Cardiology Flashcards

1
Q

Indications of Reperfusion following PCI or fibrinolysis

A
  • Pain cessation
  • Reversal of ST segment elevation with return to baseline
  • short runs of ventricular tachycardia
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Anterior Wall MI

A
  • Acute Anterior STEMI
    • LAD occlusion
    • Likely results in ventricular septal defect (VSD)
    • Murmur at Left Sternal Border
    • ST elevation in V2, V3, and V4
  • Rhythm change
    • Second degree, Type II heart block possible (Mobitz)
  • Medications
    • Beta Blocker (cardioselective) beneficial (Metoprolol)
    • Anti-platelet (aspirin)
    • Anticoagulant (heparin)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Propanolol (Inderal)

A
  • Beta-adrenergic blocker (NOT cardioselective)
  • Affects beta receptors in heart muscle and lung tissue
  • More likely to cause bronchoconstriction
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Second Degree, Type II (Mobitz)

A
  • Often develops after acute anterior MI, generally due to occlusion of LAD (left anterior descending artery)
  • LAD supplies the HIS-Purkinje system (HIS bundle)
  • Conduction disease in HIS bundle is irreversible (not subject to autonomic tone or AV blocking medications)
  • Permanent pacemaker indicated
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Inferior Wall MI

A
  • Acute inferior STEMI
    • RCA occlusion may result in an RV infarct
    • Right Coronary Artery (RCA) occlusion
      • Result in papillary muscle dysfunction or rupture of the mitral valve
      • Supplies area of the left ventricle where mitral valve is attached
      • Acute mitral regurgitation
      • Does NOT result in systolic murmur at apex of heart or lung crackles
    • RIght-sided ECG may help confirm RV infarct
  • Rhythm change
    • Due to SA node or AV node ischemia
      • Sinoatrial block
      • Second degree, type I (Wenkebach)
      • Third degree, complete
  • Treatment
    • Development of complete heart block : Assess. If serious S/S develop, begin transcutaneous pacing (TCP)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Dilated Cardiomyopathy

A
  • Results in systolic dysfunction
    • ↓ contractility
    • Compensatory arterial constriction
    • Results in higher left ventricular preload
  • Therapy aimed at…
    • ↑ contractility
    • ↓ afterload (arterial constriction)
    • ↓ preload that is too high
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Electrolytes that may cause QT prolongation

A
  • ↓ magnesium
  • ↓ potassium
  • ↓ calcium
    • QT prolongation may result in torsades de pointes ventricular tachycardia
    • If self limiting, may result in transient syncopal episode
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Retroperitoneal Bleeding

A
  • Causes signs of hypovolemia and hypovolemic shock
  • Complication of a PCI
    • Femoral artery access site
  • Back Pain
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Dressier’s Syndrome

A
  • Type of pericarditis
    • Inflammation of the sac surrounding the heart (pericardium)
  • Results after an acute MI
  • Believed to be an immune system response after damage to heart tissue or to pericardium
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Mitral Valve Regurgitation

A
  • Murmur at the apex of the heart (midclavicular, 5th ICS)
  • Occurs when the valve should be closed
  • S/S
    • Shortness of breath
    • Recurrence of chest pain
    • Loud systolic murmur at apex of heart
  • Mitral valve should be closed during systole
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Permanent Pacemaker

A
  • First letter indicates chamber paced
  • Second letter indicates chamber sensed
  • Third letter indicates response to sensing
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Cardiogenic Shock

A
  • Results in ↓ CO with resultant drop in coronary artery perfusion and compensatory vasoconstriction
  • IABP therapy
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Intra Aortic Balloon Pump (IABP Therapy)

A
  • Deflation of balloon placed into descending aorta is beneficial
  • Deflation = ↓ afterload & ↓ work of the left ventricle
  • Inflation of the balloon is beneficial because it “boluses” blood into the coronary arteries
  • Inflation = ↑ diastolic augmentation & ↑ perfusion
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Percutaneous Coronary Intervention (PCI)

A
  • Addresses cause of the problem, not only treat signs and symptoms
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Hypertrophic Cardiomyopathy

A
  • Problem with filling
  • Diastolic dysfunction
    • NOT a problem with ejection, EF is normal
  • Drug therapy
    • Beta blocker (Metoprolol)
      • Decrease heart rate to ↑ filling time
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Ventricular Septal Defect (VSD)

A
  • Holosystolic murmur @ Left Sternal Border
17
Q

Right Ventricular Infarct

A
  • Right-sided ECG may help confirm
  • S/S:
    • hypotension
    • clear lungs
    • jugular vein distention (JVD)
18
Q

Acute Inferior STEMI Treatment

A
  • Definitive treatment: Emergent PCI
  • Fluid administration
    • ↑ coronary artery perfusion
      • correcting hypotension
      • ensure adequate RV preload
19
Q

What medication is contraindicated for a patient with heart failure due to diastolic dysfunction?

A

Digoxin

20
Q

Heart Failure (Diastolic dysfunction)

A
  • Problem with FILLING, not ejecting
  • S/S
    • hypertension
    • left ventricular hypertrophy
    • EF > 40%
21
Q

Digoxin

A
  • Positive inotrope
  • ↑ wall stress
  • Worsen filling of the left ventricle
22
Q

NYHA Heart Failure Class

A
23
Q

Chronic Right-Sided Heart Failure

A
  • Clinical S/S
    • Jugular Venous Distention (JVD)
    • Peripheral edema
    • Abdominal discomfort
    • *** JVD occurs in Acute RS HF but not peripheral edema and abdominal discomfort. Those are due to liver engorgment.***
24
Q

Cardiac Tamponade

A
  • S/S
    • Sudden drop in B/P
    • Distended neck veins (JVD)
    • Muffled heart tones
    • Pulsus paradoxus
    • ​Minimal chest tube output (S/P CABG patient)
  • Treatment
    • Emergent pericardiocentesis to drain fluid
    • Emergent return to OR (S/P CABG patient)
25
Q

Left Ventricular Failure S/S

A
  • Bibasilar crackles
  • Tachypnea
  • Frothy Sputum
26
Q

Pulsus Paradoxus

A
  • Systolic pressure that fluctuates with breathing pattern by more than 12 mmHG
  • Best seen with arterial line
  • Inspiration increases thoracic pressure
    • Combined with fluid surrounding the heart in cardiac tamponade, inspiration further decreases venous return to the heart, leading to drop systolic pressure by > 12mmHg during the inspiratory phase
27
Q

Pulsus Alternans

A
  • Change in amplitude of the systolic waveform from beat to beat
  • Indicative of severe left ventricular failure
28
Q

Pulsus Magnus

A

Bounding pulse

29
Q

Atrial Fibrillation

A
  • Even with normal B/P, development of Afib drops CO by 20% to 25%
    • loss in “atrial kick” provided by normal sinus rhythm
  • Treatment/therapy
    • Cardiac glycoside (such as digoxin)
      • Weak positive inotrope
      • May compensate for loss of atrial kick
    • Calcium-channel blockers
      • Keep the rate controlled
30
Q

Left Ventricular Systolic Dysfunction S/S

A
  • S3
    • Indicative of high left ventricular pressure
  • Cough
  • Bibasilar crackles
    • Sign of pulmonary edema secondary to ↑ left ventricular end diastolic pressure (PAOP)
  • EF < 40%
31
Q

Acute Coronary Syndrome

A
  • Interventions
    • Start fluid bolus
    • Give chewable aspirin
    • ECG (needed to help make diagnosis)
32
Q

Heart Valves during Systole (left ventricular ejection)

A

Aortic Valve = Open (allowing for ejection)

Mitral Valve = Closed

33
Q

Heart Valves during Diastole (filling)

A

Aortic Valve = Closed

Mitral Valve = Open

34
Q

Dopamine

A

Low dose ( < 10 mcg/kg/min) = Affect mainly beta-1 receptors in the heart, producing a positive inotropic effect

High dose ( > 10 mcg/kg/min) = Stimulates alpha receptors in artiers and causes vasoconstriction