Cardioresp adaption to exercise Flashcards
why is exercise important?
- lowers risk of CVS disease
exercise testing is the basis for the functional evaluation of the cardio-resp system
Describe the 2 different types of muscle
- slow twitch - type I - dependent on oxidative phosphorlylation, lots of mitochondria and myoglobin, mainly postural but also some used in endurance events
- fast twitch - type IIa and type IIb - relies on creatine phosphate to regenerate ATP, large amount of glycolytic enzymes, used in spirnting and explosive events
how is exercise intensity defined in terms of oxygen uptake?
- in dynamic exercise, oxygen consumption increases with work in a linear manner
- resting oxygen consumption - 250mls/min, moderate - 1000mls/min etc
what is the reason for the increased O2 consumption in exercise?
the replacement of ATP
what is the maxinal O2 consumption called?
the VO2 max
define the VO2 max
the** maximum rate **at which the heart, lungs, and muscles can effectively use oxygen during exercise, used as a way of measuring a person’s individual aerobic capacity.
compare the values for a typical, trained and elite VO2 max
ml/min/kg - units
- typical VO2 max - 4ml/min/kg
- trained VO2 max - 6ml/min/kg
- elite VO2 max - 8ml/min/kg
what 3 aspects does O2 delivery depend on?
- getting 02 into the body and blood -respiration
- getting O2 from the** lungs to the tissues** (cardiovascular delivery)
getting o2 from the blood to the** respiring tissues** (O2 extraction)
what occurs to the O2 and C02 production in exercise?
they both increase
do arterial blood gases change in moderate exercise?
no
which states of CO2, o2 & ph would be expected to increase ventilation?
- high C02
- low O2
- low PH
what triggers the increase in ventilation at higher O2 consumptions?
low PH
what is the anaerobic threshold?
- when the demand for O2 exceeds the delivery of O2
increased HR is a feature of exercise, wahat branch of the ANS is involved in stimulating an increase in HR?
the sympathetic system
during exercise, describe the adaptions that are made to the CV system & what system activates this?
- driven by the sympathetic NS
- increase in cardiac output and redistribution of blood flow
- increased venous return and therefore SV- due to increased muscle pump, resp pump and venoconstriction
- increased contractility of the heart driven by SNS
why does the stroke volume decrease at high work loads?
- the faster the heart beats, the less time there is too fill the ventricles
- preload is too low due to decreased ventircular filling time
what does the SNS do to blood vessels?
causes vasoconstriction
what does the increased metabolite production (CO2, K+, H+ & adenosine) do to the blood vessels?
- vasodilation
in exercisem oxygen has to get from the lungs into the blood, in the blood to the tissues and from the blood into the tissues .. which of these limits performance?
- transport in blood from lungs to tissue
define O2 dept
- The amount of oxygen required to remove the lactic acid, and replace the body’s reserves of oxygen, is called the oxygen debt
what 3 stages in oxygen debt incurred?
- depletion of ATP
- depletion of creatine phosphate
- build up of lactic acid
what are the effects of training on muscle?
- increase** muscle strength and resistance to fatigue **
- muscle** hypertrophy -** increased muscle diameter
* possible shift in muscle fibre type - between type I and type II - increased glycolytic and oxidative capacity - increased ATP, mitochondrial enzymes etc
what are the cardioresp effects of training?
- bradycardia- slowed HR
- increased ventricle size and SV so stroke volume and cardiac output remain the same
- reduced blood pressure
- increased levels of myoglobin & a,3 BPG - which facilitates better O2 delivery
what are examples of things that training will not improve?
- VO2 or the cardiac output at rest
- diffusing capacity or the haemoglobin concentration
- max HR
describe the distribution of Cardiac output during exercise to different regions of the body including muscles, heart, GIT & kidneys
- there is sympathetic stimulation - which causes vasoconstriction
- there is an increase in resistance overall in the vessels due to the sympathetic system, areas like the GIT and kidneys experience lower blood flow due to the increased resistance
- in areas like the heart and the muscles, metabolites eg adenosine - will override the sympathetic vasoconstriction and there will be increased blood flow to these regions