Cardio Flashcards
Angina Pectoris Path
Exertional chest pain d/t increased demand and decreased supply
Typically caused by fixed plaque
Pain lasts LESS THAN 30 minutes and is relieved by rest and/or Nitro
Classes of Angina
I: only with unusally strenuous activity, no limitations
II: with more prolonged/rigorous activity, slight limit
III: with usual daily activity, marked limts
IV: at rest, unable to do activity
Dx of Ischemic Heart Disease
EKG: ST depression
Stress test: most useful noninvasive tool
Myocardial Perfusion imaging
Coronary angiography: GOLD STANDARD
Surgical Tx of ischemic heart disease
PTCA: not involving the left main coronary artery, vent function is near normal
CABG: for left main coronary or critical stenosis
Nitro and ischemic heart disease
Increases O2 and increases collateral blood flow, reducing coronary vasospasm and increasing dilation.
If no relief with first dose give a 2nd and 3rd evert 5 minutes. If still not relief – ACS!!!
Contraindications to Nitro
SBP <90
RV infarction
PDE-5i’s
BBs
increase diastolic timing, first line drug of choice for ischemic heart disease management
CCBs
used by patients that cannot use BBs, Prinzmetal angina
Prinzmetal angina
almost ALWAYS occurs at rest, usually between midnight and early morning
ASA and ischemic heart
prevents progression of stable angina to ACS
CAUTION in pts with PUD or increased bleeding risk
Sinus Arrhythmia
Same as NS except irregular
HR increases during inspiration and decreases with expiration
Since sinus syndrome
“Brady-Tachy”
Combo of sinus arrest with alternating paroxysms of atrial tach and brady. Commonly caused by sinoatrial node disease
NEED PACER
AV block
Interuption of normla impulse from SA to AV
A Flutter
Saw tooth waves @ 250-350 BPM with no P waves.
Rate: regular
Stable: vagal, BB, CCB
Unstable: cardiovert
Cure: ablation
Afib
Rhythm: Irregular
Narrow QRS
No p wave
80-140bpm
Can lead to ischemic stroke
Ashmens Phenomenon
occasional aberrantly conducted beats and short R-R cycles
AFIB MANAGEMENT of stable patients
rate control
BB: metoprolol but be careful in pts with reactive airway disease
CCB Diltiazem
Digoxin: Preferred in pts with hypotension or CHF
Rhythm Control: cardiovert, flecinide, sotalol, amiodarone, ablation
Afib management o Unstable patients
Cardiovert
Anticoags for Afib
Warfarin w/ goal INR 2-3
Dual Antiplatelet: ASA + clopidogrel
Long QT syndrome
d/t congenital or macrolides/TCAs and electrolyte abnormalities. Can lead to sudden cardiac death
Tx: d/c offending med and correct abnormalities
Congenital: AICD
PSVT
HR>100
Regular, narrow QRS, P waves hard to see
Paroxysmal: sudden onset and termination
PSVT types
AV nodal reentry: 2 pathways both within AV node –> MC
AV reciprocating Tach: 1 pathway in AV and second outside AV
PSVT tx
Stable: vagal, adenosine 1st line, BB/CCB
Unstable: cardiovert
Cure: ablation
Wandering atrial pacemaker/ Multifocal Atrial Tach
WAP: <100 BPM >/= 3 P waves
MAT: >100 BPM >/= 3 P waves –> SEVERE COPD
Wolff Parkinson White
Bundle of Kent excites ventricles
DELTA waves
Wide QRS
Short PR
Tx: vagal, antiiarhythmics, procainamide
AVOID ABCD: adenosine, BBs, CCBs, Digoxin
Lown Ganong Levine Syndrome
Short PR with normal QRS
Bundle of James connects with His
PVC
Premature beat from ventricle
WIDE bizarre QRS earlier than expected with a pause
T wave is opposite direction
Ventricular Tach
> /= 3 PVCs at a rate of >100bpm
No pulse: defib/CPR
Torsades De Pointes
MC d/t Hypomagnesemia, hypokalemia, V Tach that twists around baseline
Tx: IV mag
V Fib Tx
Unsynchronized cardiovert and CPR
PEA
NSR without a pulse
CPR/Epi
Dilated Cardiomyopathy etiology (95% of cases)
Post viral, MC is enterovirus
Also: chagas, ETOH, prego, cocaine
TAKE A GOOD Hx
Dilated Cardiomyopathy Sx
Systolic heart failure sx
S3
Laterally displaced PMI
Mitral/Tricuspid Regurg
Dilated Cardiomyopathy Dx/ Tx
Echo -> LV dilation, decreased EF
Tx: ACEi, Diuretics, BBs
Restrictive Cardiomyopathy etiology
Impaired disatolic function with preserved contractility
Amyloidosis MC cause
Also: sarcoidosis and infiltrative disease
Restrictive cardiomyopathy Sx/ PE
Right sided failure sx Kussmauls sign (JVP increases with inspiration)
Restrictive Cardiomyopathy dx/ tx
CXR: atrial enlargement, pulm congestion
Tx: tx sx and underlying issue
HOCM etiology
Genetic disorder of LV and/or RV hypertrophy
Subaortic outflow obstruction
HOCM Sx
Dyspnea MC CC, angina, syncope, arrythmias, sudden cardiac death
HOCM PE
HARSH systolic Cresendo-Decresendo murmur heard at lower left sternal border, similar to AS except HOCM decreases with squatting
HOCM Dx and TX
Echo: >15mm wall thickness
Tx: BB is 1st line, the myomectomy, and alcohol septal ablation
CHF L vs R
L: MC causes CAD and HTN
R: MC causes is left sided failure
CHF systolic vs diastolic
Systolic: decreased EF S3 MC form Etiology: post MI, dilated cardiomyopathy, myocarditis
Diastolic: normal EF
S4
Stiff ventricle
Etiology: HTN, LVH, Elderly
High Output CHF
metabolic demands of hte body exceed normal cardiac fxn
Thyrotoxicosis, wet beriberi, severe anemia, AV shunting
Low output CHF
Inherent problem of myocardial contraction, ischemia, chronic HTN
NY heart CHF Classificaiton
I: no sx/ limits
II: mild sx, slight limits
III: sx cause marked limits with activity, comfy at rest
IV: sx at rest, severe limits
Sx of L CHF
Increased pulmonary pressure from fluid backup
Dyspnea, Pulm edema/congestion (rales, rhonchi)
Productive cough, Transudative pleural effusions
HTN, S3/S4, cheyne stokes breathing, dusky pale skin, diaphoresis, sinus tachy, cool extremities
Sx of R CHF
Peripheral edema, JVD, GI/Hepatic congestion
N/V, RUQ t, Anorexia