Cardiac station Flashcards

1
Q

Cardiac causes of clubbing

A

Infective endocarditis
Congenital cyanotic heart disease
Atrial myxoma

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2
Q

causes of AF

A

Sepsis, mitral valve stenosis, ischaemic heart disease, thyrotoxicosis, hypertension

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3
Q

Treatment of AF haemodynamically unstable

A

urgent DC cardioversion

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4
Q

stable AF treatment

A

rate = if onset >48 hours or uncertain
= b-blockers, rate limiting CCB e.g. diltiazem or digoxin

Rhythm = if reversible cause, <48 hours, heart failure caused by AF
= flecainide, sotalol, amiodarone, AF ablation

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5
Q

CHA2DS2VASC

A

Congestive HF
hypertension
age >65
diabetes
stroke
vascular disease (MI, PAD, atherosclerosis)
age >75
sex

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6
Q

CHF investigations

A

Bloods, ECG, CXR
Measure NT-proBNP
<400 = HF unlikely
400-2000 = referral for echo + specialist review 6w
>2000 = referral + review 2w

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7
Q

CHF treatment

A

1st line = B-Blocker bisoprolol + ACEi ramipril or ARB losartan, furosemide for symptomatic relief from volume overload

2nd line – still symptomatic = mineralcorticoid antagonist = spironolactone

3rd line = specialist = ivabradine, entresto

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8
Q

CHF signs/symptoms

A

Sob, reduced ET, orthopnoea, peripheral oedema, raised JVP, crackles lung bases

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9
Q

RVF signs

A

raised JVP, hepatomegaly, pedal oedema, sacral oedema, ascites

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10
Q

LVF signs

A

pulmonary oedema, tachypnoea, tachycardia

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11
Q

CHF presentation

A

“Today I examined this gentleman’s cardiovascular system. He had tar staining on his fingers and appeared short of breath. His respiratory rate was 16, and heart rate was 120. He had an irregularly irregular pulse and his JVP was raised at 5cm above the sternal angle. The apex beat was displaced in the 6th intercostal space, anterior axillary line, Heart sounds were normal and there were no added sounds. There were bi-basal fine end inspiratory crackles and peripheral oedema present to the mid thigh” These signs are consistent with congestive cardiac failure secondary to ischaemic heart disease. I would like to take a full history to assess symptoms, and order a CXR”

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12
Q

acute and chronic causes of RVF

A

Acute = MI, PE, infective endocarditis,
chronic = left ventricular failure, cor pulmonale

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13
Q

Acute and chronic causes of LVF

A

Acute = MI, Infective endocarditis.
Chronic = ischaemic cardiomyopathy, hypertensive cardiomyopathy, valvular disease

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14
Q

STEMI treatment

A

1st = primary PCI (<12 hours from onset + <120 mins for PCI)
2nd = fibrinolysis (<12 hours, >120 mins for PCI) = alteplase

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15
Q

Initial management of ACS

A

Analgesia
Antiemetics
Aspirin 300mg
Oxygen if <94%
GTN infusion if pain, HTN or oedema

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16
Q

NSTEMI

A

Positive troponin no ECG changes

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17
Q

NSTEMI treatment

A

aspirin 300mg + fondaparinux
PCI if GRACE score >3% (within 72 hours)

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18
Q

Unstable angina treatment

A

aspirin 300mg + fondaparinux

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19
Q

reversible MI causes

A

Hypoxia
hypovolaemia
hypothermia
hypokaelamia or hyperkalamia
hypoglycaemia
Tamponade
tension pneumo
thrombosis
toxins

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20
Q

Broad complex arrythmias

A

VT
Torsades de Pointes = iV magnesium sulphate

21
Q

Narrow complex arrythmias

A

SVT, sinus tachy, AF

22
Q

Aortic stenosis signs

A

Slow rising pulse, narrow pulse pressure, Ejection Systolic Murmur, loudest on expiration, radiates to carotids DDx = aortic stenosis, HOCM, mitral regurg Causes = age related = calcification, RHD

23
Q

AS symptoms (ASH)

A

angina, syncope, heart failure

24
Q

AS investigations

A

ECG, CXR, echo + doppler, coronary angio

25
Q

AS management

A

general = MDT, RF modification, QRISK, statin, antiplatelet, HTN, angina
Surgical = open replacement (ball in cage = 3 sounds) (bileaflet = 1 sound)

26
Q

AS surgery and indications for surgery

A

TAVI or balloon valvuloplasty
Indications for surgery = symptomatic, CCF, pressure gradient <40%

27
Q

MR signs

A

Displaced apex, S1 quiet, pansystolic murmur, radiates to axilla, valvotomy scar, atrial fibrillation also present
severe if displaced/thrusting apex, LVF

28
Q

MR other DDx

A

aortic stenosis, VSD

29
Q

MR management

A

conservative= MDT, RF mod
Medical = AF = rate/rhythm Reduce afterload = ACEi, B-blocker, diuretics
surgery= repair or replace valve

30
Q

MR causes

A

RHD, IE, IHD, Papillary muscle rupture

31
Q

MR presentation

A

“Today I examined this gentleman’s cardiovascular system. On general inspection he looked alert and comfortable in bed. He had a normal pulse, and his apex beat was non-displaced. Heart sounds I + II were heard and he had a pansystolic murmur, intensity 3/6, loudest at the apex, which radiated to the axilla. There were bibasal fine end inspiratory crackles and peripheral oedema present to the mid thigh. These are consistent with mitral regurgitation with congestive cardiac failure. I would like to take a history to assess for impact on the patient’s life and request a CXR”

32
Q

AR signs

A

Collapsing pulse, head nodding with pulse = De Musset’s sign, Corrigan’s = neck pulses, ejection diastolic murmur at LSE, loudest on expiration

33
Q

AR DDx

A

Mitral stenosis

34
Q

AR management

A

conservative= MDT, RF mod,
Medical = ACEi + B-blocker, + diuretics,
Surgery = valve replacement

35
Q

AR causes

A

IE, rheumatic heart disease, aortic dissection

36
Q

MS signs

A

Mid diastolic murmur, Atrial fibrillation

37
Q

MS differentials

A

Aortic regurg

38
Q

MS causes

A

IE, RHD

39
Q

MS management

A

conservative= MDT, RF mod.
Medical = RHD prophylaxis, AF = rate control + DOAC,
Surgery = balloon valvuloplasty

40
Q

Valve presentation

A

“I examined this Gentleman’s cardiovascular system. He had a midline sternotomy scar and there was warfarin at the bedside. He had irregularly irregular pulse. There was a metallic click with the first heart sound and the second heart sound was normal. There were no additional sounds and no features of cardiac failure. These signs are consistent with atrial fibrillation and a metallic mitral valve replacement that is functioning well.”

41
Q

Post CABG signs

A

tar staining, midline sternotomy scar, scar over left saphenous vein

42
Q

Post CABG presentation

A

“Today I examined this gentlemen’s cardiovascular system. He had tar staining on his fingers, a midline sternotomy scar and a scar over his left saphenous vein. Heart sounds were normal with no additional sounds, and there were no features of cardiac failure. These signs are consistent with previous coronary artery bypass graft using the left great saphenous vein”

43
Q

Vessel used in CABG

A

Great saphenous = scar on calf
Internal thoracic artery / internal mammary artery = more common

44
Q

post-CABG management

A

DAPT = aspirin and clopidogrel for 12 months then aspirin life long
Beta blocker = bisoprolol
ACEi = ramipril or ARB = Losartan

45
Q

IE

A

Staph aureus, strep viridians, mitral valve most common, IVDU = tricuspid, fever, murmur, clubbing, petechiae, splenomegaly, haematuria, roth’s spots, Janeway lesions, osler’s nodes
Treatment = amoxicillin and gentamicin

46
Q

normal cardiac exam presentation

A

Today I performed a cardiovascular examination of X year old Y.
On general inspection the patient was comfortable at rest, did not seem breathless or in pain and did not have monitoring attached.
On closer inspection of the hands and arms, the patient did not exhibit any stigmata of cardiovascular disease such as splinter haemorrhages or Osler’ nodes.
Extremities were warm with normal capillary refill.
Pulse was regular at Z bpm, and there was no radio-radial delay or collapsing pulse.
On closer inspection of face and chest, no anaemia or scars were identified.
JVP was non raised, and carotid pulse had normal volume and character.
No heaves of thrills were identified and apex beat was non displaced at the mid clavicular line.
On auscultation of the heart, hearts sound I and II were audible with no additional sounds.
Lung bases were clear and there was not sacral or peripheral pitting oedema.
In conclusion this was a normal cardiovascular examination. To complete my examination, I would like to take full history and perform a respiratory examination.
I would like to take a full set of observations, including lying and standing BP, take an ECG reading as well as capillary glucose and take bloods, including FBC, U&Es and LFTs for baseline, and cardiac markers if indicated.

47
Q

ECG presentation

A

This is a 12 lead ECG of X year old Y.
It was taken a Z hrs on the date. The quality is adequate, and the calibre is 25mm/s.
The rate is W bpm and regular and there is no/R/L axis deviation.
Every p wave is followed by a QRS complex; thus, this is sinus rhythm.
PR interval is normal, 0.12-0.2 ms, not slurred.
The Q wave is normal (non-pathological).
The QRS complex is narrow (0.8-0.12 ms) and not prolonged. The R waves are equal in size, and there is progression in the chest leads.
The ST interval is normal/elevated/depressed/saddle elevation.
The QT interval is normal/prolonged, the T waves is normal/tall and tented/inverted/biphasic…
In conclusion this is a normal/abnormal ECG…
My main differential is…

48
Q

What are advantages and disadvantages of tissue heart valves?

A
  • Advantages: No need for warfarin anticoagulation, thus safer in women of child-bearing
    age and in the elderly.
  • Disadvantages: Reduced lifespan of prosthesis compared to mechanical valves, due to
    degeneration and calcification.