Burn Management: Acute Phase Flashcards
Acute Phase timeline - when does it begin and when does it end?
- The acute phase begins with the mobilization of extracellular fluid and subsequent diuresis
- The acute phase is concluded when the burned area is completely covered by skin grafts, or when the wounds are healed
Pathophysiology of the acute phase (5
- Diuresis from fluid mobilization occurs, and the client is less edematous
- Bowel sounds return
- Healing begins when WBCs surround the burn wound and phagocytosis occurs
- Necrotic tissue begins to slough
- Granulation tissue forms
How do partial and full-thickness burn wounds heal?
- a partial-thickness burn wound heals from the edges
- full-thickness burns must be covered by skin grafts
What is the biggest risk at this phase?
Sepsis - debride early because necrotic tissue is the breeding ground for bacteria
Partial-thickness wounds form:
eschar
- once eschar is removed, re-epithelialization begins
- epithelial buds from the dermal bed eventually close in the wound, which heals spontaneously without surgical intervention usually within 10-21 days
- clean wound with sterile water, later with intrasite gel and then silver dressing
Full-thickness wounds require
debridement because margins of full-thickness eschar take longer to separate
Acute Phase: Lab Values - Hyponatremia
Hyponatremia can develop from excessive GI suction and diarrhea
Water intoxication - dilution hyponatremia. encourage intake of fluids other than water
Acute Phase: Lab Values - Hypernatremia
May develop following successful fluid replacement, improper tube feedings, or inappropriate fluid administration
Acute Phase: Lab Values - Hyperkalemia
Noted if client has renal failure, adrenocortical insufficiency or massive deep muscle injury
Can cause cardiac dysrhythmias and ventricular failure, muscle weakness, ECG changes
Acute Phase: Lab Values - Hypokalemia
Can be caused by lengthy IV therapy without potassium
Vomiting, diarrhea
Prolonged gastrointestinal suction
Complications: Infection
- Localized inflammation, induration, and suppuration
- Partial-thickness burns can become full-thickness wounds in the presence of infection
- Wound infection may progress to transient bacteremia
- Client may develop sepsis - condition becomes critical
Complications: Musculo-skeletal system
- Decreased ROM
- Contractures (as wound heals)
Because of pain, client will prefer to assume a flexed position for comfort, encourage the client to stretch and move the burned body parts as much as possible
Complications: Gastrointestinal system
- Paralytic ileus results from sepsis
- Diarrhea from antibiotics or enteral feedings
- Constipation - decreased mobility, opioid analgesics
- Curling’s ulcer - stress ulcer d/t decreased blood flow to GI during emergent phase. give PPI and feed pt as soon as possible.
Complications: Endocrine System
- increased blood glucose production - stress response
- increased insulin production - not good at reducing blood glucose
- Hyperglycemia - all patients that come in end up on insulin SS to have glucose managed. hyperglycemia is caused by increased caloric intake necessary to meet metabolic requirements
- this stress-induced condition is reversed as metabolic demands are met
Predominant therapeutic interventions in the acute phase are: (6)
- wound care
- excision and grafting
- pain management
- physical and occupational therapy
- nutritional therapy
- psychosocial care