Cardiac stuff Flashcards

1
Q

What is the protocol for PEA?

A

Epi 1mg IV q3-5 mins

if PEA rate slow - atropine 1mg q3-5 mins max 0,04mg/kg

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2
Q

Common causes VT/VF/PEA? (Hs + Ts)

A
Hypovolemia
Hypothermia
H+ (acidosis)
Hyper/hypokalemia
Hypothermia
Tamponade
Tension pneumo
Thrombosis (coronary/PE)
Toxins (drug OD)
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3
Q

What is the protocol fora systole?

A

Epi 1mg IV q3-5mins
Atropine 1mg IV q3-5 mins (max 3g)
Transcutaneous pacing

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4
Q

Protocol for bradycardia

A
Atropine 0.5-1mg
transcutaneous pacing
dopamine 5-20mcg/kg/min
Epi 2-10mcg/min
Isoprotenerol 2-20mcg/min
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5
Q

What tachyarrhythmias would you use SYNC cardioversion?

A

UNSTABLE

  • A fib
  • A flutter
  • VT
  • PSVT

palpable pulse and unstable = synchronize!!

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6
Q

Three treatment considerations for A Fib

A

Rate control
Rhythm controol
Anticoagulate

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7
Q

Avoid these classes of meds in WPW (4)

A

BBs
CCBs
Adenosine
digoxin

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8
Q

What is the time cutoff for converting a fib before you should consider anticoagulation first?

A

48hrs

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9
Q

If A fib ongoing >48hrs, how long should pt be anticoagulated for?

A

3 weeks

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10
Q

Methods of rhythm control in a fib

A

Sync cardioversion
Amiodorone
Procainamide

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11
Q

This investigation can be done in a patient with A fib who has not been anti coagulated but hoping to cardiovert safely

A

TEE to rule out atrial clot

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12
Q

Why must AV node blocking meds be avoided in WPW?

A

Follows aberrant pathway and worsens dysrhythmia. Delta waves on EKG

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13
Q

Initial medical management for stable SVT?

A

Vagal stimulation

Adenosine

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14
Q

Best a fib management in patients with HF?

A

Digoxin or diltiazem or amiodorone

Avoid BB

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15
Q

Preliminary approach to all ACS?

A
Morphine (only if pain)
Oxygen (only if decreased  sats)
Nitro
ASA
BB
ACEi
Statin
Heparin
Clopidogrel
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16
Q

Management for ST elevation or new LBBB?

A
Suspect injury
IV: nitro, BB, heparin
ACEi after 8hr or when stable
<12hrs from onset - Cath or fibrinolysis
>12 hrs from onset - high risk to Cath lab or CABG; low risk = CCU
17
Q

Management for ST depression or T wave inversion

A

Suspect ischemia
Heparin, ASA, IV nitro, BBs
Stable = CCU
High risk = PCI or CABG

18
Q

What is malignant hyperthermia?

A

Defect in Ca2 metabolism = increased intracellular Ca2 = hyper catabolism and decreased ATP

19
Q

What are triggers for malignant hyperthermia

A

Inhalational agents (“ane”s)
Depolarizing muscle agents - SCh, decamethonium
Amide based local anesthetics

20
Q

Signs and symptoms of malignant hyperthermia?

A
High fever/diaphoretic
Tachydardia/dysrhythmia
HTN
Rigidity
Trismus
Tachypnea/cyanosis with resp acidosis and hypercapnia
HyperK
HyperCa
Metabolic  acidosis
Increased myoglobin
Increased CK
21
Q

Treatment for malignant hyperthermia?

A

Stop anesthetic and SCh
Hyperventilate with 100% O2
Dantrolene - 2-10mg/kg (blocks release of Ca); give in 1mg/kg increments
Active coolinng
Correct electrolyte and acid base abnormalities

22
Q

How to prevent malignant hyperthermia

A
Avoid triggers
Ppx dantrolene NOT indicated
Regional anesthesia if possible
Thiopental and propofol SAFE to use
Adrenaline NOT safe
23
Q

Complications of malignant hyperthermia

A
Death
DIC
Coma
Muscle necrosis/weakness
Myoglobin renal failure
Lyte abnomalities
24
Q

Protocol for pulseless VT/VF

A

SHOCK 200 –> 300 –> 360
Epi 1g IV q3-5 mins or vasopression 40U IV x1
Amiodorone 300 mg IV
If torsades - 5g MgSO4 IV