Bowel Obstruction Flashcards

1
Q

Mechanical Classification of bowel obstruction? (13)

A

Intra-luminal:
• stool Impaction
• Gallstone “ileus”
• foreign body
• trichobezoars /phytobezoars

Intra-mural
• malignancy, malignant strictures (15%)
• Inflammatory strictures (TB/ chron’s., diverticulitis, radiation colitis) (15%)
•. intestinal atresia

Extra-mural
• intraperitoneal bands and adhesions (40 %)
• Hernia
• volvulus- sigmoid (more common), cecum
• intussusception
• lymph node compression
• superior mesenteric artery syndrome

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2
Q

Anatomical Classification of bowel obstruction and major differences? (3)

A

Small bowel obstruction
- high: vomiting early and profuse with rapid dehydration; minimal distention
-Low: pain predominant with central distention, multiple central fluid level seen on AXR

Large bowel obstruction
Distension early and pronounced , mild pain; vomiting and dehydration late; proximal colon and cecum distended (competent ileocecal valve)

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3
Q

Pathological Classification of bowel obstruction and major differences? (4)

A

Simple
• one obstructive point, no vascular compromise

Closed loop
• obstruct at 2 points forming loop of grossly distended bowel, at risk for perforation (tender and perforation usually at cecum)
• >10 cm require urgent decompression
• strangulation → venous compromise → oedema → arterial compromise → gangrene and potential perforation
• causes: volvulus, bowel herniation, tight carcinomatous stricture of colon with competent ileocecal valve.

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4
Q

3 most common causes small bowel obstruction?

A
  1. Adhesions
  2. Hernias
  3. Malignancies (but still rare)
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5
Q

3 most common causes large bowel obstruction?

A
  1. Cancer
  2. Volvulus
  3. Diverticular
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6
Q

4 cardinal symptoms of bowel obstruction and differences between higher and lower obstruction?

A
  1. Pain
    • visceral colicky pain centered on umbilicus (small bowel) or lower abdomen ( large bowel)
    • complete obstruction = constant sharp pain
    • volvulus = sudden severe pain
    • paralytic ileus = no pain

2 vomiting
• high small bowel = green- blue, bile stained
• low small bowel = brown, foul smell , feculent
• large bowel: uncommon usually late symptom

  1. Abdominal distension
    • prominent in large intestine distal obstruction
    • closed loop = rif (right iliac fossa ) bulge, hyper-resonant
    • small bowel: high uncommon esp if vomiting; low central distension

4 constipation (flatus) / obstipation (nothing-complete obstruction)

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7
Q

Name 3 risk factors ischaemic bowel

A

• Atherosclerotic Rh
• heart disease
•Previous stroke

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8
Q

Findings on xray for bowel obstruction in general?

A

Erect AXR : air fluid levels
• ≥ 5 = intestinal obstruction diagnostic
• in small bowel, number of air fluid levels directly proportional to degree of obstruction and side (more-distal)

Supine AXR: dilatation of bowel

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9
Q

Investigations for bowel obstruction? (5)

A

• FBC (infections, anaemia) , uce (dehydration due to intraluminal third space loss or vomiting; acute renal failure from dehydration, k loss can perpetuate paralytic ileus)
• ABG (acidosis from bowel ischaemia; alkalosis from vomiting; lactate anaerobic respiration)
• amylase, lipase (acute pancreatitis)
• erect CXR (free air under diaphragm; aspiration pneumonia )
.Axr to assess obstruction site, severity, source etc

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10
Q

Acute Management bowel obstruction? (6)

A

• ATLS: especially breathing and spo2- may be affected by splinting of diaphragm (give oxygen)
• ngt suction large bore
• iv rehydration: water , k, cl depletion and correct electrolyte abnormalities
• catheter to monitor output: hydration
• if suspect ischaemic bowel or perforation, give prophylactic antibiotics
• surgical decompression! If bowel ischaemic, unresolved mechanical obstruction , dilation > 10cm large bowel

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11
Q

Treatment post-op paralytic ileus? (2)

A

> 3 days post op
• supportive: “drip and suck” ( Iv and ngt ), wait for peristalsis to restart
• prokinetics: erythromycin, metoclopramide, cisapride

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12
Q

Treatment sigmoid volvulus? (5)

A

Non-surgical if stable, no signs perforation or ischaemia and necrosis, no signs peritonitis
• sigmoidoscopic decompression followed by flatus tube insertion
• recur in 50%
• semi- elective sigmoidectomy within the same hospital admission

Surgical
• sigmoid colectomy with primary anastomosis (primary anastomosis only if bowel ends viable, no peritoneal contamination, haemodynamically stable)
• sigmoid colectomy with Hartman’s procedure (can be reversed in 3-6 months)
• sigmoid colectomy and formation of double barrel colostomy (paul-mikulicz procedure) with future re-anastomosis
• sigmoidopexy: fix to posterior abdominal wall (rarely done, high risk recurrence)

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13
Q

Treatment caecal volvalus?

A

Right hemi-colectomy with primary ileocolic anastomosis (not endoscopic decompression like with sigmoid, not often successful)

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14
Q

Define toxic megacolon

A

Potentially lethal complication of IBD or infections colitis, characterised by total or segmental non-obstructive colonic dilatation plus systemic toxicity

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15
Q

Clinical features and symptoms toxic megacolon? (2)

A

• Severe bloody diarrhea
• toxic patient: altered sensorium, tachycardia, fever, postural hypotension, lower abdominal distention/ tender/ peritonitis

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16
Q

Diagnostic criteria for toxic megacolon? (9)

A

• Radiologic evidence of colonic distention
• plus ≥3: FAHN
- fever > 38
- hr >120
- neutrophil > 10,500 / micoL
- anaemia
• Plus one of the following HEAD
- dehydration
-Altered sensorium
- Electrolyte disturbance
-Hypotension

17
Q

Define ogilvie syndrome

A

Acute gross dilatation of the cecum (>10cm) and right hemi-colon (occasionally extending to rectum) in absence of anatomic lesion that obstructs flow of intestinal contents. Associated with underlying disease 95% of patients

18
Q

Clinical features ogilvie syndrome?

A

• Nausea, vomiting, abdominal pain, constipation, paradoxically diarrhoea,
• abdominal distension always present

Diagnosis of exclusion - exclude toxic megacolon or mechanical obstruction

19
Q

Treatment ogilvie syndrome?

A

•Supportive
• selective use of neostigmine and colonoscopy for decompression
• Conservative therapy can be continued for 24-48 hours if no excessive pain or no excessive caecal dilatation (risk colon perforation when caecum > 12cm and when distension present for >6 days)

20
Q

Signs peritonitis on bloods?

A

Increased wCC, crp

21
Q

Signs bowel ischaemia on bloods?

A

Increased lactate and phosphate

22
Q

Management small bowel obstruction? (5) indications operative management?

A

Non-operative effective in 80%
• drip and suck always try unless signs peritonitis, strangulation, bowel ischaemia

Operative if:
• signs peritonitis, strangulation, bowel ischaemia
• water soluble contrast study hasn’t reached colon on AXR after 24h
• faecaloid drainage in ng tube
• failed non-op management 48-72 hours

Laparotomy and adhesinolysis, bowel resect if necrotic

23
Q

Name 5 red flags of constipation

A

• Distended, tympanic abdomen (mechanical obstruction)
• vomiting
• blood in stool
• weight loss
• severe constipation of recent onset or worsening in older patients

24
Q

Name 3 differentials of acute constipation

A

• Bowel obstruction: volvulus, hernia, fecal impaction
• adynamic ileus: peritonitis, major acute illness eg sepsis, bed rest, head or spine trauma
• drugs: anticholinergs (antihistamines, antipsychotics, anti parkinsonian, antispasmodics), cations (iron, aliminium, calcium , barium, bismuth ), opioids, CCB , general anaesthetics

25
Q

Name 8 differentials of chronic constipation

A

• Colon tumour
• metabolic disorders: diabetes, hypothyroid!, hypo or hyper calcaemia!, pregnancy, uraemia, porphyria.
• CNS: Parkinson, multiple sclerosis, stroke, spinal cord lesions
• PNS.: hirschsprung, neurofibromatosis, autonomic neuropathy
• systemic disorders: systemic sclerosis, amyloidosis, myotonic dystrophy, autoimmune myositis
• functional disorders: IBS, pelvic floor dysfunction,
• Dietary: low-fiber, sugar restricted, chronic laxative abuse
• dyssynergic defecation: asynchrony anorectal muscles
• drugs

26
Q

Name 7 symptoms peritonitis

A

• Fever and chills!
• abdominal pain or discomfort!
• worsening or unexplained encephalopathy
• diarrhea
• acites that doesn’t improve following diuretics
• worsening or new onset renal failure
• ileus