BPPV Flashcards
benign
not malignant
paroxysmal
recurrent, sudden intensification of symptoms
positional
placement dependent (of ear)
vertigo
false inner sensation of rotational movement
BPPV symptoms are almost always precipitated by
a change in position of the head with respect to gravity
mechanism of BPPV centers around
utricular debris (otoconia)
BPPV occurs when
otoconia falls out of utricle and into either neighboring semicircular canals/cupulas
displacement can have several causes
two main causes of BPPV
trauma
age
utriculopedal
TOWARDS
- excitatory for horizontal
- inhibitory for A/P
utriculofugal
AWAY
- excitatory for A/P
- inhibitory for horizontal
canalithiasis
free floating otoconia within the SCC resulting in abnormal endolymphatic flow with the affected canal
characteristics of canalithiasis
- delay in the onset of vertigo (1-40 seconds)= LATENCY
- nystagmus appears after latency
- fluctuation in the intensity of the vertigo and nystagmus and then decreases while in provoking position= disappears in about 60s
why is anterior BPPV rare
it is hard for crystals to get into that canal
NONPATHOLOGICAL
where do eyes go if R horizontal canal and R head turn
LEFT
NONPATHOLOGICAL
where do eyes go if R anterior canal and R head turn
LEFT TORSION and UP
NONPATHOLOGICAL
where do eyes go if R posterior canal and R head turn
LEFT TORSION and DOWN
NONPATHOLOGICAL
where do eyes go if L horizontal canal and L head turn
RIGHT
NONPATHOLOGICAL
where do eyes go if L anterior canal and L head turn
RIGHT TORSION and UP
NONPATHOLOGICAL
where do eyes go if L horizontal canal and L head turn
RIGHT TORSION and DOWN
PATHOLOGICAL
where is nystagmus if R horizontal canal and R head turn
RIGHT BEATING
PATHOLOGICAL
where is nystagmus if R anterior canal and R head turn
RIGHT TORSION and DOWN BEATING
PATHOLOGICAL
where is nystagmus if R posterior canal and R head turn
RIGHT TORSION and UP BEATING