Cardiology Flashcards

1
Q

What are some indications for an EKG?

A
  • Chest pain
  • Syncope
  • Cyanotic episodes
  • Drug ingestion
  • CHD evaluation
  • Palpitations
  • Pericarditis
  • Kawasaki disease
  • myocarditis
  • Rheumatic heart fever
  • Family history of sudden death
  • electrolyte abnormalities
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2
Q

What is the criteria for RVH on EKG?

A
  • Pure R wave in V1 > 10mm
  • Upright T wave in V1
  • RSR’ pattern in V1, where R’ > 15mm ( 10mm (>1yo)
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3
Q

What can prolong PR interval?

A
  • Increased vagal tone
  • Heart block
  • Endocarditis with abscess
  • Hyperkalemia
  • Digoxin toxicity
  • Wolf-Parkinson-White
  • Glycogen storage disease
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4
Q

What can prolong QRS? (>0.08msec if 0.10msec if >8yo)

A
  • Bundle branch block

- Junctional or ventricular rhythm (not through His-Purkinje)

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5
Q

How do you correct QT for heart rate?

A

Bazett formula:

From start of Q to end of T

QTc=QT/(RR)^1/2

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6
Q

What is a normal QTc?

A

upper limit of normal: 450msec

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7
Q

Normal axis on EKG?

A

0 to 90: +I, +II and +aVF

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8
Q

Left axis deviation on EKG?

A

0 to -90: +I, +aVL and -aVF

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9
Q

Right axis deviation on EKG?

A

90 to 180: -I, +III and +aVF

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10
Q

Extreme right axis deviation on EKG?

A

-90 to -180: -I, -aVF and +aVR

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11
Q

What is Congestive Heart Failure?

A

Clinical syndrome defined as the inadequate oxygen delivery by the myocardium to meet the metabolic demands of the body

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12
Q

What’s the pathophysiology of CHF?

A

*Hypoperfusion of end organs:

-It stimulates the heart to maximize contractility and
heart rate in an attempt to increase cardiac output

  • It also signals the kidneys to retain salt and water through the reninangiotensin system in an attempt to increase blood volume
  • Catecholamines
  • Released by the sympathetic nervous system also increase heart rate and myocardial contractility
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13
Q

What congenital lesions may cause increased pulmonary blood flow leading to CHF?

A
  • A large ventricular septal defect (VSD)
  • A large patent ductus arteriosus (PDA)
  • Transposition of the great arteries (TGA)
  • Truncus arteriosus
  • Total anomalous pulmonary venous connection (TAPVC)
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14
Q

What obstructive congenital lesions can lead to CHF?

A
  • Severe aortic, pulmonary, and mitral valve stenosis
  • Coarctation of the aorta
  • Interrupted aortic arch
  • Hypoplastic left heart syndrome
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15
Q

What are other congenital causes of CHF?

A
  • Arteriovenous malformations

- Mitral or tricuspid regurgitation, which overload portions of the heart

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16
Q

What are some acquired heart diseases that can cause CHF?

A
  • Viral myocarditis (common cause of CHF in older children and adolescents)
  • Other cardiac infections (endocarditis, pericarditis)
  • Metabolic diseases (hyperthyroidism)
  • Medications (doxorubicin, a chemotherapeutic agent)
  • Cardiomyopathies
  • Ischemic diseases
  • Dysrhythmias (including tachycardia and bradycardia)
17
Q

What are some miscellaneous causes of CHF?

A

-Severe anemia (which may cause high-output CHF)
-Rapid infusion of intravenous fluids (especially in premature infants)
-Obstructive processes of the airway (such as enlarged tonsils or adenoids, laryngomalacia, and cystic fibrosis), which may cause CHF as a result of chronic
hypoxemia that results in right-sided heart failure

18
Q

What’s evidence of pulmonary congestion in CHF?

A
  • Tachypnea
  • Cough
  • Wheezing
  • Rales
  • Pulmonary edema on chest radiograph
19
Q

What’s evidence of impaired myocardial performance in CHF?

A
  • Tachycardia
  • Sweating
  • Pale or ashen skin color
  • Diminished urine output
  • Enlarged cardiac silhouette on CXR
20
Q

What’s evidence of systemic venous congestion in CHF?

A

-Hepatomegaly and peripheral edema

21
Q

What are other signs and symptoms of CHF?

A
  • Failure to thrive
  • Poor feeding (common symptom in newborns)
  • Exercise intolerance (common symptom in older children and adolescents)
22
Q

What are late manifestations of CHF?

A

-Cyanosis and shock

23
Q

What are the goals of management of CHF?

A

To improve myocardial function and relieve

pulmonary and systemic congestion

24
Q

What is the role of cardiac glycosides (digoxin) in the treatment of CHF?

A

Increase the efficiency of myocardial contractions and relieve tachycardia

25
What is the role of loop diuretics (furosemide, ethacrynic acid) in the treatment of CHF?
Reduce intravascular volume by maximizing sodium loss, which in turn leads to diminished ventricular dilation and improved function
26
What is the role of loop inotropic medications (dobutamine, dopamine) in the treatment of CHF?
They are administered intravenously and may be used to treat severe CHF