Carotid Flashcards
aphasia
inability to express oneself, dominant hemisphere, usually left
dysphasia
impairment of speech, lack of coordination, can’t arrange words in proper order
dysphagia
difficulty swallowing
dysarrthria
imperfect articulation , slurring etc.
lateralized paresthesia
tingling, numbness, contralateral hemisphere affected
hemiparesis
lateralized weakness, contralateral hemisphere affected
hemiplegia
lateralized paralysis, contralateral hemisphere affected
amaurosis fugax
transient monocular blindness- ipsilateral carotid artery
ataxia
incoordination of muscle movements, contralateral hemisphere affected
drop attack
falling down without fainting
drop attacks are a type of
VBI = vertebrobasilar insufficiency
syncope
being unconscious without losing consciousness
syncope
temporary loss of consciousness caused by a fall in blood pressure.
vertigo
sensation of motion or spinning
dizziness
sensation of being off balance
diplopia
double vision
binocular blindness
transient, bilateral blindness, acute onset
4 causes of stroke
cardiac source (emboli) carotid origin cerebral aneurysm rupture vasospasm- sickle cell anemia congenital AVM
carotid origin
thrombus, emboli, stenosis, occlusion, atherosclerotic stenosis
vasospasm can be caused by
sickle cell anemia
cerebral aneurysm rupture causes
intracranial hemorrhage
intracranial hemorrhage aka
subarachnoid hemorrhage
intracranial aneurysm aka
Berry aneurysm
vasospasm usually follows
subarachnoid hemorrhage
brachiocepalic artery aka
innominate
distal vertebral arteries form the
basilar artery
basilar artery supplies
posterior hemisphere via PCA
first branch off aorta
innominate/ brachiocepalic
second branch off aorta
LCCA
third branch off aorta
left subclavian artery
first branch off ICA
ophthalmic artery
RCCA branches off
innominate a
first branch off ECA
superior thyroid a
ICA sits more ___ than the ECA
posterior
how to tell the ICA from the ECA
ECA has branches
the ECA has ____ branches
extracranial
flow pattern in ICA
low res
flow pattern in ECA
high res
temporal tap
to distinguish ECA
sonolucent/echolucent
anechoic
velocity changes occur when stenosis becomes
50% or greater
vertebral artery flow pattern
low res
PSV in vert a
20-70 cm/s
retrograde flow in vert a suggests
subclavian steal
distal to stenosis
turbulence, tardus parvus
prox to stenosis
increased pulsatility, low velocity
FMD
fibromuscular dysplasia
FMD affects
media of walls, women, renal arteries, ICA
FMD appearance
“string of pearls”
medial fibroplasia
most common type of FMD
prox CCA dissection occurs with
aortic dissection
carotid body tumour location
between ICA and ECA, in fork of CCA bif
carotid body tumour
slow growing, benign, asymptomatic
carotid body tumour aka
chemodectoma, paraganglioma
carotid body tumour on US
splaying of ICA, ECA
looks like thryoid tissue
highly vascular - fed by ECA
atherosclerosis affects predominantly
bif
prox disease indicated by
rounded waveform, prolonged rise to peak (AT), low amplitude (PSV)
re-stenosis of stent caused by
neo-intimal hyperplasia, kinking
NASCET
north american symptomatic carotid endarterectomy trials. 1991
% stenosis =
100 x [1- (residual lumen/true lumen)]
NASCET % stenosis
70% or greater
NASCET outcome
symptomatic patients benefitted from CEA
ACAS
asymptomatic carotid atherosclerosis study
ACAS % stenosis
60% or greater, asymptomatic males
ACAS outcome
reduced stroke risk by 6 % over 5 yrs
ACAS for women
no benefit
ACAS measurement
residual lumen compared to ICA diameter distal to stenosis