Burns Flashcards
What is the definition of a BURN
Burns are caused by a transference of energy from a heat source to the body.
What are the aims of treatment?
- Prevention
- Best Practise Care
- Prevention of disability
- Rehabilitation
What is the functioning role of the Skin?
- Regulation of body temperature
- Excretion of bodily fluids
- Sensory perception
- Protect the inner organs
Causes of Burns?
- Thermal- Flames, Flashes, Contact with hot objects & Cigarette burns
- Chemical- Acids, Alkali, Cresols & Pesticides
- Electrical- Coagulation necrosis of the tissue due to heat generated by the current. Severity is determined by:
a) Voltage
b) Duration
c) Tissue Resistance
d) Surface area affected
e) Current Pathway - Smoke inhalation- Major determinant of mortality in fire victims.
- Radiation- UV rays, Sun beds, Exposure to radioactive materials, radiotherapy (x-rays)
Burn assessments are determined by what?
- Burn assessments are determined by the tissue involved and the % TBSA
- Determined by the layer of skin that is injured.
What are the affected layers?
- Epidermal
- Superficial dermal
- Mid dermal
- Deep dermal
- Full thickness burns
Superficial Burns
- Involves only the epidermis
- Red & painful
- Caused by sun burn or minor flashes
- Small blisters may develop/ Skin peels within delayed days
- Heals within 7 days
What are the treatment for Superficial Burns?
- First aid
- Regular application of Hydrogel
- Moisturisers to soothe
Superficial Dermal Partial Thickness Burns
- Involves the epidermis and the superficial dermis
- Painful (Nerves are exposed)
- Blisters are a trademark
- Capillaries are brisk
- Heals within 14days by epithelialisation.
Mid Dermal Partial Thickness Burn
- Dermal is damaged- Preservation is vital
- Pain present but less severe
- Capillary return is present but delayed
- Need to wait 2-3days to determine progression and healing
- Heals within 14-21days: requires silver dressing
Deep Dermal Partial Thickness Burns
- Exposed deep dermal damage.
- Pale due to damage to blood vessels
- Diminished capillary refill
- Loss of sensation (mild)
- Recovery >21days
- Treatment: First aid, silver/anti- biotic dressing & surgical interventions
Full thickness burns
- Epidermis and dermal is destroyed
- Penetration to the muscles and tendons
- Sensation is lost
- White/waxy in appearance
- Leathery (eschar)
- Management: Silver/anti-biotic dressings, & skin grafting
Total Body Surface Area
- Associates with the area that is injured
- Determines facility and degree of the treatment
- Includes 3 methods:
a) Lund Browders Chart
b) Rule of Nine
c) Palmers Method
Lund Browders Method
- Determines the % TBSA affected by splitting the body into smaller areas.
Rule of Nine
- Assigns percentages in multiple of 9s to major body parts
Palmers Method
- Taking the patients hand to estimate the % of burns, palm equates to 1%
Burn Zones
Includes:
- Zone coagulation
- Zone Ischaemia
- Zone hyperaemia
What is the Zone Coagulation?
- Irreversible Necrosis
- Nearest heat source is primary injury
- Extent determined by tempt. and duration
What is the Zone of Ischaemia?
- Reduced dermal circulation
- Potentially viable tissue
- Can progress to necrosis
What is the Zone of Hyperaemia?
- Reversible
2. Increased blood flow and inflammation
Simple Burn Pathology
- Burn
- Increased vascular permeability
- Decreased intracellular volume/Oedema
- Increased hemocrit/ Decreased blood volume
- Increased viscosity
- Increased peripheral resistance
- Leads to burn shock
Simple Burn Pathology (continued)
- Increased heart rate to try to compensate
- Continued fluid loss; Fluid starts to shift outside the vascular compartment (Oedema; Pulmonary Odema)
- Decreased organ perfusion; Leads to organ damage
- Increased viscosity (increased clotting)
Burn Pathology of the Heart
- Decreased volume triggers the SNS to release catecholamine = Increases HR; Vasoconstriction
- Myocardial contractility is suppressed by inflammatory cytokine necrosis factor.
- Most fluid is lost 24-36hrs post burn. Peaking 6-8hrs
- Capillary regains their integrity and fluid shifts back into the vascular space. Resolves the burn shock.
- Diruesis begins for days to 2 weeks post.
Burn Pathology of the LUNGS
- Inhalation injury is the leading cause of death.
- Bronchoconstiction results from the release of histamine, serotonin and thromboxane.
- High Risk of Oedema
- These patients will ultimately have 100% SpO2 however are hypoxic; supplemental O2 may be needed.
- Risk of Respiratory Failure or altered Respiratory distress syndrome.