Cardiac, Thoracic, and Vascular Anesthesia Flashcards
DDx for Hypotension
- Pulmonary: Hypoxia, Hypercarbia, Tension Pneumothorax
- Hypovolemia: Fluid deficit, acute blood loss
- Cardiac: rate/rhythm abnormality, inotropic failure, myocardial ischemia, contusion, tamponade, rupture, congestive heart failure (CHF), cardiomyopathy, valvular injury
- Shock: hypovolemia, cariogenic, septic
- Surgical compression of the heart, aorta, IVC, or abdominal contents
- Embolus: pulmonary, air, fat, amniotic
- Electrolyte and hormonal abnormalities: hypoglycemia, hypocalcemia, adrenal insufficiency, ADH suppression, hypermagnesemia
- Anaphylaxis
- Deep anesthesia, drug overdose, medications (ACEs/ARBs)
- Hypothermia
- Sympathetic blockade, neuraxial block
- Ventilation
- Laparoscopy: Hypercarbia, Dysrhythmia, Increased vagal tone from excessive stretching of peritoneum, Compression of IVC, Venous Air Embolus
DDx of Hypertension
- Pre-existing HTN and end-organ dysfunction of brain, heart, kidney
- “White coat” HTN
- Pulmonary: Hypoxia, hypercarbia, pulmonary edema, OSA
- Renal: renovascular disease, renal parenchymal disease, renin-secreting tumor, polycystic kidney disease
- Neurologic: elevated ICP, spinal cord injury, Guillan-Barre syndrome, dysautonomia
- Cardiac: ischemia, stiff vessels, aortic coarctation, fluid overload
- Endocrine: Cushing’s syndrome, pheochromocytoma, thyrotoxicosis, hyperaldosteronism, hyperparathyroid
- Vascular: coarctation of aorta, vasculitis, collagen vascular disease
- Drugs: vasopressors, cocaine, MAOI +/- tyramine, TCAs, naloxone, glucocorticoids, contraceptive, withdrawal of anti-HTN or drugs
- Pain, anxiety, inadequate anesthesia,
- Bladder distension
- MH
- Hypothermia
- Electrolyte abnormalities: hypercalcemia, hypoglycemia
- Autonomic instability
PEA: H’s and T’s
- Hypovolemia
- Hypoxia
- H+ (Acidosis)
- Hyper- and Hypokalemia
- Hypoglycemia
- Hypothermia
- Toxins/Tablets
- Tamponade (cardiac)
- Tension Pneumothorax
- Thrombosis
- Trauma
Causes of Atrial Fibrillation/Flutter
- Severe Heart Disease
- Coronary Artery Disease
- Mitral Valve Disease
- Pulmonary Embolism
- Hyperthyroidism
- Cardiac Trauma
- Cancers of the Heart
- Myocarditis
Tx for Acute Atrial Fibrillation
- IV Diltiazem or Esmolol
- Start synchronized cardio version in patient with pronounced hemodynamic instability. Start with 100-200 J, then 300 J, then 360 J
- If A-Fib has been present for over 48 hrs, there is an increased risk of thromboembolism
a. Consider TEE to rule out atrial thrombus
b. Adequate anticoagulation for 3-4 weeks should be considered prior to cardio version if thrombus is present
Causes of Sick Sinus Syndrome
- A combination of symptoms caused by SA node dysfunction and manifested by marked bradycardia, sinoatrial block, or sinus arrest
- Can have associated episodes of supraventricular tachycardia; often called brady-tachy syndrome
S/S of Sick Sinus Syndrome
- Dizziness
- Confusion
- Fatigue
- Syncope
- CHF
Tx of Sick Sinus Syndrome
- Atrial or dual chamber pacemaker
2. Patents are at high risk of developing PE and should be started on anti-coagulation
Intra-operative treatment of Acute Coronary Syndrome
- Rapid assessment of which determinants of myocardial O2 balance have been compromised (usually tachycardia)
- Improve patient’s hemodynamics
- Evaluate IV access
- 100% oxygen
- Meds (Nitroglycerin, Morphine, Beta blocker)
- 12-lead EKG
- Labs (Cardiac markers, CBC, Electrolytes, Coagulation panel)
- CXR
- Inform surgeon (cancel case or finish ASAP)
- Consult cardiology
- If unstable, consider TEE
- Alert the cath lab
What is Virchow’s Triad?
- Venous Stasis
- Vessel wall damage
- Hypercoagulability
Diagnosis of PE
- Pulmonary Angiography (gold standard)
a. If renal failure, V/Q Scan (avoids IV contrast) - Labs: ABG, D-Dimer (non-specific)
Diagnosis of Venous Air Embolus (VAE)
- Signs of Cardiac Ischemia
- Signs of increased pulmonary artery pressure
- Hypotension
- Acute decrease in ETCO2
a. Increase in alveolar dead space and decrease of cardiac output - Increased ETN2 (end-tidal nitrogen)
- Precordial Doppler: “mill-wheel” murmur
- Air with aspiration of a RA multi-orifice catheter
- TEE: right ventricular dilation or hypokinesis
Tx of VAE
- Lower site of air entry below level of heart
- Notify surgeon
- Secure airway and ensure adequate oxygenation and ventilation
- Stop N2O and place patient on 100% FiO2
- Flood surgical field with saline
- Compression of proximal vein: IJ in sitting cases
- Aspiration of right atrial catheter (if present)
- IV saline bolus
- Support circulation w/ vasopressor
- Position patient in left lateral decubitus
- PEEP does NOT decrease incidence of VAE
Diagnosis of Fat Embolism Syndrome
- Hypoxemia
- Neurological abnormalities: altered mental status
- Petechiae: occurs 12-72 hours after initial trauma or instrumentation
Pathogenesis of Amniotic Fluid Embolism Syndrome (AFES)
Immune response to amniotic fluid contents or immune reaction triggered by leukotrienes or arachidonic acid within the fluid
**NOT a mechanical outflow obstruction
Risk Factors for Amniotic Fluid Embolism Syndrome
- Tumultuous Labor
- Trauma
- Multiparty
- Advanced Maternal age
- C-section
- Increased gestational age