Burns and Smoke Inhalation Flashcards

1
Q

Classification of 1st degree burn

A

superficial, limited to the epidermis, wound is red, painful, and well demarcated

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2
Q

Classification of 2nd degree burn

A

partial thickness, involves the epidermis and part of the dermis. May involve hair & glands. Wounds are painful, blister, & blanch with pressure. Tends to be wet & slippery to touch.

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3
Q

Classification of 3rd degree burn

A

Full thickness. Involves all epithelial and dermal elements. Specific wound is painless (but will often be surrounded by painful tissue so patients may report pain). It is depressed, non-edematous, and leathery. May be white, brown, or black

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4
Q

Classification of 4th degree burn

A

Deep tissue. Extends through all layers of skin and involves underlying tissue. Wound is painless but injury is extensive and often requires amputation

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5
Q

Electrical burns

A

Electrical energy is converted to heat which causes thermal injury and burns. However, unlike conventional thermal burns, the electricity may flow in unpredictable pattern and significant injury may not be evident at site of entry. Therefore, a detailed skin exam, including evaluation of the palms and soles is essential. Cardiac arrhythmias are common in electrical burns if the flow of electricity crosses through the thorax and across the heart. Extent of injury is determined by voltage type, voltage strength, the resistance of tissue, and the duration of contact.

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6
Q

Temperature related inhalation injury

A

Heat tends to affect the upper airway more than the lower airways.
Patients will develop edema, erythema, and ulcerations of lips, tongue, posterior oropharynx, and upper airway. Onset may be delayed for up to 24hrs & resolve in 4-5 days. May require early intubation before edema develops and tracheostomy if edema continues

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7
Q

Smoke related inhalation injury

A

Smoke tends to affect the lower airways more than the upper airways.
Reduced mucociliary function. Patients may develop pneumonia in part due to impaired clearance.

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8
Q

Gas-related inhalation injury

A
  • Oxygen is consumed during combustion. Fires create hypoxic environments and patients may be hypoxic on scene as well as on arrival in the ED. Carbon dioxide (CO2) & carbon monoxide (CO) are produced by combustion.
  • Water soluble chemicals (ammonia, chlorine, etc.) can lead to bronchospams, edema. Lipid soluble chemicals (phosgene, nitrous oxide, etc) ? direct cell damage, impaired ciliary clearance
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9
Q

Minor Burn Treatment

A
  • First, stop burning process. Remove clothes. Run warm water over the area until the skin temperature normal
  • Next pain control
  • Next wash burn area thoroughly with soap and water
  • Dry
  • Cover with topical ointment and sterile dressing
  • Tetanus
  • Don’t give antibiotics routinely
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10
Q

Significant burn treatment

A

Fluid Resuscitation. Give IV fluids

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11
Q

Why do massive burns lead to lots of fluid loss and electrolyte imbalances?

A
  • increased microvascular permeability that leads to extracellular edema and cell membrane defects that contribute to intracellular swelling
  • have increased metabolic and respiratory rates that lead to increased evaporation and other insensible losses and often become hypoproteinemic leading to decreased intravascular oncotic pressures.
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12
Q

What is the Parkland formula?

A

%TBSA burn x wt in kg x 4cc/kg = volume of LR that should be administered over the first 24 hours

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13
Q

What kind of fluid should you give to burn patients?

A

LR

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14
Q

How should the fluid be given?

A

Half should be given in the 8 hours following the burn and the remaining half should be given over next 16 hrs (24hrs total). Remember, this is extra fluid in addition to the patient’s baseline fluid requirements.

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15
Q

What is the target urine output to monitor for?

A

Adult urine output: 0.5cc/kg/hour

Pediatric urine output: 1-2cc/kg/hour

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16
Q

What is the leading cause of death in burn patients?

A

Sepsis

17
Q

What are the three forms of prevention and control of infection in burn patients?

A
  • Debridement of devitalized tissue: cut away dead, necrotic tissue and exposure underlying viable tissue,
  • Wound management: Limit bacterial invasion by covering affected areas with antibiotic dressings and through early wound closure with skin grafting and/or commercial products.
  • Preventing the delayed development of pneumonia and sepsis: universal precautions and general infection control practices are of paramount importance. Contact isolation with gown, gloves, and mask. Frequent changing of central lines, etc. Aggressively evaluate fevers by pan-culturing and initiating broad-spectrum antibiotics.
18
Q

When do you transfer patients to a burn ICU?

A

Adult patients with >20% TBSA burns are generally transferred to a regional burn center for evaluation. Pediatric patients with >10% TBSA burns are generally transferred to a regional pediatric burn center for evaluation.

19
Q

What should you cover burns with?

A

Just cover burns with Xeroform or SSD dressings and gauze