Cardiac Arrythmias Flashcards

1
Q

Tachycardia

A

Fast heart rate defined as more than 100 beats per minute in an adult.

Causes:
1. Increased body temperature

  1. Stimulation of the heart by the sympathetic nerves
  2. Toxic Conditions of the heart.

Fever causes tachycardia because increased temperature increases the rate of metabolism of the sinus node,which inceases its excitability and rate of rhythm.

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2
Q

Bradycardia

A

Slow heart rate defined as less that 60 beats per minute.

Bradycardia can occur from two reasons:

  1. In Atletes, The atlhetes heart is often large and considerably stonger than that of a normal person allowing the atlethes heart to pump a large stroke volume output per beat even during periods of rest.
  2. Vagal stimulation causes Bradycardia-Any circulatory reflex stimulates the vagus nerve cause release of Ach at vagal endings in the heart thus giving a parasympathetic effect-Carotid Sinus Syndrome-Baroreceptors-carotid sinus causing a barorecptor effect on the heart.
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3
Q

Sinus Arrhythmia

A

Can result from many circularory conditions that alter the of strenghts of the sympathetic and parasympathetic nerve signals to the heart sinus node.

The respiratory type of sinus arryhtmia results from spillover of signals from the medullary respiratory centre into the adjacent vasomotoor centre during inspiration and expiration.

The spillover siganls cause an alternae increase in and decrease in the number of impulses transmitted thoguh the sympathetic and vagus nerve to the the heart

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4
Q

Sinoatrial Block

A

In rare instances impulses from the sinus node is blocked before entering the atrial muscle.

Sudden ceasation of P-waves with resultant standstil of the atria.

The ventricles pick up a new rhythm with the impulse usually originating spontaneously in the AV node so that the ventricular QRS-T complex is slowed but not otherwise altered.

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5
Q

Atrioventricular Block

A

The only way in which impulses can pass from the atria into the ventricles is through the AV bundle/Bundle of His

Conditions which result in this bundle or block the impulse entirely are:

I.I.C.E
I-ischamia of the AV node or bundle fibres-Coronary insufficiency

I-inflamation of the AV node/AV bundle-depresses conductance and can be as a result of myocarditis that are caused for example: Diptheria and Rheumatic fever

C-ompression of the AV node-by scar tissue/calcfified portions of the heart

E-extreme stimulation of the heart by the vagus nerves-Carotid Sinus syndrome

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6
Q

The different types of Atrioventricular Heart Block

A

There are three types of blocks.
1st Degree AV Block
2nd Degree AV Block: Wenckback and Mobitz
3rd Degree AV Block

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7
Q
  1. First Degree AV Block/Prolonged P-R/P-Q Interval
A

The usual lapse of time between the beginning of the p wave and the beginning of the QRS complex is about 0.16 when the heart is normal

PR interval increases in length with a fast heartbeat and decreases with a slow heartbeat.

When PR interval increases to greater than 0.20 seconds the pt is said to have first-degree incomplete heart block

Defined as a delay of conduction from the atria to the ventricles but not an actual blockage of conductance.

Acute Rhuematic Heart disease can be measured using the P-R interval

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8
Q

Second Degree Block

A

There are two types of Second Degree Blocks:

  1. Wenckeback Periodicity
  2. Morbitz

There are some dropped beats,There will be an atrial p-wave but no QRS-T wave.

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9
Q

Wenckeback Periodicity

A

Characterized by progressive prolongation of the PR interval until a ventricular beat is dropped and is then followed by resetting of the PR and repeating of the abnormal cycle

Almost always caused by abnormality of the AV node.

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10
Q

Morbitz

A

There is usually a fixed number of nonconducted p waves for every qrs-complex

It is represented in a ratio.

Caused by an abnormality of the bundle of his-purkinje system and may require implantation of a pacemaker to prevent progression to complete heart block and cardiac arrest.

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11
Q

Complete A-V Block/Third Degree Block

A

When the condition that causes poor conduction in the AV node/ bundle becomes severe,complete block of the impulse from the atria into the ventricles occurs.

Ventricles spontaneously establish their own signal usually originating in the AV node or distal to the block.

Therefore the p-waves become dissociated from the QRS-T complexes

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12
Q

Stokes-Adam Syndrome-Ventricular Escape

A

The total block comes andd goes,impulses are conducted from the atria into the ventricles for a period of time and then are suddenly no longer conducted.

The duration of the block may last for minutes,hours ,days before conduction returns.

This condition occurs in hearts with borderline ischemia of the conductive system

The peridic fainting spells are know as the stokes-Adams Syndrome

Artificial Pacemakes may be needed.

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13
Q

Incomplete Intraventricular Block-Electrical Alternans

A

Most of the factors which cause AV-Block can also block impulse conduction in the peripheral ventricular Purkinje system

Electrical Alternans results from partial intraventricular block every other heartbeat

Conditions which depress the heart such as Ischemia,Myocarditis or digitalis toxicity can cause incomplete intraventricula block

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14
Q

Premature Contractions

A

A premature contraction is a contraction of the heart before the time that the normal contraction would have been expected.

This condition is know as Extrasystole,Premature beat or Ectopic Beat.

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15
Q

What are the causes of Premature Contractions

A

Possible causes of Ectopic Foci are:

  1. Local areas of ischemia
  2. Small calcified plaques at different points in the heart.
  3. Toxic irritation of the AV node,Purkinje system or myocardium caused by infection ,drugs, nicotine or caffeine.

Mechanical initiation of premature contractions are also frequent during cardiac catheterization,large numbers of premature contractions often occur when the catheter enters the right ventricle and presses against the endocardium

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16
Q

Premature Atrial Contractions

A

P waveoccured too soon futhermore the p-r interval is shortened

The interval between the premature contraction and the next succeeding contraction is slightly prolonged-Compensatory Pause.

One of the reasons for this pause is that the premature contraction originated in the atrium some distances from the sinus node and the impulse had to travel through a considerable amount of atrial muscle before it discharged the sinus node.

PAC occur frequently in healthy people.Indeed they are often occur in athletes whose hearts are healthy .

Mild toxic conditions resulting fromsuch factors as smoking, lack of sleep, ingestion of too much coffe, alcoholism and use of various drugs.

17
Q

Pulse Deficit

A

When the heart contracts prematurely, the ventricles will not have filled with blood normally and the stroke volume output during that contraction is depressed or almost absent

The pulse wave passing through the peripheral arteries after a premature contraction may be so weak that it cant be palpated in the radila artery.

Thus, a deficit in the number of radial pulses occurs when compared with the actual number of contractions of the heart

18
Q

AV-Nodal/AV Bundle Premature Contractions

A

P-wave is superimposed onto the QRS-T complex because the cardiac impulse traveled backward into the atria at the same time that it traveled forward into the ventricles.

AV-Nodal premature contractions have the same significance and cause as atrial premature contractions

19
Q

Premature Ventricular Contractions

A

PVCs cause specific effects in theECG’s

  1. The QRS complex is usually considerably prolonged-impulse is conducted through slowly conducting muscle of the ventricles rather than through the Purkinjee system
  2. The QRS complex has a high voltage
  3. T-wave has an electrical potential polarity exactly opposite to that of the QRS complex-because the slow conduction of the impulse.

Some PVCs are relatively benign in their effects on the overall pumping by the heart,they can result from factos such as-cigarettes,excessive intake of caffeine