Care of the elderly Flashcards

1
Q

What increases the susceptibility of acquiring C. difficile?

A
  1. Antibiotic related: Fluoroquinolones (ciprofloxacin), cephalosporins (cefotaxime), penicillin and clindamycin
  2. In contact with another person who has C. diff
  3. PPIs
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2
Q

How is C. diff spread?

A

In bacterial spores within faeces

prevented by handwashing, room sterilisation, limited antibiotic use

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3
Q

What are the symptoms of a C. diff infection?

A

Watery diarrhoea
Fever
Nausea
Abdominal pain

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4
Q

How is c. diff diagnosed?

A

Blood culture: C diff toxin positive

Stool culture: c diff toxin positive

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5
Q

What is the management of C. diff?

A
  1. Stop offending antibiotics.
  2. infection control
  3. oral metronidazole
  4. if severe - vancomycin
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6
Q

What are some of the complications of c. diff

A
dehydration
pseudomembranous colitis
toxic megacolon
perforation of the colon
sepsis
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7
Q

How can c. diff be prevented?

A
  1. prevent cross infection

2. reducing antibiotic misuse

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8
Q

How likely is an elderly person going to fall within one year of having a fall?

A

66%

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9
Q

What are the risk factors for falling?

A
  1. Age >80
  2. Low weight
  3. Dependent on others for ADLs
  4. Previous fall (in the last 12 months)
  5. Fear of falling
  6. Balance problems
  7. Gait and motility problems ie parkisons
  8. Pain
  9. Drugs ie polypharmacy, antihypertensives
  10. Cardiovascular conditions
  11. Cognitive impairment
  12. Urinary incontinence (rushing to the toilet)
  13. Stroke
  14. Diabetes
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10
Q

What increases the risk of injury from falling?

A
  1. Weak bones (osteoporosis, osteomalacia, paget’s diseaese, bone mets)
  2. predisposition to falls
  3. poor self protection (lack of subcutaneous fat, loss of consciousness, neuropathy)
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11
Q

What questions should be asked when a patient presents with a fall?

A
  1. Is it an isolated event? is there a pattern to the falls?
  2. What causes the fall?
  3. What was the patient doing at the time?
  4. Was there a loss of consciousness?
  5. Was there any warning signs before the fall?
  6. What was there post-fall state like?
  7. Collateral history
  8. When was their last eyesight and optician review?
  9. Ask about past medical history
  10. Polypharmacy and drugs?
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12
Q

What examinations should be done in a person who has fallen?

A
Vital signs (postural hypotension)
Cardiac examination
Neurological examination
Systemic examination
Mini-mental state examination
Blood glucose
Visual impairment assessment
'Timed up and go test'
'Turn 180 degree test'
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13
Q

What is the management of a fall?

A
Treat any injuries
Treat underlying causes
Medication review
Physiotherapy review
OT review
Optician review
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14
Q

How can falls be prevented?

A
  1. Group and home based exercise programmes
  2. Home safety interventions
  3. T’ai chi
  4. Risk assessment of environmental factors
  5. Treating underlying medical condition
  6. Alcohol reduction
  7. Vitamin D for ‘weak bones’
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15
Q

What is osteoporosis?

A

A progressive systemic skeletal disease characterised by reduced bone mass and micro-architectural deterioration of bone tissue

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16
Q

What is an osteoporotic fracture?

A

Fractures resulting from mechanical forces that wouldn’t ordinarily result in fracture

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17
Q

What does a T score of below -2.5 SD from normal indicate?

A

Osteoporosis

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18
Q

What is the epidemiology of osteoporosis?

A

Women are more likely to get it than men

Age related disease (bone mass declines with age, and is accelerated when women hit menopause)

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19
Q

What are the risk factors for fragility fractures?

A
REDUCED BMD
Increasing age
Female sex
Low body mass (<19kg/m2)
Parental history of a hip fracture
Parental history of fragility fracture
Corcticosteroid therapy
Cushing’s syndrome
Alcohol (>3 units a day)
Smoking
If increased risk of falling due to medical conditions
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20
Q

What are some of the secondary causes of osteoporosis (medical conditions)

A

Rheumatoid arthritis and other inflammatory arthropathies.
Prolonged immobilisation or a very sedentary lifestyle.
Primary hypogonadism (men and women).
Primary hyperparathyroidism.
Hyperthyroidism.
Post-transplantation.
Chronic kidney disease.
Gastrointestinal disease such as Crohn’s disease, ulcerative colitis and coeliac disease.
Untreated premature menopause (<45 years) or prolonged secondary amenorrhoea.
Type 1 diabetes mellitus.
Chronic liver disease
Chronic obstructive pulmonary disease

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21
Q

What is the presentation of osteoporosis?

A

Asymptomatic until patient has a fall resulting in a fragility fracture
Fractures are commonly at the spine, hi[ and wrist

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22
Q

What are the investigations for osteoporosis?

A

Xray
DEXA scan
Bloods (FBC, ESR, CRP, U&E, LFTS, TFTS, hormones, serum immunoglobulins and urinary bence Jones’ proteins)

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23
Q

What is the management of osteoporosis?

A
  1. Lifestyle advice
  2. Adequate calcium and vitamin D intake
  3. Bisphosphonates
  4. Denosumab
  5. Treatment of fractures
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24
Q

What is incontinence and what are the different types?

A
  • An involuntary leakage of urine
    1. Functional
    2. Stress
    3. Urge
    4. Mixed
    5. Overflow
    6. True incontinence
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25
What are the characteristics of functional incontinence?
- patient is unable to reach the toilet in time - usually have poor mobility - can be unfamiliar to surroundings
26
What are the characteristics of stress incontinence?
- Involuntary leakage during effort or exertion, coughing or sneezing. - Due to an incompetent sphincter - Can be associated with a genitourinary prolapse
27
What are the characteristics of urge incontinence?
- Involuntary urinary leakage accompanied by, or immediately preceded by urgency of micturition - Detrusor instability or hyperreflexia = involuntary detrusor contraction - Can be idiopathic or secondary to neuro problems
28
What are the characteristics of mixed incontinence?
Involuntary leakage or urine associated with urgency and exertion, effort, coughing or sneezing
29
What is the characteristics of overflow incontinence?
-Chronic bladder outflow obstruction. Often seen in prostatic disease in older men. Can lead to obstructive nephropathy
30
What is true incontinence?
Fistulous track between vagina and ureter or bladder or urethra causing continuous leakage or urine
31
What is the epidemiology of incontinence?
Prevalence increases with age
32
What are the risk factors for incontinence?
Women: pregnancy and vaginal delivery, Diabetes mellitus, high BMI, hysterectomy, UTIs, oral oestrogen therapy Men: LUTS, infections, functional and cognitive impairment, euro disorders Prostatectomy
33
What is the management for incontinence?
- Temporary containment pads - pelvic floor exercises - Stress: duloxetine or surgery - Mixed: antimuscarinics (oxybutynin) - intermittent self-catherterisation - bladder training - Botulinum toxin type A for neuro conditions - urostomy
34
What are some neurological conditions that can result in incontinence?
1. Stroke 2. Parkinsons 3. Multiple sclerosis 4. Dementia 5. Spinal cord injury
35
What is parkinsons?
Neurodegenerative disease | -decrease in dopamine due to depletion of the substantia nigra
36
What are the parkinsonian cardinal features?
- rigidity of movement - hypertonia (resting tremor) - bradykinesia - postural instability
37
What is the aetiology of parkinsons?
UNKNOWN - ?genetic factors - ?synthetic opioid impurity - ?pesticide exposure
38
What is found histiologically in Parkinsons?
Lewy bodies in the substantia nigra
39
What is the epidemiology of parkinsons?
Typically develops between 55-65 | slightly more common in men
40
What are the clinical features of parkinsons?
- resting tremor - pill rolling of thumb and fingers, improved by voluntary movement - rigidity - leadpipe/cogwheel rigidity - bradykinesia - slowness of voluntary movement, difficulty initiating movement, monotonous speech, shuffling gait -sleep disturbances, shuffling gait, reduced arm swing, depression and associated with lewy body dementia
41
What investigations are done in parkinsons?
1. Clinical | 2. Exclude other causes for parkinsonism ie CT for head injury, multi infarct dementia, medication review
42
Pharmacological management of parkinsons
1. Levodopa with carbidopa 2. Dopamine agonists (ropinirole) 3. Catecholamine-O-Methyltransferase inhibitors (Entacapone) 4. Monoamine-oxidase B inhibitors (Selegiline) 5. Anti-cholinergic (Benztropine)
43
What's the non-pharmacolgoical management of parkinsons?
1. Physiotherapy 2. Occupational therapy 3. SALT 4. Deep brain stimulation
44
What is drug induced parkinsonism and how is it treated?
Antipsychotics and antihypertensives are known to block dopaine receptors When the drug is stopped, the symptoms stop
45
What is parkinsonian dementia?
- dementia with lewy bodies - visual hallucinations, falls and memory loss - changes in executive functioning ie planning the day, anticipating events, understanding jokes
46
What is a stroke?
- A clinical syndrome caused by a disruption of bloody supply to the brain causing a focal or global disturbance to cerebral functions - >24 hours
47
What are the 2 types of stroke?
1. Ischaemic infarction (85%) | 2. Haemorrhage (15%)
48
What are ischaemic infarctions caused by?
1. thromboembolisms 2. atheromas 3. trauma 4. infection 5. malignancy
49
What are haemorrhagic stroke classical features?
1. meningism 2. severe headache 3. coma within a few hours
50
What's the aetiology of stroke?
1. thrombosis in situ 2. atherothrombolism 3. heart emboli 4. cns bleed (aneurysm rupture) 5. vasculitis 6. space occupying lesion 7. venous sinus thrombosis
51
What's the epidemiology of a stroke?
->65 years old
52
What are the risk factors for stroke?
1. hypertension 2. smoking 3. diabetes mellitus 4. heart disease (valvular, ischaemic, af) 5. peripheral artery disease 6. post-tia 7. carotid artery occlusion 8. combined oral contraceptive pill 9. hyperlipidaemia 10. excess alcohol 11. clotting disorders
53
What's the presentation of stroke?
SUDDEN ONSET - cerebral hemisphere infarcts: contralateral hemiplegia, contralateral hemiplegia, homonymous hemianopia, dysphasia - posterior circulation ischameia: ataxia, motor defecits and weakness, crossed syndrome, sensory defecit, diplopia, dysphagia, locked in syndrome
54
What are the investigations for a stroke?
CT SCAN: excludes haemorrhagic stroke for thrombolysis | BLOODS: FBC, ESR, hypoglycaemia, syphillis screen
55
Management of ischaemic stroke
aspirin 300mg thrombolysis (once haemorrhagic stroke has been excluded as cause) -long term antiplatelet therapy -carotid endarectomy
56
Management of heamorrhagic stroke
surgery
57
What's the MDT approach to treating a stroke?
1. physio 2. occupational therapy 3. TREATMENT OF RISK FACTORS
58
What's the ABCD2 score?
to determine the likelihood of having a stroke after having a TIA - Age >60 - Blood pressure >140/90mmHg - Clinical features (unilateral weakness, or speech disturbance without weakness) - Duration of symptoms (>60 mins+ or 10-59mins) - Diabetes
59
What is a subarachnoid haemorrhage?
Spontaneous arterial bleeding into the subarachnoid space | Caused by a rupture of ‘berry aneurysms’ or congenital arteriovenous malformations
60
What are the clinical features of a SAH?
- Sudden onset of severe (thunderclap headache) - Absence of similar headaches in the past - Nausea and vomiting - Sometimes loss of consciousness - meningeal irritation - focal neurological signs - papilloedema
61
What are the investigations for a SAH?
CT head lumbar puncture MR angiography (establishes fitness for surgery)
62
What's the management for SAH?
1. bed rest 2. supportive measures 3. nimodopine (reduces artery spasming) 4. surgery
63
What is a subdural haematoma?
- an accumulation of blood in the subdural space | - usually caused by a rupture of bridging veins in the sagital sinus
64
Who are most at risk of having a subdural?
elderly or alcoholics - due to atrophied brains - increased likelihood to fall
65
what are the clinical symptoms of a subdural?
headache drowsiness fluctuating confusion LATENT INTERVAL BETWEEN INJURY AND SYMPTOMS
66
How are subdurals diagnosed?
ct head
67
What is the management of a subdural?
surgical removal of the haematoma
68
What is an extradural haematoma?
-injuries that fracture the temporal bone and rupture the middle meningeal artery
69
what is the typical presentation of an extradural haematoma?
- history of head injury with brief loss of consiousness - followed by lucid interval - rapid deterioration with focal neurological sigsn
70
What's the treatment for an extradural haematoma?
surgical drainage
71
What are pressure ulcers?
localised damage to the skin and/or underlying tissue that usually occur over a bony prominence resulting from pressure
72
What causes pressure ulcers?
- external pressure causes obstruction of the blood capillaries - friction damages the superficial vessles - shearing (separation of the skin from underlying tissues due to opposing forces) - moisture
73
Where are common sites for pressure sores?
sacrum coccyx heels hips
74
What are risk factors for pressure sores?
- immobile - microclimates - malnutrition - low bmi - arteriosclerosis - neuropathies - age >70 - dry skin - malignancy
75
What are the stages of severity of pressure sores?
-Stage 1: intact skin, non-blanchable redness -Stage 2: partial thickness loss of dermis: Shallow open ulcer with red pink wound bed -Stage 3: full thickness tissue loss, subcutaneous fat may be visible -Stage 4: full thickness tissue loss, with exposed bone, tendon, muscle. Slough may be present. Deep tissue injury - A purple or maroon localized area of discolored intact skin or blood-filled blister due to damage of underlying soft tissue
76
What is the treatment for pressure sores?
- debridement - dressings - smoking cessation - treatment of medical conditions which can slow down healing process ie infection
77
How can pressure sores be prevented?
- regular movement of the pt - balanced diet with adequate protein and vit c - support surfaces ie pressure sore mattresses