CAD Flashcards

1
Q

Define CAD

A

Narrowing or blockage of coronary arteries

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2
Q

Risk factors for CAD unique to women

A
  • Smaller coronaries, less developed collateral circulation
  • Elevated inflamm state with loss of estrogen
  • Lower baseline HDL
  • Vague symptoms are normal
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3
Q

What is the main component in athero plaques?

A

LDL

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4
Q

How does DM contribute to CAD development?

A

Lowers HDL

Raises TGs and LDL

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5
Q

What is the most important diagnostic tool in CAD?

A

History

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6
Q

Medical management of CAD?

A
  • BP, DM control, lifestyle changes
  • Platelet inhibition
  • NTG
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7
Q

Treatment of STEMI

A
  • PCI (time is muscle!)
  • Oxygen
  • ASA 325 mg chewed
  • NTG sublingual
  • Morphine
  • Heparin
  • B blocker
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8
Q

Difference between STEMI and NSTEMI?

A
  • STEMI = full thickness infarct

- NSTEMI = partial thickness infarct

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9
Q

What is prinzmetal angina?

A
  • Coronary artery spasm
  • Generally in clean coronaries in younger women
  • Occurs in early AM
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10
Q

What causes prinzmetal angina?

A

Cold, stress, cocaine, smoking, vasoconstricting meds

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11
Q

What are complications of MI?

A
  1. Cardiogenic shock
  2. Heart failure
  3. Dressler’s syndrome
  4. Arrhythmias
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12
Q

What is cardiogenic shock?

A
  • Large LV infarct leads to significant decrease in contractility
  • Results in very low BP with inadequate supply of oxygen rich blood to body
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13
Q

What is Dressler’s syndrome?

A
  • Fever, pleuritis, pericarditis

- Caused by autoimmune reaction to damaged heart muscle, can occur weeks or months after MI

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14
Q

Why are ACEI/ARBs used post-MI?

A

Shown to minimize LV dilation post-MI resulting in lower rates of HF and improved survival

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