Body Flashcards
54F with abdominal pain, n/v
Closed loop obstruction
There is a cluster of fluid-distended loops of small bowel in the pelvis that are notable for relative absence of intraluminal gas, minimal enhancement of their mucosa, infiltrated mesentery, engorged mesenteric vessels and interloop ascites. The distal small bowel is of normal caliber.
—> findings suggestive of ischemia/closed loop include minimal enhancement of the mucosa and engorged mesenteric fat/vessels, and focal ascites.
—> in pt w/appropriate hx could also be a post-op intramesenteric hernia
54m w/cirrhosis 2/2 alcohol
Arterial phase CECT w/multiple hyperdense/hypervascular foci c/w tumor (these wash out on PV phase where the tumor thrombus is also more apparent) we also see cirrhosis w/small nodular border
Criteria to distinguish portal vein tumor thrombus (vs. bland thrombus) are contiguity with parenchymal tumor, expansion of the lumen of the vein, and enhancing thrombus.
Incidentally seen liver lesion.
Isodense to liver on other phases
FNH
homogeneously hypervascular on arterial phase imaging and nearly isodense to liver on other phases; the additional presence of a central scar is essentially pathognomonic.
—> in a younger woman w/o hx of cancer, can be dx from the CT appearance
—>hemangioma would be hypervascular but isodense to vessels, not liver.
Middle aged man c/o intermittent crampy abdominal pain and constipation
Diverticulosis w/concentric/circular muscular hypertrophy
—Also known as myochosis
get ‘cogwheel’ narrowing of the colonic lumen
RUQ pain, n/v
Gallstone eroded into duodenum, w/pneuobilia
”Bouveret syndrome”
—> gallstone iléus is by same mechanism but have SBO
—> mirizzi’s is where a stone intermittently compresses the CBD
64M w/hx of cirrhosis. P/w acute RUQ pain and this lesion had a pseudoaneurysm
HCC
Hypervascular liver mass in pt w/cirrhosis that spontaneously bleeds is essentially diagnostic of HCC
—> mets dont usually go to cirrhotic livers
—>
24 year old female with pelvic pain
Turbo-ovarian abscess
complex fluid next to the ovary w/appropriate history
IgG4 disease manifestations
MC is autoimmune pancreatitis
Can also get hepatobiliary involvement
in the chest, can see soft tissue infiltration/abnormal nodes and aortitis
—> can sometimes have normal IgG4 levels
Grandmother tumor
Serous Cystadenoma
many small cysts (>6 cysts
that are <2 cm) that may have a solid
appearance on CT due to apposition of
many cyst walls. MRI is useful to show
the cystic nature of the lesion.
hypervascular(unlike other cystic pancratic neoplasms), doesn’t cause ductal dilatation or tail atrophy (unlike adeno)
Central stellate calcs are classic
Mother Tumor
Mucinous Cystic Neoplasm
Middle aged women
benign but has malignant potential
consists of a single or a few
large cysts (<6 cysts that are >2 cm)
and typically occurs in the pancreatic
body and tail.
has a capsule (only other is SPEN)
can have peripheral calcs
Daughter Tumor
Solid and Papillary Epithelial Neoplasm (SPEN)
Young women and children, low malignant potential, typically resected.
large mass with heterogeneous solid and cystic areas.Hemorrhage is typical. Has a capsule, as does mucinous cystic neoplasm.
Crossing duct sign in Pancreas Divisum
failure of ventral and dorsal ducts to fuse
(duct of santorini is superior and drains to small (minor) papilla) while CBD always drains to major papilla
–> in divisum, main pancreatic duct drains into santorini and minor papilla, while wirsung only drains a small amount of pancreas and drains w CBD into major papilla
Pancreatic manifestations of VHL
inherited multisystemic disease w/increased risk of multiple malignancies and cysts in many organs
–>serous cystadenoma (grandma tumor) and neuroendocrine tumors
pancreatic manifestations of CF
either fatty atrophy of the pancreas
or replacement with innumerable cysts
fatty replacement of the pancreas –> exocrine insufficiency, neutropenia and bone dysplasias
Schwachman-Diamond Syndrome
2nd MCC pancreatic atrophy in childhood(first CF)
Corpus luteal cyst—>
unilocular cyst with thick, crenulated walls that enhance on CT (from peripheral vessels, correlates with ring of fire on US)
Type of functional ovarian cyst that forms when corpus luteum fails to involute after ovum is released
remnants of the follicle after ovulation; ranges from 2-5 cm in diameter but involutes as it matures. The corpus luteum produces estrogen and progesterone, maintaining optimum conditions for implantation if the ovum is fertilised:
fertilised: the corpus luteum continues to produce these hormones and maximizes the chance of implantation into the endometrium; it reaches a maximum size at ~10 weeks and finally resolves at around 16-20 weeks
not fertilised: the corpus luteum involutes and turns into a corpus albicans by around 2 weeks
In some instances (like twin pregnancies), may be more than one corpus luteum - (e.g. two corpora lutea)
what grade ?
Grade 1:
subcapsular hematoma or contusion, without laceration
what grade?
Grade II Renal trauma
superficial laceration ≤1 cm depth not involving the collecting system (no evidence of urine extravasation)
perirenal hematoma confined within the perirenal fascia
Grade III Renal Trauma
laceration >1 cm not involving the collecting system (no evidence of urine extravasation)
vascular injury or active bleeding confined within the perirenal fascia
What grade?
Grade IV renal trauma
laceration involving the collecting system with urinary extravasation
laceration of the renal pelvis and/or complete ureteropelvic disruption
vascular injury to segmental renal artery or vein
segmental infarctions without associated active bleeding (i.e. due to vessel thrombosis)
active bleeding extending beyond the perirenal fascia (i.e. into the retroperitoneum or peritoneum)
what grade?
Grade V
shattered kidney
avulsion of renal hilum or laceration of the main renal artery or vein: devascularisation of a kidney due to hilar injury
devascularised kidney with active bleeding
+ calcs/staghorn calculus
xanthogranulomatous pyelonephritis
layering calcified stones w/in the renal parenchyma
renal diverticulum.
will see it fill on CT also
hematocrit sign in hemorrhage
specific for coagulopathic hemorrhage
–> e.g. over anticoagulation w/warfarin
Schistosomiasis
(calcs in the bladder wall)
Other forms include hepatosplenic (fibrosis and portal HTN, splenomegaly)
pulmonary (
RCC OR Multilocular cystic nephroma
*can’t be distinguished just on imaging appearance
both can enhance and calcify.
Ovarian Cystadenoma (serous)
usually unilocular cystic mass with thin walls
Mucinous is multilocular with thin walls also
Ovarian mass, multiloculated with soft tissue and thick septae
mucinous cystadenocarcinoma
Serous is unilocular, also thick walled / solid components
—> hepatic adénome seen in young women on OCPs, usu heterogeneously hypervascular w/foci of fat, necrosis, and hemorrhage
what are possible patterns of enhancement for this lesion
hepatic cavernous hemangioma
–cyst with enhancing mural nodule
–peripheral, nodular enhancement, isodense to vessels on all series
>1 year post transplant
PTLD
retroperitoneal adenopathy. Can involve adrenals, kidney, GI, other organs etc.
Here—indistinguishable from NHL in immunocompetent patient
seen in what syndrome
Gardner syndrome
familial adenopolyposis
multiple osteomas: especially of the mandible, skull, and long bones
epidermal cysts
fibromatoses
desmoid tumors of mesentery and anterior abdominal wall
Other abnormalities include:
supernumerary teeth/duodenal tumors/papillary thyroid carcinoma
Multiple of these:
Odontogenic keratocyst
Seen in Gorlin Syndrome/basal cell nevus syndrome
phakomatosis—>
Craniofacial anomalies, multiple BCCs, shortened 4th metacarpals
—> hepatic adénoma
young women on OCPs, usu heterogeneously hypervascular w/foci of fat, necrosis, and hemorrhage
Horseshoe kidney
middle aged woman with fatigue and pruritis
PBC (primary biliary cholangitis)
lace-like fibrosis and periportal halo sign (hypodensity around portal veins thought 2/2 portal edema)
can see periportal hyperintensity (cuffing) on MR
segmental hypertrophy, splenic hypertrophy
recurrent pyogenic cholangitis
present w/recurrent RUQ, fever, and jaundice
diagnostic clues: intra- and extrahepatic biliary dilatation and multilevel strictures with intraductal pigmented calculi(T1 bright), usually in the absence of gallbladder calculi
a/w clonorchis or ascariasis (liver fluke) infection (serves as a nidus for pigmented gs formation)
–reduced arborization of peripheral ducts on MR–‘arrowhead sign’, multiple strictures
hepatolithiasis
causes include recurrent pyogenic cholangitis, ductal stasis, caroli disease
common in asia and the pacific (rare in america)
What is the entity?
Why is this not Ogilvie/colonic ileus?
Toxic megacolon
with ahaustral appearance of the transverse colon. Get sloughing of normal mucosa, and patient is very sick
Ogilvie syndrome/colonic ileus
characterized by disproportionate dilation of cecum and ascending colon without obstruction. Will have normal appearing haustra/transverse folds and mucosa, patient not as sick
Pt with brief syncopal episode after HD
Amyloid Spondyloarthropathy
associated with chronic renal disease/hemodialysis
advanced disc space loss with endplate erosion/destruction and malalignment, (findings that closely parallel infectious discitis)
Epiphrenic diverticulum
Pulsion diverticulum
Notice that there aren’t any RUGAE in the thing that looks like it could be intrathoracic stomach
Happens in old people with dysmotility
