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Pediatric Heart > Cardiology > Flashcards

Cardiology Flashcards

1
Q

What is the only artery in our body that carries deoxygenated blood?

A

Pulmonary artery (arteries carry blood away from body)

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2
Q

Where does fetal circulation occur?

A

The placenta. Not the Lungs.

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3
Q

What is the foramen ovale

A

Opening between two atria; closes hours after birth

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4
Q

In order to switch from fetal to pulmonic circulation. What has to occur?

A

Clamping of umbilical cord (increases SVR)

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5
Q

The foramen ovale closes when:

A

Right sided heart pressure decreases in resistance

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6
Q

The ductus arteriosis connects:

A

pulmonary artery and aorta. Smooth muscle senses increase 02 in blood and decrease in prostaglandin (signals that placenta is gone) and it will contract close.

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7
Q

If a child is squatting frequently, why are they doing it?

A

Increase blood flow to the lungs. Usually this is caused by hypoxia and is common in TOF.

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8
Q

Fatigue from decreased cardiac output occurs:

A

In the evening

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9
Q

Cyanosis is more common in:

A

Poor perfusion d/t decreased CO. Examples: patent ductus arteriosis or foramen ovale

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10
Q

Point of maximal impulse is found in the

A

4th intercostal space in babies and kids

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11
Q

If PMI is raised, widened or far to left

if raised on lower left sternal border

A

suspect left ventricular hypertrophy.

suspect right ventricular hypertrophy

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12
Q

Murmmurs stemming from pulmonary valve are best heard

A

Left; second intercostal space

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13
Q

Ventricular septal defects are best heard in

A

the left lower sternal border/erb’s point

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14
Q

Av. infant HR

Toddler

A

120-180 bpm

80-105bpm

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15
Q

Blood pressure ___ with age, HR ___with age

A

increases; decreases

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16
Q

Extracardiac signs include

A

Cyanosis, enlarged liver (normal to have 1-2cm in newborns), respirations (labored or tachypnea), failure to thrive, displaced PMI, sinus arrhythmia, or murmurs

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17
Q

Pulse oximetry must be done within____ after birth

A

24hr. They need a pre-ductal right hand and post ductal foot)

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18
Q

Passing pulse oximetry must be

A

> 95%, only <3% points or less difference in r hand and foot

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19
Q

Failing pulse ox.

A

<90%

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20
Q

Repeat pulse ox if:

A

if >90 but less than 95% or >3% difference, repeat in 1 hour.. If the same after 3rd screen, they failed.

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21
Q

S1 is the closure of the_____

A

Mitral and tricuspid valve best heard over the heart’s apex and coincides with the start of ventricular systole and precede a palpable carotid pulse.

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22
Q

S2 is the closure of

A

closure of aortic and pulmonic valves. Please her over Erb’s point or pulmonic area

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23
Q

Splitting of S2 can be normal? True or false.

A

True. Can occur in children upon inspiration.

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24
Q

S3 is caused by

A

vibrations heard during rapid/passive ventricular filling, low pitch, and best heard with bell of stethoscope. Best heard in <20 or athletes

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25
Q

S4 occurs at the end of____

A

diastole when atrial contraction further stretches and fills the ventricles. Abnormal and best heard with bell in lateral left recumbent position

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26
Q

Heart murmurs are classified by

A

Location and Intensity

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27
Q

Grade 1

A

Faint, possibly intermittent, heart with stethoscope

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28
Q

Grade 2 murmur

A

Faint, heard as soon as you place stethoscope

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29
Q

What grade is an easily heard murmur, moderately loud?

A

Grade 3

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30
Q

what grade is a loud murmur, possible with palpable thrill that may radiate to flow of blood

A

Grade 4

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31
Q

Mummurs that are loud enough to be heard only with the edge of stethoscope on chest wall and are ALWAYS accompanied by thrill and radiation

A

Grade 5

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32
Q

Loud enough to be heard without stethoscope touching chest wall; always accompanied with thrill and radiation

A

Grade 6

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33
Q

Mitral regurgitation causes

A

high pitch, blowing holosystolic murmur; heard best at apex; radiates to axilla. Incompetent mitral valve causing back flow (often seen in rheumatic heart disease)

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34
Q

Systolic heart murmurs (Mr. Pass wins Most Valuable Player)

A

Mitral Regurgitation, Physioloic (innocent, functional), Aortic Stenosis, Systolic, Mitral valve prolapse

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35
Q

Aortic Stenosis

A

crescendo decrescendo (softer) that radiates to carotids with sometimes palpable thrill.

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36
Q

Mitral Valve prolapse

A

Mid systolic click murmur due to valve balloning in atria.

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37
Q

Diastolic heart murmurs (Ms. ARDS)

A

Mitral Stenosis, Aortic Regurgitation (early diastolic murmur heard best in aortic/pulmonic area or mitral area, soft and blowing), Diastolic.

38
Q

Mitral Stenosis is a___

A

Mid late diastolic murmur

39
Q

Cyanotic heart defect is:

A

Cardiac anomaly where oxygenated blood entering the aorta and systemic circulation is mixed with deoxygenated blood/ can result from any condition that increases pulmonary vascular resistance (right to left shunt) or structural defat that allows aorta to receive blood from right side of heart

40
Q

Assess for ____ in cyanotic heart defects

A

Cyanosis, especially while crying. CBC shows polycythemia as hypoxia stimulates body to increase RBC production, clubbing of digits

41
Q

Tetralogy of Fallot (TET) has 4 defects…what are they

A 
4 defects:
Pulmonary valve stenosis
Right ventricular hypertrophy
Ventricular Septal Defect
Overriding Aorta (oxygenation and deoxygenated blood)
42
Q

TET is a:

Severity is based on degree of dyspnea and right heart hypertrophy in history

A

cyanotic heart defect: mixing of oxygenated blood and deoxygenated blood into systemic circulation. stems from increased pressure in R ventricle. blood shunts right to left across VSD, forcing deoxygenated blood into L side

43
Q

Acute blue spells may cause a child to squat to increase _____ and force blood into pulmonary artery and therefore lungs

A

SVR

44
Q

Infindibular spasm (muscle below pulmonary valve) in TET

A

creates obstruction to blood flow in lungs

45
Q

Assessment of TET

A

Grade 3-5 harsh systolic ejection murmur ;eft mid upper sternal border with thrill, sternal lift secondary to RV hypertrophy, cyanosis of mucous membranes, dyspnea

46
Q

Transposition of Great Vessels

A

Aorta is attached to R side, PA to left ; parallel circulation. May give prostaglandins to keep PAD open but will need surgery

47
Q

Hypoplastic Left Heart Syndrome

A

Consists of aortic valve atresia, mitral atresia, or stenosis, small or absent left ventricle, or severe hypoplasia of ascending aorta or arch. Prostaglandins given to keep ductus open. Need 3 stage surgical repair

48
Q

The difference of cyanotic and acyanotic heart defects is that

A

blood entering the aorta is fully oxygenated

49
Q

Acyanotic heart defects include

A

VSD (left to right side shunt or increased blood volume on right side will cause right sided hypertrophy and increased blood flow to lungs)

50
Q

CHF is frequently seen in Acyanotic Heart defects d/t

A

left to right shunting

51
Q

Early symptoms of acyanotic heart defects

A

Tachycardia, diaphoresis, tachypnea, fatigue, and mild cyanosis

Other signs: congested cough, hepatomegaly, poor growth and development

52
Q

VSD

A

hole between R and L ventricle. Most common congenital heart defect.
Watch for CHF, patch placed when child is older, correction can lead to heart block, can spontaneously close by age 3.

53
Q

VSD interventions

A

Digoxin, monitor fluis, Lasix, high calorie formula, surgery consult

54
Q

Artrial Septal Defect

Management: per cardiology

A

May fatigue early, Exertional dyspnea, thin, frequent URIs, may or may not have a murmur (1-3, in pulmonic area), possible l sternal border left, FIXED S 2 split

55
Q

Obstructive defects

A

Valves/Ventricles are narrowed to the point that it completely blocks flow of blood.

56
Q

Pulmonary Stenosis

A

Narrowing of valve increased right side pressure. Asym or exertion dyspnea or Right side heart failure, chest pain with exercise, murmur 2-4 harsh, mid late, systolic ejection, hear at l sternal border over pulmonic radiates to neck, back, and lungs)

57
Q

Aortic Stenosis

A

Severity ranges from mild to extremely severe. Activity intolerance, fatigue, chest pain in addition to Left side heart failure, pulmonary congestion, syncope.

Murmur:3-4 loud, harsh, crescendo/decresendo heard best over upper right sternal border radiating to neck and left lower sternal border and apex. Thrill at sternal notch.

58
Q

Coarctation of Aorta

Repair :angioplasty

A

Decreases blood flow from trunk and extremities and increases flow to head and arms which will predispose the child to a stroke

Assess for: full bounding pulses in arms and weak or absent pulse in legs

Assess for increased BP in arms/decreased in legs

Palpate warm upper body and cold lower.

59
Q

Chest Pain
Non cardiac
Costochondritis

A

Palpate the chest

unilateral sharp stabbing pain exacerbated by deep breathing, no sings of inflammation or tenderness on palpation

60
Q

Tietze Syndrome

A

Hx. recent URI with lots of coughing. nonsupprative inflammation of costochondral, costosternal, or sternoclavicular joint
Usually in older kids for NSAIDs okay

61
Q

Idiopathic Chest Pain

A

Sharp pain that lasts a few seconds and localizes to middle of stream, exacerbated by deep breathing or manual pressure on rib cage

62
Q

Noncardiac Chest Pain Treatment

A

Rest, Reassurance, Analgesia- warm compress, non steroidal or anti inflammatory drugs NSAIDS x 1 week

63
Q

If pulmonary or Airway related

A

We think of bronchitis, plural effusion, asthma, pneumonia. Ask what they were doing when the pain started.

64
Q

Cardiac Chest Pain causes

A

Aortic dissections (Marfan syndrome which affects smooth muscle and have larger arm span than height, can cause extremely severe painful tearing in mid sternum)
Palpitations
Congential heart disease
pulmonary hypertension (heaves)
toxic exposure
If laying down or distant heart sounds (pericarditis)

65
Q

Analysis of fasting lipid profile identifies which 2 major dislipidemic patterns in childhood?

A

familial hypercholesterolemia and combined dsylipidemia.

66
Q

Non-HDL-C (TC-HDL-C) is

A

new accurate screen for dyslipidemia in childhood. Accurate in non fasting state.

In adults, better predictor of cardiac events than LDL-C.

67
Q

2-8y are not usually screened for lipids. However they will be tested twice with results averaged if:

A

Parent, grandparent, aunt/uncle, or sibling with MI, angina, stroke, CABG at <55 M or <65 F.
Parents with TC >240 mg/dL or known dyslipidemia
Child has DM, HTN, or BMI >95th % or smokes
Child has moderate to high risk medical condition

68
Q

Universal screening for lipids starts at

A

9-11

Non fasting HDL-C.

69
Q

If Non-HDL C is >145 or HDL <40 when screening, what is the next test?

A

Two fasting lipid profiles
OR
FLP rewear FLP in 2w but within 3 months

70
Q

17-21 second lipid screening with

A

Non-FLP calculating HDL-C or FLP

If greater than 21 you can do a FLP and if abnormal repeat in 2 w but within 3 months and average score.

71
Q

When to start pharmacotherapy in a child with HTN

A 
Persistent HTN despite lifestyle change for 6 months
Presence of target end organ damange
DM 1 or 2
Kidney disease
secondary HTN
Stage II HTN
symptomatic primary HTM
multiple risk factors
72
Q

Meds used for HTN treatment:

ACE (1m-16yo)

A

ACE inhibitors (preventing angiotensin 1 to 2, increase plasma renin and reduce aldosterone)
Lisinopril and Captopril
Monitor BP q 1-3 hours, BUN, Cr, WBC, serous K and glucose
Adverse: hypotension, tachycardia, syncope, dyspnea, fatigue, dizziness, HA, per K, muscle cramps, diminished renal function.

73
Q

ARBs

>6

A

Selective and competitive angiotensin II receptor antagonists. reduces vasoconstriction and aldosterone)

Losartan 0.7mg
Monitoring is the sane.

Adv effects: chest pain, cough, URIs, nasal congestion, anemia, thrombocytopenia, fatigue, dizziness, hypoglycemia

74
Q

Beta blockers

Metroprolol and Atenonolol 1mg

A

Monitor HR/BP
SE: nightmares, insomnia, confusion, depression, bradycardia, AV block, hypotension, chest pain, edema, CHF, Raynaud’s, bronchospasm, nausea, pruritus

75
Q

Calcium Channel blockers (Amlodipine)

6-17yo

A

prevents calcium ions from entering vascular smooth muscle and myocardial cells via specific slow calcium channel during depolarization
2.5-5mg
monitor: BP, electrolytes, BUN/Cr, CBC, and UA
Adv effects: flushing, palpitations, peripheral edema, pulmonary edema, nose bleeds, fatigue, dizziness, HA, diarrhea, anorexia, constipation, dyspepsia, dysphasia, pancreatitis, diploma, hyperglycemia, thrombocytopenia, eye pain

76
Q

What disease results in vasculitis of small and medium blood vessels (coronary arteries at most risk for aneurysm) and is the leading cause of acquired heart disease in children?

A

Kawasaki disease

77
Q

T or F: Kawasaki Disease is self limiting (6-8w), happens in kids less than 5, and has an unknown etiology

A

T

78
Q

Symptoms of Kawasaki

A

Pink eye, oral mucosa change, enlarged lymph nose, patchy rash, peeling skin

79
Q

Acute phase heart effects

A

Hyperdymanic precordium, tacky, Gallop rhythm, innocent flow murmur fever, depressed myocardial contractility d/t myocarditis, mitral regurgitation pan systolic murmur, prolonged PR with unspecific T wave changes,

80
Q

Acute phase (day 0-14) assessment

A

Persistent high fever (102) that is unresponsive to antipyretics (lasts longer than 5 days)* must have
Swelling of conjunctiva without draining
Rash (polymorphous exam them)
Swollen red hands ad feet
Cervical lymphadenopathy
Inflammation of mouth, lips, and tongue (strawberry tongue)

81
Q

Subacute phase (2-4w)

A

Begins with resolution of fever and ends when all outward symptoms are gone.
Arthritis or arthralgia on large weight bearing joints
Hands and feet peel
Irritability persists

82
Q

Convalescent Phase:

A

Begins when all clinical signs of KD have resolved and ends when blood values normal (ESR, CRP)
ESR and CPR remain elevated, thrombocytosis still present, arthritis still present

83
Q

Lab findings of Kawaskai

A 
Leukocytosis : WBC >15,000
Anemia can develop
ESR and C-reactive protein elevated
Thrombocytosis- Platelet count 500,000 to >1 million (highest 2-3rd week and gradual return 2-8w)
Echo: screen for coronary enlargement
84
Q

Treatment of Kawasaki

A

Aspirin (anti-inflam/anti-platelet)
Dose 80-100 mg/kg a day (duration varies from 48h-72h after child is afebrile to 14 days) then begin low dose 3-5 mg/kg per day until no evidence of coronary changes by 6-8w after illness onset

IVIG
Anti-inflammatory effect, modulation of cytokine production, suppress antibody synthesis, and neutralize bacterial super antigens

Dose: 2g per kg (give within first 10 days)

85
Q

Kawasaki Follow Up

A

Pharm Therapy: ASA, antiplatelet therapy or warfarin
Physical activity: should avoid contact or high impact d/t risk of bleeding
Testing: Cards follow up (echo, ECG, risk, stress test, maybe angiogram

86
Q

What is an acquired autoimmune immune-complex disorder occurring 2-5w after.a group A beta-hemolytic streptococcal infection?

A

Rheumatic fever

87
Q

T or F, streptococcal infection is found in the heart?

A

F.
RF is caused by production of antibodies against the toxin and these antibodies attack the heart valves because of similarities in antigenic markers

88
Q

Five major manifestations of RF

A

Migratory arthritis (involving large joints)
Carditis and valvulitis (pancarditis)
CNS involemtn (chorrea = sudden involuntary movements)
Erythema marginatum (temporary, disk=-like red macule that are non pruitiic and faded in center)
Subcutaneous nodules

89
Q

4 minor RF manifestations

A

arthralgia, fever, elevated acute phase reactants (ESR/CRP), prolonged PR

90
Q

AHA Jones criteria

A

2 major criteria or 1 major and 2 minor criteria for RF

91
Q

RF tx.

A

Aspirin (80-100mg/kg daily or 4-8 in adults) for anti-inflame and relief os symptoms
Severe carditis (cardiomegaly, CHF, 3rd degree block) need conventional HF meds
Antibiotics for group A beta hemolytic strep

92
Q

Refer to cards when:

A

Chest pain (with exertion, palpitations, sudden syncope with exercise)
Abnormal EKG or echo
Hx. of cards surgery.intervention
Family hc. or sudden cardiac death
Kawasaki
First degree relative with hypercholesterimia

Pediatric Heart (3 decks)

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