Ch 19 postoperative Flashcards
immediately after surgery and continues until the patient is discharged from medical care
postoperative period begins
in a postanesthesia care unit (PACU)
patient’s immediate recovery period is managed
the anesthesia care provider (ACP), OR nurse, and PACU nurse.
patient’s admission to the PACU is a joint effort among
postanesthesia care
3 phases of
their condition and the type of anesthesia they received.
patients move through the phases of care is determined by
is stable and recovering well, the patient may rapidly progress through Phase I to either Phase II care or an inpatient unit.
Phase I care on admission to the PACU
accelerated progress is called rapid postanesthesia care unit progression (RPP)
(in phase 1) patient may rapidly progress through Phase I to either Phase II care or an inpatient unit AKA
which involves admitting ambulatory surgery patients directly to Phase II care.
Another accelerated system of care is fast-tracking,
• Care during the immediate postanesthesia period
• ECG and more intense monitoring (e.g., arterial BP monitoring, mechanical ventilation)
Goal: Prepare patient for transfer to Phase II level of care, an inpatient unit, or intensive care setting
Phase I
• Ambulatory surgery patients
• Fast-tracking (i.e., patients who have bypassed Phase I level of care)
-Goal: Prepare patient for transfer to extended observation, home, or extended care facility
-Extended Observation
• Extended care or observation after transfer/discharge from Phase I or Phase II levels of care
-Goal: Prepare patient for self-care
Phase II
• Various levels of care offered in the same environment
Blended Levels of Care
detect respiratory depression in high-risk patients.
transcutaneous carbon dioxide (PtcCO2) and end-tidal CO2(PetCO2) (capnography) monitoring are used to
can help you detect respiratory distress early
Volumetric capnography and acoustic respiratory rate monitoring
explain all activities to the patient from the time of admission to the PACU.
hearing is the first sense to return in the unconscious patient,
sensory and motor blockade may still be present and you should assess dermatome levels
patient received a regional anesthetic (e.g., spinal, epidural),
sensory and motor function first returns distal to the site where the anesthetic was given. This means the areas near the site of injections are the last to recover.
recovery from regional anesthesia,
In the immediate postanesthesia period the most common causes of airway compromise include obstruction, hypoxemia, and hypoventilation
(postanesthesia) most common causes of airway compromise include
(1) have had general anesthesia; (2) are older than 55 years of age; (3) have a history of tobacco use; (4) have preexisting lung disease and/or sleep-disordered breathing; (5) are obese; (6) have co-morbidities (e.g., renal disease, diabetes, hypertension); or (7) have undergone airway, thoracic, or abdominal surgery.
Patients at high risk include those who
and in the immediate postoperative period. High-risk patients should be monitored in a critical care or postanesthesia care unit.
Pulmonary complications pose the greatest risk to patients in the postanesthesia period
is often caused by the patient’s tongue blocking the airway
Airway obstruction
base of the tongue falls backward against the soft palate and occludes the pharynx. It is most pronounced in the supine position and in the patient who is extremely sleepy after surgery.
Airway obstruction pathophysiology
, a partial pressure of arterial oxygen (PaO2) less than 60 mm Hg, is characterized by a variety of nonspecific clinical signs and symptoms, ranging from agitation to somnolence, hypertension to hypotension, and tachycardia to bradycardia. Pulse oximetry will show low O2 saturation (less than 92%).
Hypoxemia
is atelectasis
most common cause of hypoxemia after surgery
may be the result of bronchial obstruction caused by retained secretions, decreased respiratory excursion, or general anesthesia.
Atelectasis (alveolar collapse)
when mucus blocks bronchioles or there is not enough alveolar surfactant (substance that holds the alveoli open)
Atelectasis occurs
include pulmonary edema, pulmonary embolism (PE), aspiration, and bronchospasm.
Other causes of hypoxemia
fluid overload, heart failure, prolonged airway obstruction, sepsis, or aspiration.
pulmonary edema. It may be the caused by
of gastric contents into the lungs.
After surgery patients are at risk for aspiration
anesthesia depresses the respiratory protective airway reflexes. Gastric aspiration is a potentially serious emergency. It may result in laryngospasm, pneumonia, and pulmonary edema
risk for aspiration of gastric contents into the lungs. This can occur because
is the result of an increase in bronchial smooth muscle tone with resulting closure of small airways.
Bronchospasm
Airway edema develops, causing secretions to build up in the airway. The patient will have wheezing, dyspnea, use of accessory muscles, hypoxemia, and tachypnea.
Bronchospasm results
aspiration, endotracheal intubation, pharyngeal suctioning, or an allergic response.
Bronchospasm may be due to
more often in patients with a history of smoking, asthma, and chronic obstructive pulmonary disease (COPD).
Bronchospasm occurs with ppl having history of
,, is characterized by a decreased respiratory rate or effort, hypoxemia, and increasing hypercapnia (increasing PaCO2).
Hypoventilation( a common complication in the PACU)
result from depression of the central respiratory drive (from anesthesia or use of opioids), poor respiratory muscle tone (from neuromuscular blockade or disease), or a combination ofboth.
Hypoventilation may result from
are atelectasis and pneumonia, especially in patients with co-morbidities (e.g., OSA, COPD, heart failure) and after abdominal and thoracic surgery
Common causes of respiratory problems
hypoventilation, immobility and bed rest, ineffective coughing, and history of tobacco use.
development of mucous plugs and decreased surfactant production is directly related to
the respiratory passages have been irritated by heavy smoking, COPD, pulmonary infection, or dry mucous membranes that occurs with intubation, inhalation anesthesia, and dehydration.
Increased bronchial secretions occur when
pneumonia.
Without intervention, atelectasis can progress to
Allergic drug reaction
Mechanical irritation from intubation
Fluid overload
Laryngeal edema (airway obstruction)
Irritation from endotracheal tube, anesthetic gases, or gastric aspiration
Most likely to occur after removal of endotracheal tube
Laryngospasm
Secretion stimulation by anesthetic agents
Dehydration of secretions
Retained thick secretions
Muscular flaccidity associated with ↓ consciousness and muscle relaxants
Tongue falling back
Inhalation of gastric contents into lungs
Aspiration (hypoxemia)
Bronchial obstruction caused by retained secretions or ↓ lung volumes
Atelectasis (hypoxemia)
↑ Smooth muscle tone with closure of small airways
Bronchospasm (hypoxemia)
Fluid overload
↑ Hydrostatic pressure
↓ Interstitial pressure
↑ Capillary permeability
Pulmonary edema (hypoxemia)
Thrombus dislodged from peripheral venous system and lodged in pulmonary arterial system
Pulmonary embolism (hypoxemia)
Medullary depression from anesthetics, opioids, sedatives
Depressed central respiratory drive (Hypoventilation)
Tight casts, dressings, abdominal binders
Positioning and obesity preventing lung expansion
Mechanical restriction (Hypoventilation)
Shallow breathing to prevent incisional pain
Pain
Neuromuscular blockade
Neuromuscular disease
Poor respiratory muscle tone
is normally colorless and thin in consistency.
Mucus from the trachea and throat
normally thick with a pale, yellow tinge
Sputum from the lungs and bronchi is
a respiratory infection.
Changes in sputum (e.g., color) may indicate
Position the unconscious patient in a lateral “recovery” position
Postoperative Patient Positioning
position maximizes expansion of the thorax by decreasing the pressure of the abdominal contents on the diaphragm.
Once conscious, place the patient in a supine position with the head of the bed elevated
, deep breathing, coughing, and use of an incentive spirometer help prevent alveolar collapse and move respiratory secretions to larger airway passages for expectoration
Once the patient is more awake
requires the patient to inhale as deeply as possible and, at the peak of inspiration, hold the breath for a few seconds, and then exhale. This is followed by another deep breath and cough.
sustained maximal inspiration,
helps by giving visual feedback of respiratory effort
use of an incentive spirometer
involves inhaling slowly and deeply through the nose, holding the breath for a few seconds, and then exhaling slowly and completely through the mouth.
Diaphragmatic or abdominal breathing
is essential in mobilizing secretions
Effective coughing
and stimulates the cough reflex. Splinting an abdominal incision with a pillow or a rolled blanket supports the incision and aids in coughing and expectorating secretions
If secretions are in the respiratory tract, deep breathing often moves them up
hypotension, hypertension, and dysrhythmias
most common cardiovascular problems include
those with altered respiratory function, those with a history of cardiovascular disease, older adults, the debilitated, and the critically ill.
patients at greatest risk for altered cardiovascular function include
can cause hypoperfusion to the vital organs, especially the brain, heart, and kidneys
Hypotension
of disorientation, loss of consciousness, chest pain, and oliguria reflect hypoperfusion, hypoxemia, and the loss of physiologic compensation
Clinical signs Hypotension
is fluid and blood loss, which may lead to hypovolemic shock.
most common cause of hypotension in the PACU
in myocardial infarction, cardiac tamponade, or PE, results in an acute drop in cardiac output
Primary heart dysfunction, which may occur
of the negative chronotropic (rate of heart contraction) and negative inotropic (force of heart contraction) effects of drugs, such as β-adrenergic blockers, digoxin, or opioids. Other causes of hypotension include decreased systemic vascular resistance and dysrhythmias.
Secondary heart dysfunction occurs because
from sympathetic nervous system stimulation. This may be the result of pain, anxiety, bladder distention, or respiratory distress. Hypertension may be related to hypothermia and preexisting hypertension.
Hypertension most often occurs
hypoxemia, hypercapnia, electrolyte and acid-base imbalances, circulatory instability, preexisting heart disease, hypothermia, pain, surgical stress, and many anesthetic agents.
Many problems can cause dysrhythmias. These include
. Such imbalances may result from a combination of the body’s normal response to the stress of surgery, excessive fluid losses, and IV fluid replacement. The body’s fluid status directly affects cardiac output.
On the clinical unit, postoperative fluid and electrolyte imbalances are contributing factors to heart problems
, which maintains both blood volume and BP
Fluid retention during postoperative days 1 to 3 can result from the stress response
and adrenocorticotropic hormone (ACTH) and activation of the renin-angiotensin-aldosterone system (RAAS)
Fluid retention results from the release of antidiuretic hormone (ADH)
the adrenal cortex to secrete cortisol and, to a lesser degree, aldosterone
ACTH stimulates
and causing marked release of aldosterone. Both mechanisms that increase aldosterone lead to significant sodium and fluid retention, thus increasing blood volume.
Fluid losses resulting from surgery decrease kidney perfusion, stimulating the RAAS
when chronic disease (e.g., heart, kidney) exists, or when the patient is an older adult.
Fluid overload may occur during this period of fluid retention if we infuse IV fluids too rapidly,
blood loss during surgery, or slow or inadequate fluid replacement can decrease cardiac output and tissue perfusion. Losses from vomiting, bleeding, wound drainage, or suctioning can contribute to fluid deficits.
Fluid deficits from untreated preoperative dehydration,
can result from urinary and gastrointestinal (GI) tract losses.
Hypokalemia
the heart’s contractility and may contribute to decreases in cardiac output and tissue perfusion.
Low serum potassium levels directly affect
urine output of at least 0.5 mL/kg/hr and a normal serum creatinine are considered
indicative of adequate renal function.