Cardio Flashcards

1
Q

Diagnostic and laboratory studies

Abdominal Aortic Aneurysm

A

1: Initial imaging study of choice: Abdominal Ultrasound (also used to determine presence, size, and extent, and monitor progression)
2: GOLD STANDARD: Angiography

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2
Q

Formulating most likely diagnosis

Abdominal Aortic Aneurysm

A
  • Men >65 yo; smoker
  • Asx until expanding/ruptures:
  • Ruptured = sudden, severe, constant, back/flank abdominal/groin and/or in shock
  • palpable pulsatile abdominal mass on PE
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3
Q

Pharmaceutical therapeutics

Acute MI

A

IV fluids, O2, ASA, NTG, beta blocker, ACE- I, statins, anticoags, antiplatelets,

(every patient s/p MI leaves on S.A.B.A)

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4
Q

Formulating most likely diagnosis

Acute MI , anterior

A

ST elevations in V1-V4

ST depressions in 2 other contiguous leads

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5
Q

Pharmaceutical therapeutics

Adverse Drug Effect: Antiarrhythmics

A
  • Amioderone SE’s = “Eye, thy, liver, lung” / optic neuritis, thyroid disease, hepatitis, pulmonary fibrosis
  • Na+ channel blockers OD = Wide complex QRS
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6
Q

Formulating most likely diagnosis

AFIB

A

EKG: Irregularly irregular rhythm, no P waves, usually narrow complex QRS.

Presents as either an arrhythmia or an embolic event (Green, red, yellow light)

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7
Q

Pharmaceutical therapeutics

Aneurysm, thoracoadbominal

A

BBs - help to reduce shearing forces, decrease expansion, and ruptures risk

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8
Q

Pharmaceutical therapeutics

Angina

A

Stable = Nitro/BB/CCB

Unstable = “MONA”

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9
Q

Clinical Intervention

Aortic Aneurysm

A

Lifestyle mods (quit smoking is the main one), decrease DM, control cholesterol, regular exercise

Meds: BB, statin, daily low does ASA, plavix
anticoags,

Surgical Revascularization

(many AAA’s that rupture die of CV collapse prior to arrival 2 hospital)

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10
Q

Clinical Intervention

Aortic Stenosis

A
  • Aortic Valve replacement only effective treatment
  • Mild AS: No exercise restrictions
  • Severe AS: Avoid physical exertion/vasodilators (ex. nitrates or negative inotropes (Ex. BB/CCB)
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11
Q

Clinical Intervention

Arrhythmia, Complete heart block

A
  • Acute/symptomatic: Temporary pacing –> permanent pacemaker
  • Definitive tx: Permanent pacemaker
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12
Q

Formulating most likely diagnosis

Arrhythmia, PAC’S

A

PAC is a premature “beat”/QRS that has a P wave before it

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13
Q

Formulating most likely diagnosis

Arrhythmia, Paroxysmal SVT

A

Heart rate >100 bpm; rhythm usually regular with narrow QRS complexes; P waves hard to discern due to rapid rate

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14
Q

Clinical Intervention

Arrhythmia, Paroxysmal SVT

A

1: Vagal nerve stimulation with maneuver
2: Adenosine
3: CCB ( verapamil)
4: Cardioversion

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15
Q

Clinical Intervention

Arrhythmia , V-FIB

A

These patients are in cardiac arrest, CPR and defibrillation (unsynchronized cardioversion)

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16
Q

Clinical Intervention

Arrhythmia , V-TACH

A
  • Stable sustained V-tach: Antiarrhytmics (amiodarone**, lidocaine, procainamide)
  • Unstable V-tach WITH a pulse: synchronized cardioversion
  • V-tach NO PULSE : Defibrillate (unsynchronized cardioversion + CPR
  • If proceeds to tornadoes: IV MAG
17
Q

Pharmaceutical therapeutics

A-fib

A

1: Slow heart rate (BB/CCB)
2: Anticoag (almost always)
3: Convert (pharmacologically or electrically)

18
Q

Applying basic scientific concepts

Atrial Septal Defect

A

Oxygenated L-atrium blood crosses ASD and mixes with deoxygenated R-atrium blood; greater blood volume in R-atria can lead to R-HF, arrhythmia, pulmonary HTN

19
Q

Formulating Most likely diagnosis

Atrial Septal Defect

A
  • Systolic ejection crescendo=decrescendo murmur best heard @pulmonic area (LUSB);
  • Widely fixed split S2 that does NOT vary with respiration
20
Q

Clinical intervention

Cardiac Arrest

A

1: CPR
if in V-TACH or V-FIB : DEFIBRILLATE
Give epinephrine and other medications as indicated

21
Q

Formulating most likely diagnosis

Cardiac Tamponade

A
  • Beck’s Triad: JVD, muffled heart sounds, hypotensive
  • pulses paradoxus: >10mmHg decrease in SBP w/ inspiration
  • Electrical alternans on EKG
22
Q

Using diagnostic and laboratory studies

Cardiac Tamponade

A

ECHO: Ventricles collapse in diastole (+ presence of an effusion)

23
Q

Clinical Intervention

Cardiogenic Shock

A

1: Emergent ECHO
2: O2, isotonic fluids, (only shock we do NOT use agressive fluids)
3: Positive inotropes
4: Pressors
5: Admit to ICU and tx underlying cause

24
Q

Formulating most likely diagnosis

Coarctation of the Aorta

A
  • **Suspect in a child with secondary HTN
  • BL lower extremity claudication
  • Systolic murmur that radiates to back/scapula or chest
  • Systolic BP in upper extremities > lower extremities
  • Delayed or weak femoral pulses
  • CXR: Rib notching/ “3” sign
25
Q

Clinical Intervention

Coarctation of the Aorta

A

Surgical Correction : Balloon Angioplasty +/- stent

PGE1 (prostaglandin) pre-op to decrease sis/improve lower extremity blood flow

(** remember prOstaglandins keep PDA Open)

26
Q

Applying Basic scientific Concepts

CHF

A

Poor pumping –> blood stalls and backs up in the circulation