Cardiology Flashcards
effect of valsalva early strain(2)
decrease venous return
decrease all murmurs except HCM and MVP
effect of valsalva late release(2)
increase venous return
increase right sided murmurs
effect of standing(2)
decrease venous return
similar to the strain phase of valsalva
effect of squatting(3)
increase venous return
increase afterload by kinkingof femoral arteries
increase reverse flow
effect of handgrip(3)
increase afterload
increase blood pressure
increase reverse flow across valve
murmurs getting louder with valsalva(2)
HCM
MVP
why during valsalva murmur get louder in MVP (2)
decrease left ventricular volume
increase of leaflet prolapse
why during valsalva murmur get louder in HCM (2)
decrease left ventricular volume
increase gradient
effect of standing resembles what other effect
valsalva
murmurs that get louder with squatting(3)
aortic regurgitation
mitrel regurgitation
VSD
murmurs that get softer with squatting(2)
HCM
MVP
why murmurs get softer with squatting in HCM (4)
more blood less murmur
increase preload
decrease gradient across outflow obstruction
decrease obstruction and decrease afterload
why murmurs get softer with squatting in MVP(2)
increase left ventricular size
decrease mitral valve leaflets prolapse
murmurs getting louder with handgrip(3)
aortic regurgitation
mital regurgitation
VSD
murmurs getting softer with handgrip(3)
HCM
increase gradient across outflow obstruction
decrease flow
auscultation in mitral valve prolapse(2)
single or multiple non ejection clicks
plus
mid to late systolic of mitral regurgitation
CHF with ejection fraction a 55 dx
diastolic dysfunction
number 1 cause of diastolic dysfunction
HTA
rx of diastolic dysfunction(2)
diuretics
antihypertensives
physiopatho in diastolic dysfunction
impaired ventricular filling due to poor myocardial relaxation or diminished ventricular compliances
cause of AFIB in diastolic dysfunction(3)
left ventricular dilation
leads to left atrial dilation
which in turn causes atrial fibrillation
HTA in the setting of bilateral nontender masses
autosomal dominant polycystic kidney disease
HTA in the setting of bilateral nontender masses best test to do
abdomen ultrasonogram
clue for autosomal dominant polycystic kidney disease(5)
HTA Hematuria proteinuria palpable renal masses progressive renal insufficiency
flank pain in autosomal polycystic kidney disease cause(3)
renal calculi
cyst rupture or hemmorrage
upper urinary tract infection
the early common finding in autosomal polycystic kidney disease
HTA
extra renal manif of autosomal polykidney disease(5)
cerebral aneurisms hepatic and pancreatic cysts cardiac valve disorder colonic diverticulosis ventral and inguinal hernias
management of APKD(3)
follow blood pressure and renal function
aggressive control of cardiovascular risks factors
ACE inhibitor for HTA
end stage renal diasease in APKD(2)
dyalisis
renal transplant
patient with HTA is seen in consultation in history , he exercices regularly an eats low salt diet .but he drinks 3-4 glasses of wine every day and 6-8 beers on week end .he quits smoking 3 years ago next step in management of HTA in this patient
counsel for reduction of alcohol
quid of excessive alcohol intake
> 2 drinks a day
quid of binge drinking
> 5 drinks in a row
lifestyle modification in HTA(6)
low salt diet diet rich in fruit and vegetables low fat dairy products regular aerobic exercices lose weight limit alcohol intake
patient with TA 160/85 while supine and 135/70 while standing dx
orthostatic hypotension
EKG for AFIB(3)
narrow qrs complex
no organised P waves
irregularly irregular rythm
stable patient with afib Management
Rate control
medication used for rate control
Betablocker
calcium blocker like Diltiazem
use of digoxin for rate control in AFIB(2)
AFIB due to heart failure
patient unable to tolerate B blocker or Calcium channel blocker
indication of cardiversion in Patient with AFIB(4)
less than 48 h
patient with hypotension
pulmonary edema
ischemic heart disease
what to do before beginning cardioversion in AFIB more than 48 h(2)
anticoagulation 3-4 weeks
plus
rate control
best test to see if AFIB is complicated with heart thrombus
TEE
patient with chest pain sus elevation of ST segment and ventricular premature beats administration of lidocaine will cause what in this patient
increase the risk in asystole
advantage and drawback of lidocaine in acute coronary syndrome(2)
decrease risk of VFIB
increase the risk of asystole
patient with history of rhinitis and eczema in childhood is coming for chest pain .ekg shows st segment depression .he is placed on aspirin bblocker etc.2 days later he develops respiratory distress with wheezing and prolonged expiratory.cause of that
medication side effect Bblocker and Aspirin
causes of acute dyspnee in hospitalized patients(7)
arrythmia bronchoconstriction CHF/hypervolemia infection/pneumonia asppiration pleural effusion PE anxiety
patient with cardiac disease or (electrolytes abnormalities) develops dizziness tachycardia(or braadycardia) during hospitalisation dx
arrythmia
patient with history of asthma ,is placed on aspirin and Bblocker develops wheezing and pprolonged expiratory phase during hospilaisation Cause of that
bronchoconstriction
patient with cardiac disease develops crakles high jugular venous pressure>8 cm h2o lower extremity edema cause of that
CHF
accidentaly patient has received 2000 cc de liquide develops dyspnea,develops crackles DX
hypervolemia
characteristics clinique of pleural effusion in tyhe context of acute dyspnee (2)
decreased breath sounds
dullness to percussion
clue for anxiety in the setting of acute dyspnea in hospitalised patient(4)
tachycardia
tachypnee
normal lung exam
normal oxygenation
EG in anterolateral MI
st segment elevation in 1 avl,v1-v3
what can happen in anterolateral MI(2)
muscle ischemia or rupture—>
mitral regurgitation
MI causing typically mitral regurge and why(2)
posteroseptal MI
a cause of solitary blood supply of of the post medial papillary muscle
consequence of lmitral regurge in anterolateral MI or post septal MI(4)
increase left atrial pressure
but no changes in left atrium size
in left ventricular sizes
and no changes in left ventricular ejection fraction
patient with chest pain during exercice but normal baseline resting EKG ,next step
exercice EKG
why patient with SLE are at risk for acute coronary syndrome(2)
most of the they are reiceiving prednisone
prednisone and Lupus cause acelarated coronary atherosclerosis
syncope during exercice(3)
aortic stenosis
HOC
VTAC
murmur in aortic stenosis(3)
2 nd intercostal space
radiation in caritids
crescendo-decrescendo
disease with pulsus parvus and tardus
aortic stenosis
quid of pulsus parvus and tardus
aotic stenosis
other finding in aortic stenosis
weak S2
S4
three possible symptoms in AS(3)
syncope during exercice
exertionnal angina
dyspnea
definitive dx of AS
echocardiogram
rx of symptomatic AS
valve replacement
patient with chest pain with normal QRS complex 80msec(n
first degree heart block
clue for first degree heart block(2)
prolonged PR interval
P wave always follows QRS unlikely other heart block
rx of first degree heart block with normal QRS duration
abservation
First degree AV block with prolonged QRS
electrophysiologic testing to determine the nature of the delay of conduction below the AV node
patient with history of respiratory infection one week ago develops Ta =100/60 distended neck veins and heart sounds distant dx
pericardial effusion
xray in pericardial effusion
enlarged cardiac silhouette
ekg clue for pericardial effusion
electrical alternans
quid of electrical alternans
qrs complexes whose amplitude vary from beat to beat on ekg
definitive dx in pericardial effusion
echocardiogram
quid hypertensive urgency(2)
severe HTA > ou egal 180/120
no symptoms ,no end organ damage
two divisions for hypertensive emergency(2)
malignant HTA
Hypertensiive encephalopathy
clue for malignant HTA(2)
severe HTA
plus
papilledema and retinal hemorrage
clue for hypertensive encephalopathy(2)
severe HTA
plus
cerebral edema and non localizing neurologic symptoms and signs
symptom in cerabral edema(4)
headache
nausea
vomiting
plus non localizing neurologic symptoms
quid of non localizing neurologic symptom(4)
restlessness
confusion
seizures
coma
organ atteint in malignat HTA(2)
rein
eye
rein problem in malignant HTA
nephrosclerosis
quid of manif of nephrosclerosis(3)
acute renal failure
hematuria
proteinuria
auscultation finding in aptient with aortic stenosis
systolic murmur ejection radiating to the apex and carotid arteries
teens and early twenties with AS cause
bicuspid valve
elderly with AS cause
Calcification of the trileaflet valve
muscle pain in patient taking statin
statin induced myopathy
mechanism of action of statin
inhibition of intracellular synthesis pathway
action of station intracellularly(3)
inhibit HMG co A reductase enzyme
prevent conversion of HMG co A to mevalonic acid
increase the number of cell membrane LDL receptors
why statin can induce myopathy
by decreasing co enzyme synthesis Q 10
role of Q10 coenzyme
involve in muscle cell energy
clue supraventricular tachycardia on EKG(4)
narrow QRS complex
tachycardia
no regular P waves as they are buried within QRS complex
retrograde P wave can occur
dx and management of supraventricular tachycardia(2)
adenosine
or vagal maneuvers
action of adenosine(3)
slows the sinus rate
increases AV nodal conduction delay
can cause a transient block in AV node conduction
role of adenosine in supraventricular tachycardia(2)
can help to identify P waves to clarify dx of atrial flutter or atrial tachycardia
terminate paroxysmal supraventricular tachycardia by interrupting the AV nodal reentry circuit
quid of vagal maneuvers(3)
carotid sinus massage
valsalva
eyeball pressure
patient smoker complain of cramping pain in his right thigh after walking 2 blocks ,the pain goes away once he stops and rests for several minutes
PAD
best initial management in PAD intermittent claudication
exercice therapy
indication of cilostazole in PAD
persistent symptom despite adequate supervised exercice therapy
indication of surgery in PAD
persistent symptom despite adequate supervised exercice therapy and cylostazole
HTA basic testing(4)
urinalysis for occult hematuria and urine protein creatinine ratio
chemistry panel
lipid profile
baseline ECG
when to search for secondary HTA(4)
severe or malignant HTA
resistant HTA requiring > ou egal a 3 drugs
sudden blood pressure rise in patient with previosly controled HTA
age of onset
patient with HTA ,hypokaliemia and hyperglycemia and weight gain dx
adrenal cortical disease
cushing disease
cause of cushing syndrome(4)
adrenal cortical hyperplasia
acth producing pituitary adenoma (cushing disease)
ectopic ACTH production
exogenous steroids
clue for cushing(7)
poximal muscle weaness central adiposity thinning of the skin psychiatreic problem hypokaliemia hypertension hyperglycemia
psychiatric problem in cushing(3)
sleep disturbances
depression
psychosis
quid of preload measurement(2)
right atrial pressure
pulmonary capillary wedge pressure
normal right atrial pressure
mean 4 mm of HG
normal pulmonary wedge pressure
mean of 9 mm de HG
quid of cardiac index
pump function measurement
normal cardiac index
2.8-4.2 l/mn/m2
quid of systemic vascular resistance
measure afterload
normal systemic vascular resistance
1150l/mn/m2
normal mixed venous oxygen saturation
60%-80%
the only parameter increase in Hypovolemic schock
everything is low except systemic vascular resistance
the only two parameters decrease in cardiogenic shock
everything is high except cardiac pump function
mixed venous oxygen saturation
the only shock syndrome with low vascular resistance and increased mixed venous oxygen saturation
septic shock
patient with hypotension, normal Pulmonary wedge pressure and increased mixed venous saturation
septic shock
hwat’s the underlying basic pathophysiology in septic shock
decrease systemic vascular resistance due to overall peripheral vasodilation
swanz ganz catether in septic shock(4)
low pulmonary wedge pressure
low systemic vascular resistance
increased cardiac output
high mixed venous oxygen saturation
origin of formation of AFIB focii
pulmonary veins
quid for atrial flutter origin
reentrant circuit that rotates around the tricuspid annulus
quid for paroxysmal supraventricular tachycardia origin
reentry circuit most commonly oinvolved the AV node or via accessory bypass tract
patient on digoxin and furosemide present with wide complex tachycardia what to check
serum electrolytes
effect of furosemide(2)
low K
low MG++
effects of low K and low Mg++
ventricular tachycardia
risk factor for digoxin toxicity
low K
consequence of digoxin toxicity
ventricular tachycardia
side effect of thiazide (5)
hyperglycemia increased LDL cholesterol and plasma triglycerides hyponatremia hypokaliemia hypercalcemia
hypergluc in thiazide(4)
G= glycemia
L=lipidemia
U=uricemia
C=Calcemia
in swanx ganz catheter clue for cardiogenic shock(2)
reduced cardiac index
elevated pulmonary wedge pressure
how ‘s systemic vascular resistance in cardiogenic shock
high to maintain adequate perfusion of tissue
the most contributory factor in CHF edema
increased renal sodium retention
cause of increased renal sodium retention in CHF(2)
low renal perfusion—-> stimulation of renin aldosterone system—>hypoperfusion renal secondary to cardiac output
renal arteries are constricted
patient with palpitations HR 160 suddenly with no history of haert problem.Symptoms improves when immersing face in cold water dx
paroxysmal supraventricular tachycardia
the cold therapy work s by affecting what
atrioventricular node conductivity
cause of supraventricular tachycardia
accessory conduction pathways
why you can have hepatomegaly,ascites, increased JVP in constrictive pericarditis
decreeased diastolic filling leafing to cardiac output impairment
common cause of constrictive pericarditis(4)
radiation therapy
viral pericarditis
cardiac surgery
idiopathic
kussmaul sign
failure of JVP to decrease during inspiration
other name of constrictive pericarditis
inelastic pericardium
dx of constrictive pericarditis(3)
calcified pericardium in xray
thickened pericardium on CT or MRI scanning
cardiac catheterisation
rx of constrictive percarditis(2)
diuretics
or
pericardiectomy
after anterior wall MI patient develops pleuritic chest pain improving when sitting and leaning forward.EKG shows diffuse ST segment elevation dx
acute pericarditis
laps de temps pour developper acute pericarditis post MI
within the first several days
EKG for acute pericarditis(2)
diffuse ST segment elevation
PR depressions
quid of lone AFIB
presence of paroxysmal persistent or permanent AFIb with no evidence of cardiopulmonary or structural heart disease
rx of lone AFIB
nothing
paroxysmal AFIB
reccurrent > a 2 episodes that terminate spontaneously in
persistent AFIB
episodes lasting more than 7 days
longstanding persistent AFIB
pesistent for more than 1 year duration
permanent AFIB
persistent with no further plans for ryhtm controls
CHADS 2 score 0(2)
no anticoagulation
aspirin preferred
CHADS 2 score 1 (2)
anticoagulation preferred
or
aspirin
CHADS 2 score 2-6
anticoagulation
cause of restrictive cardiomyopathy(4)
sarcoidosis
amyloidosis
hemochromatosis
fibrosis endomyocardial
clue for restrictive cardiomyopathy in echo
symmetrical thickening of the left ventricular walls and slightly reduced systolic function
the only reversible cause of restrictive cardiomyopathy
hemochromatosis
echo with interventricular septum thickness
hypertrophic cardiomyopathy
primary rx of hemochromatosis
phlebotomy
quid of the anti-ischemic nitrate action
systemic vasodilation rather than coronary dilation
systemic venodilation lowers (ventricular)preload and left ventricular end diastolic volume reducing wall stress and myocardial oxygen demand
dilation of capacitance vessels
action of nitrate
reduced left ventricular volume
supraventricular tachycardia in patient hemodynamically unstable management
DC cardioversion
anterior wall myocardial infarction with pulmonary edema what medication to give and why
furosemide
furosemide causes venodilation which further decreases the preload
anterior wall myocardial infarction with pulmonary edema what medication u cant give and why
betablocker
can worsen acute heart failure
other medication can be used in pulmonary edema caused by anterior wall myocardial infarction and why
Morphine
decrease prload and anxiolytic
patient with syncope with history of respiratotry infection 2 weeks ago EKG shows electrical alternans best next step in this patient
percardicenthesis
quid of electrical alternans
une onde qrs longue suivie d’une courte
EKG of pericardial effusion(3)
electrical alternans
sinus tachycardia
low QRS voltage in large pericardial effusion
quid of sinus tachycardia with electrical alternans
large pericardial effusion
problem in HIC(2)
abnormal mitral leaflet motion= systolic anterior motion of the mitral valve septal hypertrophy
cause of systolic dysfunction
MI
…
catetherisation during systolic heart failure(3)
CI decreased
left ventricular end diastolic volume increased
total peripheral resistance increased
how ‘s the left ventricular end diastolic heart failure
normal
patient with tachysystolic AFIB what to do to improve the left ventricular function in those patients
control the rate and the rythm
why tachysystolic AFIB causes significant left ventricular dialtion and depressed EF(4)
tachycardia
neurohumoral activation
absence of atrial kick
atrial ventricular desynchronisation
importance of atrial kick
it accounts for 25% of LV end diastolic volume
tachysystolic AFIB (3)
irregular irregualr rythm
tachycardia
no P waves ion EKG
cardiac problem in hemochromatosis(3)
cardiac conduction abnormalities
dialted cardiomyopathy
heart failure
the greatest risk factor for printzmetal angina
smoking
young female with nocturnal chest pain lasting 15-20 mn .EKG shows St segment elevation in lead 1 avl,v4-v6 during the episode rx
diltiazem
or
nitrate
why to not give bblocker or aspirin in printz metal angina
cause vasoconstriction
other name of printz metal
variant angina
after long trip to central asia female using OCP develops hemoptysis and pleuritic chest paincause of these symptoms
pulmonary infarction
number one cause of pleuritic chest pain
PE
gold standard Dx in PE
helical CT
patient with chest pain palpitations is seen in emergency .Physical exam reveals HTA ,dilated pupils ,small amount of blood at the external nares St segment elevation in V1-V4.explanation of the symptoms
drugs induced vasospasm
cocaine abuse
why you cant give bblocker to patietn in cocaine abuse
unopposed alpha agonist will worsen vasospasm in cocaine abuse
cause of St segment elevation(4)
MI
Cocaine abuse
acute pericarditis
printzmetal
clue for aortic regurge
wide pulse pressure
manif of wide pulse pressure in reality
water hammer pulse
=pounding heartbeat
way for the patient hear better the pounding heart(2)
lying supine and
lying on the left
most common cause of aortic dilation in The US(2)
aortic root dialtion
bicuspid aortic valve
the greater non pharmocologic rx with greatest impact on HTA and why(2)
weight loss
reduce HTA of 5-20 per 10 kg loss
the second non pharmocologic rx with greatest impact on HTA and why(2)
DASH diet
reduce HTA 8-14 mm de hG
thethird non pharmocologic rx with greatest impact on HTA and why(2)
exercice
reduce HTA 4-9 mm de hg
the 4 e non pharmocologic rx with greatest impact on HTA and why(2)
dietary sodium
reduce HTA 2-8 mm de hg
the 5 e non pharmocologic rx with greatest impact on HTA and why(2)
alcohol intake
reduce HTA 2-4 mm de hg
quid of DASH diet(2)
Diet rich in fruits and vegetables
and low saturated fat and total fat
time to work out in HTA(2)
30 min /day
5-6 days /semaine
dietary sodium restriction in HTA
alcohol intake restriction in HTA(2)
2 drinks /day in men
1 drink /day in women
first line rx for newly dx hypertension satge 1
lifestyle modification
patient with pedal edema ascite emigrating from china to come in the US.chest xray reveals decreased heart sound and an accentuated sound directly after the second heart sound in ear;y diastole .chest xray shows ring calcification around the heart and jugular venous pressure tracings show prominent x and y descents cause of the patient symptoms and Dx
tuberculosis
constrictive pericarditis
clinical presentation of constrictive pericarditis(4)
fatigue and dyspnee on exertion
peripheral edema and ascites
high jugular venous pressure
pericardial knock
dx findings in constrictive pericarditis(2)
X and Y descents during jugular venous pulse tracing
imagind shows pericardial thickening and calcification
pericardial knock
early heart sound after S2
heart dysfunction in constrictive pericarditis
diastolic
endemic areas for TB(3)
africa
india
china
EKG for Mobitz type 1(wenkeback)
PR interval growing slowly progressively leading up to a dropped beat
problem in mobitz one
impaired AV node conduction
sudden tearing chest pain in aptient with chest xray showing widened mediastinum dx and medical condition causing that
dissection aortic
HTA
drugs increasing the riosk of bleeding when taking warfarin(9)
acetaminophen NSAIDS antibiotis/antifungal amiodarone canberry juice ginkgo biloba viit E omeprazole thyroid hormone selectice serotonin reuptake inhibitors
drugs decreasing the effect of warfarin(6)
rifampin carbamazepine oral contraceptives ginseng st jhon's wort green vegetables(spinach)
dose of acetaminophen to cause bleeding with warfarin ingestion
> 2 g /jour for 1 week
the most important factor for survival in out hospital sudden cardiac arrest
time to rythm analysis and defibrillation=elapse time to effective resuscitation
en d’autres mots:prompt effective resuscitation with adequate bystander CPR,prompt rythm analysis and defibrillation
number one cause of outhospital sudden cardiac arrest(2)
sustained VTAC
sustained VFIB
both cause by MI or ischemia
murmur in aortic dissection
diastolic murmur in left sternal border
3 clinical findings in aortic dissection with 2 you make the DX
tearing chest pain radiating in the back
variation in pulse or blood pressure between the right and the left arm
widened mediastinum
complication of dissection aortic
extend to pericardium=tamponnade
extend to coronary arteries=stroke
extend to carotid arteries=stroke
dissection aortic plus hemiplegia dx
stroke
incidence of aortic dissection when 2 clinical symptoms are present see question above
80 %
patient found with pulsatile mass above umbilicus creat 2.0 and TA:160/90 dx and best test to confirm the DX(2)
abdominal aneurism of aorta
abdominal ultrasound
quid of BNP
release by dilated ventricle
value for BNP to Dx CHF(4)
> 100 pg /ml
specificity 76
sensitivity 90
predictive value 83
importance of BNP
helps to differentiate dyspnea of cardiac origin with any other origin
cause of right Heart failure in COPD
pulmonary artery systolic pressure
sequence of event causing right heart failure in COPD
hypoxemia causes constriction of the pulmonary artery and with time pulmonary hypertension—> will lead to right ventricular hypertrophy and right ventricular failure
does right ventricular failure cause pulmonary edema
it s not a common cause of pulmonary edema
management of STEMI(6)
oxygen nitrates antiplatelet therapy anticoagulation bblockers prompt reperfusion with PCI
antiplatelet therapy used in STEMI
platelet P2y12 receptor inhibitor
anticoagulation used in STEMI
bivalirudin is preferred over heparin
ideal first rx for STEMI
prompt reperfusion with PCI
clue for benign essential tremor(3)
tremor worst with activity
improves with with alcohol
family inheritance autososmal dominant
HTA plus benign esential tremor Rx
propranolol
the most effective non pharmacological rx of HTA
weight loss
clue for venous insufficiency(4)
pedal edema
medial ankle ulcer
dilated and tortuous superficial veins
normal physical exam
initial rx of venous insufficiency(3)
leg elevation
exercice
compression stockings
method to hear aortic regurgitation murmur(4)
diastolic murmur
best heard along the left sternal border at the third and fourth interspaces
best heart when you apply firm pressure with the diaphragm of the sthetoscope while patient is sitting up leaning forward and holding the breath in full expiration
cause of aortic regurge in developed countries in young adults
bicuspid aortic valve
cause of aortic regurge in developing countries in young adults
rheumatic heart disease
common cause of aortic regurgitation involving the aortic valve leaflet(8)
rheumatic heart disease endocarditis bicuspid aortic valve trauma myxomatous degeneration ankylosing spondylitis acromegaly medications
common cause of aortic regurgitation involving trhe ascending aorta or aortic root disease (8)
hta aortitis syphilitic ankylosing spondylitis dissection aortic ehlers danlos IBD reactve arthritis Marfan syndrome
Medication to hold for 48 h prior to cardiac testing(3)
Bblocker
calcium blocker
nitrates
medication to hold 48 h prior to vasodilator stress test
dipyridamole
medication to hold 12 h prior to vasodilator stress test
caffeine containing food or drinks
medication you can continue prior to to cardiac stress testing(5)
ACE inhibitor ARBs digoxin statins diuretics
gold standard Dx of CAD
coronary angiography
indication of amiodarone(3)
ventricular arythmias
rythm control in AFIB
left ventricular systolic dysfuction
toxicity of amiodarone(7)
hypo or hyper thyroidism hepatotoxicity bradycardia heart block pneumonitis neurologic symptoms visual disturbances
visual probelm associated with amiodarone(2)
corneal microdeposits
optic neuropathy
heart problem with amiodarone(2)
Qt prolongation
risk de torsades de pointes
dermatologic problem associated with amiodarone
blue gray skin discoloration
neurologic problem associated with amiodarone
peripheral neuropathy
gastrointestinal and hepatic problem associated with amiodarone(2)
elevated transaminases
hepatitis
mark for IV drug user in USMLE
needle tracks on arms
IV drug user with fever andround lesions in lungs and sinus tachycardia.what accompanying finding is expected
systolic murmur that increases with inspiration
bug in infective endocarditris in IV drug user
staph aureus
what increases the risk of infective endocarditis in IV drug user
HIV infection
holosystolic murmur increasing with inspiration quid of that
tricuspid involvement
IE with round lung opacity
septic pulmonary emboli
what must be done in young patient with systemic HTA
evaluation for coarctation of aorta
assessment of coarctation of aorta in physical exam(3)
search for brachio femoral delay
upper extremity hypertension lower extremity hypotension
continuous cardiac murmur from large collaterals
acqiured cause of coarctation of aorta
maladue de takayasu
chest xray for aaortic coarctation
notching of the 3 th-8th ribs from enlarged intercostal arteries
confirmatory dx for aortic coarctation
echocardiography
rx of aortic coarctation(2)
balloon angioplasty
plus or minus
stent
complication of CABG
AFIB
AFIB in hemodynamically unstable patient rx
DC cardioversion
EKG of AFIB(3)
absent P waes
an irregularly irregylar rate
narrow QRS complex