Cardiac Flashcards

1
Q

What can atrial dysrhythmia cause?

A

Decrease CO, no atrial kick (lost), hypotension, and thrombi (can cause stroke).

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2
Q

What happens to the atria and ventricles during diastole?

A

atria contract/ventricles relax and fill

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3
Q

What happens to the atria and ventricles during systole

A

atrial relaxation/filling and ventricular contraction

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4
Q

S1 sound caused by the closure of what valves?

A

atrioventricular valve closure; “Lub” sound

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5
Q

S2 sound caused by closure of what valves?

A

aortic/pulmonic valves close; “Dub” sound

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6
Q

When can S3 be considered normal?

A

In children

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7
Q

When is S3 produced?

A

when heart is overfilled or poorly compliant (Ex: CHF/fluid overload)

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8
Q

When is S4 heard?

A

Just before the S1 is heard

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9
Q

What causes murmurs?

A

turbulent blood flow- through heart valves or septal defect

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10
Q

What are the Cardiac Enzymes?

A

CKMB, Troponin, Myoglobin

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11
Q

What can hyperkalemia cause

A

Asystole and ventricular dysrhythmias

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12
Q

Can you list 3 exams used to monitor heart function?

A

Cardiac stress test, echocardiogram, transesophageal echo, myocardial perfusion scan, electrophysiological testing (invasive), holter, Muga scan, ct scan, PET scan, MRA

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13
Q

What is CVP?

A

Central venous pressure; pressure within the superior vena cava/right atrium and reflects end diastolic pressure; “filling pressure” of right ventricle

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14
Q

What are some nursing actions we would expect to do after a cardiac cath insertion?

A

Evaluate distal peripheral pulses, color, warmth & sensation
Evaluate insertion site for hematoma and bleeding
Bedrest 6-8 hours post procedure, and keep affected limb straight 6-8 hours and do not elevate head more than 20 degrees if femoral artery used.
Encourage PO fluids to promote renal excretion of dye
Monitor urine output (should be at least 30 ml per hour)
Repeat H&H, BUN, Creatinine (dye can cause renal failure)
Possible sand bag to insertion site

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15
Q

What type of diet should someone with CAD follow?

A

Low cholesterol, low salt, high fiber

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16
Q

What is the area of dead muscle known as and how would this reflect on the ECG?

A

“Zone of Infarction” and seen by new Q waves reflecting lack of depolarization in the dead cells

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17
Q

When does a STEMI occur?

A

STEMI usually occurs because of plaque rupture leading to complete occlusion of the artery

18
Q

How should we first assess an MI is occurring?

A

Thorough history and clinical presentation

19
Q

What are some signs and symptoms of an MI occurring?

A

Crushing substernal chest pain, may or may not radiate to left arm and/or jaw that is unrelieved with rest and nitrates
Diaphoresis and cool, clammy, pale skin, cyanosis
Dyspnea with and without crackles
Nausea and vomiting
Syncope and hypotension
Palpitations
Dysrhythmias such as PVCs, tachycardia, or bradycardia
Weakness
S3 sound indicating left ventricle involvement
Anxiety and fear, restlessness, feelings of impending doom

20
Q

How is Morphine usually given for an acute MI?

A

if pain not relieved with nitro: usually morphine sulfate 2 to 4 mg may be given every 5 to 15 minutes via I.V. push (NO IM’s)

21
Q

What is a normal Left Ventricular EF?

A

ranges from 50-70%

22
Q

What does EF 35 - 40% mean?

A

May confirm diagnosis of systolic heart failure

23
Q

What does an EF < 35% mean?

A

risk of life-threatening irregular heartbeats that can cause sudden cardiac arrest and death. An implantable cardioverter defibrillator (ICD) may be recommended for these patients

24
Q

List some ways nurses can help those with Heart failure?

A

High fowlers position (sometimes called orthopneic position)with legs in dependent position if severely short of breath
High concentration of oxygen (may need intubation if severe respiratory distress or Bi-pap)
Suction if needed (Lt. side failure frothy sputum)
Monitor vital signs
Cardiac monitoring for dysrhythmia
Assess for edema (peripheral, JVD, hepatomegaly, ascites, sacral)
I&O (insert foley, accurate I&O to determine effectiveness of treatment), avoid unnecessary IV fluids
Low sodium diet, low cholesterol diet; may be on fluid restriction
Daily weight and abdominal girth
Monitor peripheral pulses and capillary refill
Monitor ABGs and serum potassium levels (Lasix and Digitalis cause excretion of K+)

25
Q

What are some later signs of Endocarditis?

A

Loud regurgitant heart murmur or change in character of an existing murmur in the presence of fever is a classic sign
Petechiae of the skin (upper ant. trunk), buccal, pharyngeal, and conjunctiva. Splinter hemorrhages of nail beds. Occasionally Osler’s nodes(tender raised lesions on fingers or toes), Roths spots (hemorrhagic areas with white centers on retina of eye), and Janeway lesions (purple macules on palms and soles)
Emboli from vegetative lesions causing splenic, renal, cerebral, or pulmonary infarction or peripheral occlusion.
Splenic infarction: pain in upper lt. quadrant radiating to the left shoulder and abdominal rigidity.
Renal infarction: hematuria, pyuria, flank pain , and decreased output.
Pulmonary infarction is most common in right sided endocarditis and often occurs among IV drug users and after cardiac surgery. Cough, pleuritic pain, pleural friction rub, dyspnea, and hemoptysis

26
Q

Difference between false and true aortic aneurysms?

A

False versus true (false aneurysm is usually caused by trauma; the vessel wall of the vessel is ruptured and blood escapes into the surrounding areas causing a clot to form) Treatment based on size; with those >5 CM usually requiring surgical repair

27
Q

After sustaining an MI, the patient is most at risk for developing what in the first 24 hours?

A

V Fib

28
Q

How will calcium channel blockers (CCB) influence contractility?

A

They will decrease contractility and must be used with caution in those with heart failure)
Decrease in contractility (inotropic effect) decreases cardiac workload and O2 demands

29
Q

What are some manifestations of Cardiac tamponade?

A
Paradoxical pulse
Narrowed pulse pressure, hypotension
Tachycardia
Weak peripheral pulses
Distant, muffled heart sounds
Jugular venous distention
High central venous pressure
Decreased level of consciousness
Low urine output
Cool, mottled skin
30
Q

What are some symptoms of right sided heart failure?

A

Peripheral edema, jugular vein distension, and hepatojugular reflex

31
Q

What assessment finding can given us more information about left sided heart function?

A

Status of breath sounds

32
Q

What is a risk assessment for CAD?

A

A risk assessment for coronary artery disease (CAD) is
determined by comparing the total cholesterol to high density lipoprotein (HDL) and a ratio can be calculated by dividing the total cholesterol level by the HDL level. The ratio provides more information than either value alone
and an increased ratio indicates an increased risk

33
Q

What are the goals of management of an AMI?

A

To dissolve the lesion that is occluding the coronary artery and to increase blood flow to the myocardium. Options: emergent percutaneous intervention, such as angioplasty, emergent coronary artery bypass surgery, or thrombolytic therapy if the patient has been symptomatic for less than 6 hours.

34
Q

Let’s discuss the differences between pain in Angina vs in an MI

A

Angina: substernal chest discomfort maybe only radiating to Left arm, precipitated by exertion or stress, relieved by NTG or rest, lasts less than 15 min. MI: substernal radiating to Left arm, pain/discomfort in jaw, back, shoulder, or abdomen; occurs without cause; relieved only y opioids; lasts 30 mins or more; frequent associated symptoms like nausea/vomiting

35
Q

When would we usually see an asymptomatic MI occur?

A

In elderly patients, in women, and in diabetic patients

36
Q

When would a CABG be needed?

A

For significant (greater than 50%) occlusion is seen the left main coronary

37
Q

Why would we need to know that a patient is taking Sildenafil citrate prior to giving Nitroglycerin?

A

Meds for erectile dysfunction potentiate the hypotensive effects of nitrates

38
Q

What are some contraindications to thrombolytic therapy? (absolute)

A

If patient has had any prior intracranial hemorrhage
Any known structural cerebral vascular lesions
Any known malignant intracranial neoplasm
An ischemic stroke within 3 months EXCEPT acute ischemic stroke within 3 hours
A suspected aortic dissection
Active bleeding
Significant closed-head or facial trauma within 3 months

39
Q

What kind of activity should we recommend for those with Coronary artery disease?

A

Begin walking the same distance at home as in the hospital (usually 400 feet) 3 times each day
Carry nitroglycerin with you
Check your pulse before, during, and after the exercise
Stop the activity for a pulse increase of more than 20 beats/min, shortness of breath, angina, or dizziness
Make gradual increases in walking distance
After an exercise tolerance test and your HCP approval, walk at least 3 times each week, increasing the distance every other week, until the total distance is 1 mile
Avoid straining

40
Q

What is a major manifestation of MI in people older than 80yrs?

A

Disorientation or acute confusion due to poor cardiac output and inadequate coronary perfusion