Case introduction Flashcards
define coronary artery disease and ischaemic heart disease
narrowing of the lumen of the coronary arteries resulting in an imbalance between the supply of oxygen and myocardial demand resulting in myocardial ischaemia and chest pain
what is atherosclerosis
a process where over time, the walls of the arteries can become furred up with fatty deposits known as atheroma
what are the 2 types of angina
stable and unstable
what are the types of acute coronary syndromes
NSETACS
- unstable angina
- NSTEMI
what are STEMI
- heart attack
- myocardial infarction
what is stable angina
attacks are brought on by an obvious trigger (eg exercise) and improve with medication and rest
what is unstable angina
attacks are more unpredictable, occurring with no obvious trigger and continuing despite resting
what are NSTEMI
- narrowed blood vessels (atherosclerosis) and thrombolysed plaques (atheromatous plaque rupture)
- may get transient blocked artery, causing pain
- thrombolysed plaques- blood will pass but risk of going onto developing STEMI/death
- treated according to severity
- reduction of platelet activity
- treatment to relieve symptoms
- treatment to reduce cardiovascular risk
what are STEMI and how are they caused
- narrowed due to build up of atherosclerosis and then clot has blocked artery
- need to reopen blocked coronary artery - more pain than angina and lasts longer
- sweaty, nausea and pale
- GTN S/L doesn’t always work
give examples of signs and symptoms of coronary heart disease
- signs: ECG and biochemical markers (troponin)
2. symptoms- chest pain, anxiety, sweaty, shortness of breath, nausea
give examples of non modifiable risk factors for CVD
- family history
- age
- gender
- ethnicity
give examples of modifiable risk factors for CVD
- hyperlipidaemia
- smoking
- hypertension
- diabetes mellitus
- personality
- abdominal obesity
describe the process of diagnosis of CVD
- ECG
- NSTEMI: ST depression/T wave inversion
- STEMI: ST elevation
- Previous STEMI: Q waves - cardiac enzymes- troponin T on presentation
- history- good patient story and PMH