cardiac Flashcards
High arched palate
Marfans
Orange tonsils
Tangiers disease
Loeys Dietz
arterial tortuosity , hypertelorism wide or split uvula aneurysms at the aortic root cleft palate club foot
bifid uvula
Loeys Dietz
blue sclera
osteogenesis imperfecta
marfans
why should you do a fundoscopic exam in infective endocarditis
friable vegetation on valve- embolism- retinal occlusion
Hollenhurst plaque
BRAO, due to lipid plaque emboli, occurs thru internal carotid artery. seen in atherosclerosis
Wegners granulomatosis
Saddle nose, acute necrotizing granuloma of the upper and lower respiratory tract.
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Small-vessel vasculitis Necrotizing granulomatous vasculitis (NGV) Microscopic polyangiitis (MPA) Churg-Strauss syndrome (CSS) Medium-vessel vasculitis Polyarteritis nodosa Kawasaki disease Large-vessel vasculitis Takayasu arteritis Giant cell arteritis
Immune-complex mediated vasculitis
Goodpasture's syndrome Henoch-Schönlein purpura Behçet's disease Essential cryoglobulinemia IgA nephropathy
Secondary vasculitis
SLE Rheumatoid arthritis Antiphospholipid antibody syndrome Infection Drug-inducedInflammatory bowel disease Hypocomplementemic urticarial vasculitis Paraneoplastic
Superior vena cava syndrome
most commonly bronchogenic carcinoma
Shortness of breath is the most common symptom, followed by face or arm swelling
Difficulty breathing
Headache
Facial swelling
Venous distention in the neck and distended veins in the upper chest and arms
Upper limb edema
Light headedness
Cough
Edema of the neck, called the collar of Stokes
Pemberton’s sign
Pemberton’s sign
Presence of latent pressure in the thoracic inlet.
The maneuver is achieved by having the patient elevate both arms until they touch the sides of the face.
A positive Pemberton’s sign is marked by the presence of facial congestion and cyanosis, as well as respiratory distress after approximately one minute
Straight Back Syndrome
Thoracic deformity characterized by loss of the normal upper thoracic spinal kyphosis
Reduced antero-posterior diameter of the chest causing a compression or “pancaking” of the heart and great vessels so as to appear enlarged.
This is accompanied by a leftward displacement of the heart, resulting in cardiac murmurs, chest pain and tracheal compression
Mitral valve prolapse (MVP) has been reported in 64% of patients
Eruptive xanthomas
Lipena retinalis
sever triglycerigides
palmar crease xanthoma
type 3 hyperlipidemia
Carneys syndrome
lupus pernio
multiple atrial myxomas
erythema nodosum
Holt oram
Absence of radial bone
1st degree heartblock
ASD
fixed thumb
osler Weber rendu
Hemorrhagic hereditary telengectasia
malar telengectasia
mitral stenosis, scleroderma
Jaundice
RHF, congestive hepatomegaly (cardiac cirrhosis)
janeway lesions
non tender, raised slightly hemorrhages on the pals and soles
infective endocarditis
oslers nodes
painful nodes on hands/ feet. See in in IE
Homan sign
posteriror calf pain on active dorsiflexion of the foot against resistance
Mitral regurgitation
holosystolic, high-pitched “blowing murmur.” Loudest at apex and radiates toward axilla. Enhanced by maneuvers that increase total peripheral resistance or expiration.
Tricuspid regurgitation
Tricuspid regurgitation
Holosystolic, high-pitched “blowing murmur.” Loudest at tricuspid area and radiates to right sternal border, Enhanced by maneuvers that increases total peripheral resistance and with with inspiration.
Aortic stenosis
Crescendo-decrescendo systolic ejection murmur following ejection click. Radiates to carotids/apex. Pulses are weak compared to heart sounds.
Ventricular septal defect
Holosystolic, harsh-sounding murmur worse with inspiration. Loudest at tricuspid area. Newborns