Cerebellar Dysfunction Flashcards

1
Q

To which side of the body does the cerebellum sends information? (Ipsi or contralateral)

A

ipsilateral

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What are the functional division of the Cerebellum?

A
  1. Vestibulocerebellum
  2. Spinocerebellum
  3. Cerebrocerebellum
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Which lobe contains the vestibulocerebellum?

A

flocculonodular lobe

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Vestibulocerebellum input from and output to?

A

Input

vestibular nuclei

CN8

output

Vestibular nuclei

lateral vestibular spinal tract

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Function of the vestibulocerebellum

A

Integrates vestibular information and dictates motor control of neck and eye muscle

Allow to keep eye stable while head moves (gaze stabilization) & keep head over BOS

Control Gait and postural control

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Injuries to the vestibulocerebellum can lead to….

A

ataxic gait (path deviation TOWARD the side of the lesion)

inability to stabilize gait

deficit in postural control

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Which zone contains the spinocerebellum?

A

vermis

intermediate

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

input and output of the spinocerebellum

A

Input

spinal cord: Dorsal spinocerebellar tract & Vestibulocerebellar tract

Output

Vestibular nuclei

red nucleus

reticular formation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Function of the Dorsal spinocerebellar tract (DSCT)

A

sensory info from organ GTO, sensory, proprioception

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Function of the ventral spinocerebellar tract (VSCT)

A

sensory from LE & Postural control

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Function of the spinocerebellum

A

Integrates information from all decending motor tracts (VSCT)

real time information to be more successful with an intent action

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

location of the cerebrocerebellum

A

cerebellar hemispheres

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

input/output of the cerebrocerebellum

A

input:

deep pontine nuclei

output

thalamus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Function of the cerebrocerebellum

A

indirect information from all 4 cortical lobes

  • Areas 6,4,3,1,2,5
  • Primary visual cortext (17)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Lesions of the Cerebellum will lead to what types of deficits?

A

Impaired ability to perform controlled, precise, coordinated movements

must be large enough for symptoms to appear

hypotonia of MSRs (pendular reflex)

NO PARALYSIS and WEAKNESS (usually)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Function of Cerebellum in Movement

A

A regulator

feedback and feed-forward conrol ssytem

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Cerebellum input/output

A

input:

information about body position and muscle action

output:

to descending motor systems at the brainstem level

18
Q

Role of spinocerebellum in movement

A

output occurs while motion is occuring

the state of the motion, drives the next command fromt he cerebellum

19
Q

Role of Cerebrocerebellum in movement

A

feed-forward

receives infromation from PMA, SMA< and primary motor and sensory areas

modify the action before it takes place

Theory: manages the model of control system

20
Q

Hallmark signs of Cerebellar Dysfunction

A
  1. Ataxia (limb movement, trunkal, gait)
  2. Nystagmus
  3. Dysmetria
  4. dysdiadochokinesia
  5. Decomposition of movement ipsilateral to leasion
21
Q

Clinical Presentation of Vestibulocerebellar injury

A

Mimics disease in vestibular nuclei or PVS

Ataxic gait (fall toward side of lesion, inadequate postural correction)

Nystagmus

Vertigo/dizziness

22
Q

What is nystagmus

A

oscillating eye movement in which eyes move slow in one direction and rapidly in the other direction

23
Q

Features of Gait Ataxia

A

High stepping

stagger to the side of lesion

irregular foot placement

irregular timing

patient use decomposition of complex movements to control incoordination which results in robotic look

24
Q

Clinical Presentation of Spinocerebellar and Cerebrocerebellar injury

A

impairment in precision of movement

Dysdiadochokinesia

Dysmetria

decomposition of movement

pendular muscle stretch reflex

hypotonia (limb feel heavy)

25
Q

Dysdiadochokinesia

A

disruption in timing of muscle

difficulty with Rapid alternating movement (RAM)

26
Q

Dysmetria

A

Performance deterioration as motor act progresses

27
Q

What test can be used to test for dysmetria?

A

finger to nose test

28
Q

Dysmetria vs. Tremor

A

Dysmetria: form of incoordination secondary to cerebellar dysfunction. Performance deteriorates as target is approached (cerebellar injury)

Tremor:

rhythmic movement at constant frequency and amplitude. Result of continuous oscillatory beavior in central motor areas (e.g. Parkinson)

29
Q

What is decomposition of movements?

A

poor timing of motor acts

normal synergistic movement of complex motor tasks is broken down and initiated separately

difficulty with sequencing repetitive task

ataxia

30
Q

Role of Cerebellar in motor learning

A

modification of function in relation to experience

e.g. riding a bike

transfer of motor tactics from conscious to unconscious

Adaptive feedforward control system

Internal stored model controls learned movement

31
Q

with cerebellar lesions, movement must use ______ and thus producing uncoordinated movements

A

slow sensory feedback loops

32
Q

Theory of motor learning & cerebellum

A

Theory I:

CB learns small simple programs, facilitates triggering of small programs, in order, for complex motor tasks, recognizes and detects event sequences

Theory II:

CB role in adaptation, anticipatory ms activity across several joints, modification activity

Theory III:

CB influence not purely motor, role in mental imagery/practice, role in rapid shifts of attention

33
Q

Examination of Cerebellum should include

A
  1. RAM
  2. gait
  3. dysmetria
  4. decomposition
  5. VOR
  6. change of symptoms with position changes
34
Q

Consideration when tx patient with CB injury

A
  1. Postural control
  2. Motor Learning (need more attention)
  3. Attention (problem with switching task)
  4. Motor control (less automatic)
  5. Functional tasks

Get tired very easily: block practice

decrease distraction/obstacles

1 feedback at at time

concurrent tactile feedback, auditory feedback att he end

35
Q

Ways to work on Dysmetria

A

Temporary reduction techniques

  • as primer for functional tasks
  • PNF
  • full body movements
  • T-band
  • Frenkel exercises (Progressive exercises for ataxia and coordiantion
  • Wt to dampen ataxia (use proximal not distal)
36
Q

Intervention for ataxic gait

A

Encourage stiffening of joints (ankle)

practice setup part to whole

frequent feedback

use of device (walker tends to get far away, encourage small steps)

37
Q

There is an increase firing of what structure during acquisition of complex motor task?

A

Cerebellum

38
Q

Effect of Visual-vestibular dysfunction

A

Diplopia

impact on function (decrease attention on sx)

Use of visual exercises (smooth pursuit, saccades, VOR)

habituation exercises (full body position changes to get used and not get dizzy)

39
Q

+ Prognosis of CB injury recovery

A

spared cerebral cortex

unilateral injury

degenerative disease vs acute injury

40
Q
  • prognosis of CB injury
A

bilateral injury

damage to deep CB nuclei

damage to others aspects of brain

concomitant abuse of alcohol