fluid drains from the pleural space
Several pathologic conditions may cause the accumulation of greater amounts of fluid, termed
pathology of pleural effusion.
-Pleural fluid may accumulate because of blockage of lymphatic drainage (e.g., from cancer)
- an imbalance between intravascular and oncotic fluid pressures, as in heart failure. —–Purulent pleural fluid with bacterial infection is called empyema.
cause of pleural effusion.
Purulent pleural fluid with bacterial infection is called
empyema.
is the major muscle of respiration
Diaphragm
, moves downward, and increases intrathoracic volume.
During inspiration the diaphragm contracts
At the same time, the internal intercostals relax and the external intercostal muscles contract. This increases the lateral and anteroposterior (AP) dimension of the chest
During inspiration the diaphragm contracts
, each innervated by the right and left phrenic nerves
diaphragm is made up of 2 hemidiaphragms
the spinal cord between C3 and C5, the 3rd and 5th cervical vertebrae
phrenic nerves arise from
in hemidiaphragm paralysis on the side of the injury. Complete spinal cord injuries above the level of C3 result in total diaphragm paralysis and dependence on a mechanical ventilator.
Injury to the phrenic nerve results
refers to the process of obtaining O2 from the atmospheric air and making it available to the organs and tissues of the body
Oxygenation
by partial pressure of O2 in arterial blood (PaO2), arterial O2 saturation (SaO2)
lungs’ ability to oxygenate arterial blood adequately is evaluated
dissolved O2 and hemoglobin-bound O2
O2 is carried in the blood in 2 forms:
the amount of O2 dissolved in the plasma
PaO2 represents
is the amount of O2 bound to hemoglobin in comparison with the amount of O2 the hemoglobin can carry.
SaO2 (defined)
-Unexplained apprehension
-Unexplained restlessness or irritability
-Unexplained confusion or lethargy (can be early or late)
Early signs of inadequate Oxygen (CNS)
-Combativeness
-Coma
Late signs of inadequate Oxygen (CNS)
-Tachypnea
Dyspnea on exertion
Early signs of inadequate Oxygen (respiratory)
-Dyspnea at rest
-Use of accessory muscles
-Retraction of intercostal spaces on inspiration
-Pause for breath between sentences, words
Late signs of inadequate Oxygen (respiratory)
-Tachycardia
-Mild hypertension
-Dysrhythmias (can be early or late)
Early signs of inadequate Oxygen (cardiovascular)
-Hypotension
-Cyanosis
-Cool, clammy skin
Late signs of inadequate Oxygen (cardiovascular)
-Diaphoresis
-Decreased urine output
-Unexplained fatigue
can be early or late signs of inadequate Oxygen
involves inspiration, or inhalation (movement of air into the lungs), and expiration, or exhalation (movement of air out of the lungs)
Ventilation
from an area of higher pressure (atmospheric) to one of lower pressure (intrathoracic).
Gas(o2 & Co2) flows
occurs, neck and shoulder muscles, as well as other accessory muscles of respiration, can aid the effort
dyspnea (shortness of breath)
is passive
expiration
is the tendency for the lungs to return to their original size after being stretched or expanded.
Elastic recoil
is due to the elastin fibers found in the alveolar walls and surrounding the bronchioles and capillaries.
elasticity of lung tissue
allows the chest to passively decrease in size (volume). When intrathoracic pressure rises, air moves out of the lun
elastic recoil of the chest wall and lungs
expiration to become an active, labored process
Exacerbations of asthma or chronic obstructive pulmonary disease (COPD) cause
Abdominal, intercostal, and accessory muscles (e.g., scalene, trapezius) help expel air during labored breathing.
body parts help expel air during labored breathing
is a measure of the ease of expansion of the lungs
Compliance (distensibility)
-This is a product of the elasticity of the lungs and elastic recoil of the chest wall
Compliance (distensibility)
, it is harder for the lungs to inflate
compliance is decreased
when there is destruction of alveolar walls and loss of tissue elasticity, as in COPD.
Compliance increases
occurs with conditions that increase fluid in the lungs (e.g., pulmonary edema, ARDS, pneumonia), make lung tissue less elastic or distensible (e.g., pulmonary fibrosis, sarcoidosis), or restrict lung movement (e.g., pleural effusion)
cause of compliance when it’s decreased
refers to any obstacle to airflow during inspiration and/or expiration
resistance
is changes in the diameter (size) of the airways
main factor affecting airway resistance
patient with an acute asthma attack has narrowed airways, resulting in
- presence of secretions in the bronchi
example of increased resistance.
decreases resistance by increasing the diameter of the bronchi, promoting air entry.
bronchodilators
Changes in compliance and/or resistance can seriously
affect both oxygenation and ventilation.
responds to chemical and mechanical signals
Located in the brainstem, the respiratory center (the medulla)
sends impulses to the respiratory muscles through the spinal cord and phrenic nerves.
medulla (function)
is a receptor that responds to a change in the chemical composition (PaCO2 and pH) of the fluid around it
chemoreceptor
are found in the medulla.
Central chemoreceptors (located)
They respond to changes in the hydrogen ion (H+) concentration.
-An increase in the H+ concentration (acidosis) causes the medulla to increase the respiratory rate and VT.
-A decrease in H+ concentration (alkalosis) has the opposite effect.
Central chemoreceptors (function)
causes the medulla to increase the respiratory rate and VT.
An increase in the H+ concentration (acidosis) // chemoreceptors
has the opposite effect
A decrease in H+ concentration (alkalosis) // chemoreceptors
primarily by their effect on the pH of the cerebrospinal fluid.
Changes in PaCO2 regulate ventilation
This lowers the cerebrospinal fluid pH and stimulates an increase in respiratory rate.
PaCO2 level is increased, more CO2 is available to combine with H2O and form carbonic acid (H2CO3).
are found in the carotid bodies at the bifurcation of the common carotid arteries and in the aortic bodies above and below the aortic arch.
Peripheral chemoreceptors
and may result in chronically elevated PaCO2 levels.
- largely because of a hypoxic drive from the peripheral chemoreceptors
COPD change lung function
are found in the conducting upper airways, chest wall, diaphragm, and capillaries of the alveoli.
Mechanical receptors (location)
They are stimulated by a variety of physiologic factors, such as irritants, muscle stretching, and alveolar wall distortion.
Mechanical receptors (stimulated)
are irritant, stretch, and juxtacapillary (J) receptors
3 major types of mechanical receptors
are found in the conducting airways.
Irritant receptors (location)//(type of mechanical receptors)
sensitive to inhaled particles and aerosols and, when stimulated, initiate the cough reflex.
Irritant receptors (effect)//(type of mechanical receptors)
, in the smooth muscle of the airways, aid in the control of respiration.
Signals from stretch receptors//(type of mechanical receptors)
lungs inflate, stretch receptors activate the inspiratory center to inhibit further lung expansion. This is called the Hering-Breuer reflex, and it prevents overdistention of the lungs.
stretch receptors (function)// (type of mechanical receptors)
prevents overdistention of the lungs.
Hering-Breuer reflex (from stretch receptor)