Board Review Mix 2 Flashcards
ASD with abnml p wave axis
sinus venosus , < 30 degrees
what to do with PDA after NEC?
immediate surgical closure
immigrant with PDA, risk of IE? where? organisms?
15% total IE in other countries, IE on PA end of PDA
strep species
rupture sinus of vals any, MC location, associated defects?
VSD in 50% (outlet septal defect), RV MC
coronary sinus diverticulum is associated with?
accesory pathway
late truncus repair? most important consideration?
PVR < 8. reactive PVR < 8 if less than2 years old
another name for asymetric congenital MS?
parachute mitral valve
anatomic feature of arcade mitral valve?
paps connected directly to the valve leaflets, easy to see on PS long
mitral valve prolapse, when are sports not OK?
If SVT or NSVT on holter
location AV node with ccTGA?
anterior location
partial pericardial defect, presentation? Dx? common risk?
Dx: MRI, Risk incarceration of the LAA
Restrictive cardiomyopathy, MC arrythmia?
Atrial flutter
oxygen excess omega formula
oxygen delivery / oxygen consumptions
Art O2 / (Art O2 - SVO2)
Art O2 / AVO2 difference
higher is better
anticoagulant mechanisms:
acetylation of cyclo-oxygenase to inhibit production of thromboxane A2
Aspirin - acetylation of cyclo-oxygenase to inhibit production of thromboxane A2
heparin - mucopolysaccharide that nc rate of AT3 neutralizing thrombin, factor X
streptokinase - interacts with plasminogen to result in plasmin complex
tPA - binds to fibrin
warfarin - vit K blocker
anticoagulant mechanisms:
mucopolysaccharide that inc rate of AT3 neutralizing thrombin, factor X
heparin - mucopolysaccharide that inc rate of AT3 neutralizing thrombin, factor X
Aspirin - acetylation of cyclo-oxygenase to inhibit production of thromboxane A2
streptokinase - interacts with plasminogen to result in plasmin complex
tPA - binds to fibrin
warfarin - vit K blocker
anticoagulant mechanisms:
interacts with plasminogen to result in plasmin complex
streptokinase - interacts with plasminogen to result in plasmin complex
Aspirin - acetylation of cyclo-oxygenase to inhibit production of thromboxane A2
heparin - mucopolysaccharide that nc rate of AT3 neutralizing thrombin, factor X
tPA - binds to fibrin
warfarin - vit K blocker
anticoag mech:
binds to fibrin
tPA - binds to fibrin
Aspirin - acetylation of cyclo-oxygenase to inhibit production of thromboxane A2
heparin - mucopolysaccharide that nc rate of AT3 neutralizing thrombin, factor X
streptokinase - interacts with plasminogen to result in plasmin complex
warfarin - vit K blocker
how to calculate MVV?
5 x total lung capacity
35-40 x FEV1
what does inert gas rebreathig actually measure?
effective pulmonary blood flow - without shunts, assumed to be Qs
how does T wave in lead V1 change over age?
starts as poitive
negative by age 7 DAYS
positive in adulthood in some
Qt proloning antiobitocs
azithromycin
erythromycin
difference in EKG between cocaine and myocarditis
cocaine is focal ST seg change
myocarditis is diffuse
what does alternative T waves mean?
LQTS - risk of 2:1 block
significant coarct narrowing?
50% decrease in CSA = 10 mmHg
shortening fraction change with age?
starts high (45%) then falls circumferential predominates at first
metal with most artifact
stainless steel
associated CHD with coarctation? 1st and 2nd
BAV 1st
VSD 2nd
holodiastolic flow reversal indicates what about the aortic valve
consistent with severe regurgitation
endogenous pulmonary vasodilators
dilators: acetylcholine, badykinin PGE E1,E2,I2 (vowels = open), iNO
constrictors: ATII, Leukotrienes, PGE F2, thromboxane A2, serotonin
mesalamine puts you at risk for
myopericarditis (seen in kids with ulcerative colitis)
ectopia cordis asociated with which cardiac abnormalities? other defects? survival?
conotruncal
midline defects
10% survival to 1 year
levosimendan MOA
way to remember
benefits?
Calcium sensitizer: binds troponin C< inc sensitivity to calcium, also open K channels in smoothe muscle, reducing afterload via vasdilation
LEVER - C - MEN : Leverages calcium MEN
No data to support benefits in adults or KIDS.
early vs late HLHS formation
early: superior aspect of septum primum deviates anterior and leftward, causes restrictive atrial septum, limits LV preload
late: aortic valve issue (endocardial cusion)
marked drop in impedence of a pacemaker lead
insulation breach - prevented by axillary placement
subclavian crush as lead passes between first rib and clavicle
most common presenting symptom of coronary cameral fistula
left to right shunt