CAP 8- Dental caries - factors affecting imitation and progression of lesions Flashcards

1
Q

what involved in the initation of the carious process?

A

Plaque formation (on a biofilm)

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2
Q

what is a biofilm?

A

Community of micro-organisms attached to a surface

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3
Q

Describe a biofilm.

A

-Community with collective physiology – work together and interact
-Commensal organisms
-Site to site variability
Tooth surface
Within the Biofilm
-Extra-cellular material
>Bacterial
>Oral Cavity

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4
Q

Where can biofilms form?

A
  • On enamel for coronal caries
  • On root for root surface caries
  • On the root canal when pulp has died off and become infected
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5
Q

what is need for the imitation of the carious process?

A

susceptible tooth surface

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6
Q

What happens to the susceptible tooth surface to innate the carious process?

A

-Pellicle formation:
Acellular, proteinaceous film derived from saliva
-Bacterial colonization (0-4 hours) in stagnation areas (S. sanguis, S. oralis, S. mitis, Actinomyses species, Gram negative bacteria, mutans streptocci (2%))
-Microcolony formation (4-24 hours)

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7
Q

Describe the unique process of how occlusal caries starts and progresses.

A

-Biofilm will start to form bacteria in the biofilm will break down any sugar to produce acid and cause demineralisation
-Contines to get deeper until it hits ADJ and then the areas of demineralisation will merger causing a larger area
(Fluroide makes the enamel strongers and more resistant to this process)

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8
Q

Name one common plaque stagnation area.

A

Approximal surface with adjacent tooth

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9
Q

What happens at plaque stagnation areas?

A
  • Gingival recession – root caries

- Adjacent to partial dentures

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10
Q

What forms adjacent to restorations (around poorly polished and finished restorations)?

A

plaque stagnation areas

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11
Q

Describe the microbial succession of the carious process.

A

Strept in first 1-4 deominate

But over 1-14 days this dominance can change to actinomyces

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12
Q

what is acidogenic?

A

transport sugars in diet and convert to acid – lactic acid

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13
Q

What is converted to acid?

A

Extracellular and intracellular polysaccarides – plaque matrix

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14
Q

What is aciduric?

A

thrive at low pH

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15
Q

what is the specific plaque hypothesis?

A
  • Approx 300 species of intra-oral micro-organisms
  • Specific limited number involved in carious process
  • ? immunise against these
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16
Q

What is the non-specific plaque hypothesis?

A
  • Caries results from the over-all activity of the micro-organisms within the biofilm
  • All plaque should be removed
17
Q

What is the ecological plaque hypothesis?

A

-Plaque can accumulate at all tooth sites
-Balance of microflora can change depending on local environment:
>Sugar
>Saliva
>Stagnation areas
-Favours aciduric and acidogenic species
-Remove plaque from susceptible sites and decrease frequency of sugar intake

18
Q

Describe the Stephan curve.

A

pH within the biofilm on the surface pf the tooth will decrease after sucrose rinse and then buffering of saliva will increase and the pH will rise
(cariogenicty under 5.5)

19
Q

what autoimmune conditions produce a lot of caries?

A

Autoimmune conditions such as sjorgens that have a lack of saliva

20
Q

Describe what is seen in a ground section examined in polarised light -imbibition in water.

A
  • When section is soaked in water you will see 2 zones
  • Majority of mineral is lost in body of lesion
  • Surface zone has very low demineralisation
  • Thickenes of surface zone can indicate speed of caries – slow caries has a thicker surface zone
21
Q

What 2 zones are seen in ground sections examined in polarised light - ambition in quinoline?

A
  • Translucent zone

- dark zone

22
Q

Name the 2 zones of carious dentine.

A
  • the outer zone

- the inner zone

23
Q

Describe the outer zone.

A

-The outer zone - demineralised, collagen denatured, infected and not remineralisable:
>Note for occlusal caries, only lesions radiographically visible extend to middle third of dentine and beyond, and are heavily infected.
>For proximal lesions lesions deeper clinically than they appear radiographically

24
Q

Describe the inner zone.

A

-The inner zone - Dentine is demineralised, minimally infected, collagen fibers intact and dentine remineralisable.
>Note in deep caries, bacteria within the lesion obtain substrate from oral cavity, and possibly pulp (tissue glucose).

25
Q

what challenges the biofilm?

A
  • Fissure sealant studies
  • Ultraconservative caries removal
  • Stepwise excavation
  • Hall technique
26
Q

what are fissure sealants?

A
  • Low viscosity resins that flow inton the poits and fissures of posterior teeth
  • Primary prevention and also used above caries lesions that extend into the dentine
27
Q

what effect does acid etch have alone?

A

75% reduction in viable organisms

28
Q

What happens in the first visit of stepwise excavation?

A
  • Remove only superficial layer of infected necrotic dentine.
  • Ensure peripheral caries removal complete.
  • Place Ca OH lining and Glass Ionomer.
  • Leave for 6-12 months for pulp dentine complex reactions
29
Q

What happens in the second visit of stepwise excavation?

A

Re-clean cavity and restore

30
Q

Describe the outcome of a clinical and microbiological study of deep carious lesions during stepwise excavation using long treatment intervals.

A
  • All sites after first excavation were soft, wet, pale and heavily infected
  • sealed for 6-12 months
  • all became harder, dry , dark and reduction in level of infection
31
Q

What is ultraconservative caries removal?

A

Clinical performance of sealed composite restorations placed over caries compared with sealed and unsealed amalgam restorations

32
Q

Describe how ultraconservative caries removal works.

A
  • Remove superficial bit of tissue and apply composite
  • Composite is thoigh to be stronger than a fissure seal
  • No change in lesuons after 10 years no further deterioration
33
Q

How does microbiology affect caries?

A
  • Decrease in microbial load
  • Reduction in microbial diversity
  • Reduction in nutrient amount and complexity – derived from pulp
  • Pulpal nutrient decrease in time with pulp dentine complex reactions