Burns: Classifications of Burns Flashcards

1
Q

What is classified as a burn?

A

A burn is an injury to tissue caused by contact with dry heat (fire), moist heat (steam or hot liquid), corrosive chemicals, electric current, or radiation

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2
Q

A burn is influenced by: (3)

A
  1. Temperature of burning agent
  2. Duration of contact time
  3. Type of tissue that is injured
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3
Q

Types of Burn: Thermal Exposure or Contact (3)

A

Flame
Scalding
Contact with hot objects

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4
Q

Types of Burns: Chemical Burns

A

contact, concentration, volume and type are significant factors
- Alkalis
- Acids
- Organic

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5
Q

Types of Burns: Smoke and Inhalation Injury

A

Smoke inhalation is a major predictor of mortality in burn patients
Related to severity and type of gases or smoke inhaled

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6
Q

3 types of smoke and inhalation injury

A
  1. carbon monoxide poisoning and asphyxiation
  2. Inhalation injury above the glottis
  3. Inhalation injury below the glottis
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7
Q

Carbon Monoxide poisoning and asphyxiation

A
  • Skin colour is cherry red when a patient has been exposed to carbon monoxide poisoning.
  • Carbon monoxide replaces O2 on the hemoglobin causing hypoxia at the tissue level. Carboxyhemoglobinemia.
  • Death occurs when CO levels exceed 60% but the pt is symptomatic at 10%
  • CO binds to hemoglobin with 200x more affinity to oxygen
  • CO decreases cellular respiration at cytochrome a3 - no ATP is made, no muscle function, stop attempting to breathe and die.
  • Treat with 100% oxygen via non-rebreather
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8
Q

Upper airway injury: Signs

A

Thermally produced from hot air or smoke. Looks like edema, blistering or redness of the oral pharynx or larynx, mechanical obstruction can occur quickly. SIGNS: facial burns, singed nasal hair, hoarse voice, darkened membranes, thick black sputum, clothing burns.

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9
Q

Lower Airway Burns

A

Usually chemically produced d/t duration of exposure to the smoke or toxic fumes. Chemicals that are in the fine particulate matter. Gets into the airways and causes inflammation. Manifestations: acute respiratory distress.
Tissue injury to the lower respiratory tract, clinical manifestations may not happen until 12-24 hours after surgery

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10
Q

What are we most worried about with inhalation burns?

A

Swelling and Edema

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11
Q

What is the treatment for smoke and inhalation injury?

A

High flow 100% oxygen in non-rebreather mask. For at least 80 minutes

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12
Q

Electrical Burns: Types

A

Lightening or contact with high voltage wire produces heat as electricity moves through the body
Extent of damage can be difficult to determine since damage is mostly below the skin (iceberg effect)
Remember they may have fallen
Usually an entry and exit where it grounded out

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13
Q

Electrical Burns: Cause of immediate death

A

Cardiac failure
want to have AED on them to see if there is a shockable rhythm

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14
Q

Electrical Burns: Influenced by (5)

A

Duration of contact
Intensity of current (voltage)
Type of current (direct or alternating)
Pathway of current
Resistance of tissues as it passes through the body

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15
Q

Electrical Burns: effect on muscle and internal organs

A
  • contact with electrical currents can cause muscle contractions strong enough to fracture the long bones and vertebrae
  • current that passes through vital organs will produce more sequelae than current that passes through other tissues
  • due to this and possible falls all patients with electrical burns should be placed on spinal precautions - hardcollar
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16
Q

Electrical burns: Myglobinemia

A

Damage of the muscles beneath level of skin - Myoglobin is released - large proteins. could end up with obstruction of kidneys and AKI secondary to breakdown of proteins caused by burn. seen in high CK blood test.

17
Q

Radiation Burns: most common source
influenced by: (5)

A

Sunburns - skin erythema, edema, pain.
1. distance from radiation
2. strength of radiation source
3. duration of exposure
4. extent of body surface area exposed
5. amount of shielding between person and source

18
Q

Severity of Injury is determined by: (4)

A

Treatment of burns is related to the severity of the injury
1. burn depth
2. extent of burn
3. location
4. patient risk factors

19
Q

Partial-thickness:
First degree burn

A

partial-thickness burns involve injury to epidermis and dermis
- first-degree burn - superficial: epidermis; painful, appear red, with no blistering initially (after 24h, skin may blister and peel)

20
Q

Partial thickness burn: second degree

A

second degree partial thickness burns: epidermis and dermis involved; appear wet or blistered and are extremely painful but can heal on their own if area small and there is no infection

21
Q

Full thickness: third degree

A

damage throughout dermis into subcutaneous tissue; unless area small and no infection, grafting is necessary
nerve endings destroyed. dry, waxy white.

22
Q

Full thickness: fourth degree

A

involves skin, fat, muscle, and sometimes bone
sensation is lost in full thickness. dead skin has leathery appearance (eschar), burned skin is painless but the surrounding skin is very painful. need graft

23
Q

Extent of Burn

A

Burn size expressed as a percentage of total body surface area (TBSA) using the:
- Lund Brower Chart
- “Rule of 9’s”

24
Q

Concern with burns on the face, neck, circumferential chest or back?

A
  • mechanical obstruction related to edema or leathery scar tissue might inhibit respiratory function
25
Q

Concern with burns on the hands, feet, joints, or eyes:

A

makes functioning difficult and inhibits future functioning

26
Q

Concern with burns on the ears, nose, buttocks and perineum?

A

high risk for infection. Ears, nose - poor blood supply and exposure to bone

27
Q

Concern with burns on the extremities?

A

cause circulatory impairment distal to burn and subsequent neurologic impairment. Compartment syndrome - circumferential burning of an extremity

28
Q

The Burn patient with heart disease and/or COPD?

A

Pneumonia
Fluid overload
Older adults heals slowly
Significant demands placed on the body by a burn injury
Mechanism of injury: history of the type of burn and injuries beyond the burn. What was the fine particulate matter that would cause chemicals to be released
Comorbidities

29
Q

What causes edema immediately following direct injury to the tissue?

A
  • immediately following burn injury vasoactive substances are released from injured tissue (these substances initiate changes in capillary integrity and allow plasma to seep into surrounding tissue) - edema
  • direct damage to vessels from heat further compromises vascular integrity and sodium-potassium pump fails causing cellular edema
  • the burn-injured client’s hemodynamic balance, metabolism and immune status is altered
30
Q

If the tissue is not cooled:

A

and continues to heat up cell necrosis occurs