Cardiac notes Flashcards
Inotropic
increase or decrease contractility
digoxin: positive inotropic (stronger)
most meds : negative inotropic (depress contractility
watch for CHF because not pumping blood out
Dromotropic
conduction system (impulses)
positive : increased conduction
most meds : interfere with conduction
treat tachy arrythmias
drugs have potential for blocks
chronotropic
rate @ SA node
stimulate SA node
women
have weird angina
diagnostic evaluation procedure
ECG
troponin
cardiac enzymes
isoenzymes
myoglobulin
WBC
Sed rate
CK
creatinine kinase
goes up fast then decreases
best for determining early MI
Isoenzymes
CKMB (MB means heart)
WBC
increases because of inflammatory response to attack on heart
sed rate
stays increased for awhile
ECG changes
Q - does not go back to normal
ST elecated
T wave inversion
emergency management of MI
O2
2 IV’s
ECG
nitro/asprin/morphine
bloodwork/x-rays (enlarged heart)
assess antiplatelet/anticoagulation?thrombolytics
beta blockers/ antidysrhythmics
thrombolytics
TPA
4-6 hours after start of chest pain (asap pre-damage)
TPA goes to where its needed
side effects: hemorrhage/anemia
hypotension/fever
bronchospasm/anaphyaxis
periorbital swelling/itching/uticaria/headache
dysrhythmias
can cause another clot when a large clot is broken up
Heart failure
right side -fluid backup
left side - pulmonary
systolic ventricular dysfunction
not enough blood ejection
foward failure
decrease cardiac output -fluid backs up
diastolic ventricular dysfunction
left ventricule cant relax enough to accumulate blood to pump out
right vs. left side heart failure
arteries Left
veins right
rt. failure can be caused by left failure
rt ventricular MI
high output failure
cardiac ouput can be ok
increase metabolic demands on heart
hyperthyroid
septecemia
what diagnoses Heart Failure
BNP
from fluid in ventricles
chest xray
echocardiogram
diltiazem (cardizem)
calcium channel blocker