Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

A2 Psychology > Biopsychology > Flashcards

Biopsychology Flashcards

You may prefer our related Brainscape-certified flashcards:
Psychology 101 flashcards GRE® Psychology flashcards AP® Psychology flashcards
1
Q

Parts of the brain

A 
Motor cortex
Somatosensory cortex
Visual cortex
Wernicke's area
Auditory cortex
Broca's area
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Motor cortex

A

Controls voluntary movement.

Frontal lobe.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Somatosensory cortex

A

Detects senses.

Parietal lobe.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Visual cortex

A

Processes information from sight/eyes.
Receives information via the phrenic nerve.
Occipital lobe.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Auditory cortex

A

Processes information from hearing/ears.

Temporal lobe.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Broca’s area

A

Controls production of speech.

Frontal lobe.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Wernicke’s area

A

Responsible for understanding of language.

Temporal lobe.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What are the two brain hemispheres connected by

A

Corpus callosum

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Left hemisphere handles…

A

Language functions.

Also logic, analysis and problem solving.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Right hemisphere handles…

A

Spatial comprehension, emotions and facial recognition.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What do the left and right hemispheres control?

A

Left controls: right body

Right controls: left body

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Split brain surgery

A

Severs the corpus callosum in severe cases of epilepsy. This stops seizures spreading across the brain.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Effects of split brain surgery study

A

Sperry and Gazzaniga 1967

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Sperry 1968
Method and results

Effects of split brain surgery

A

Method: 11 participants, androcentric. Control group (with corpus callosum).
Participants covered one eye and looked at fixed point on screen. Pictures projected on left or right of screen at high speed so no time for eye movement.
Result: picture shown on right, participants could say or write what it was no problem. If shown on left, participants could not say or write what it was, but could feel for the representative object in left hand if what had been shown to left visual (right hemisphere) but didn’t know why they selected that.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Sperry 1968
Conclusion and evaluation

Effects of split brain surgery

A

Conclusion: Shows different areas of the brain specialise in different functions.
Left hemisphere receives from right visual converts sight into spoken and written language.
Right hemisphere receives from left visual can produce non-verbal response.
Evaluation: got qualitative and quantitative data. Small sample - low pop validity. Epilepsy patients also on meds - affected brains? Can’t compare to non epilepsy. Low ecological validity as artificial experiment situation - hard to generalise to real-life.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Methods of studying the brain

A

fMRI
EEG
ERP
Post Mortem Examination

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

fMRI

A

Carries out task while in scanner, part of brain involved will be more active (more oxygen there from red blood cells) and is coloured on computer.
Used to diagnose medical problems as show damaged/diseased areas of brain.
+non-invasive
- expensive
- not good for those with claustrophobia
- poor temporal resolution (don’t show changes over time)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

EEG

A

Shows overall electrical activity of the brain - picks up signal of many neurons.
Electrodes on scalp and activity recorded which produces a pattern of waves.
Used in sleep studies.
Help diagnose depression/schizophrenia/anorexia nervosa.
Use to diagnose.
+non-invasive
+ cheaper
+good temporal resolution
- poor spatial resolution (hard to work out specific area of brain waves originate).

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

ERP

A

Method of EEG but patient carries out activity to see how wave patterns change in response to a stimulus. Same task carried out to cancel ‘noise’ and focus on specific function.
Used in memory research- information processing in the brain.
Help diagnose those with psychiatric disorders by comparing waves to normal people.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Post mortem examination

A

Shows structural abnormalities that could explain condition.
+Provided evidence for localisation of function.
E.g. Paul Broca did PM on two people with speech problems due to brain damage and found they both had damage to same area (now called Broca area).
- person had to die so they won’t benefit from results.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Disadvantage of all methods of studying the brain

A

Don’t allow us to determine cause and effect.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

What does fMRI stand for

A

Functional magnetic resonance imaging

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

What does EEG stand for

A

Electroencephalogram

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

What does ERP stand for

A

Event-related potential

25
Q

Evidence for Broca’s area controlling language

A

Carried out post-mortem on patient Tan who had speech problems during his life.
Paul Broca was able to identify what became known as Broca’s area in the brain.

26
Q

Brain plasticity

A

The brain is flexible and can change shape.

27
Q

How does the brain change shape

A

Bridging: new connections are formed due to use and new stimulus.

Pruning: where connections are lost due to lack of use.

28
Q

Studies for Brain plasticity and functional recovery?

A

Kuhn 2014
Davidson et al 2004
Kempermann 1998
Maguire 2000

29
Q

Kuhn 2014

A

Participants play super mario bros for 30mins per day for 2 months. Compared to group not playing.
Increased grey matter of gamers -motor cortex, cerebellum (coord of movement) and hippocampus.
+ longitudinal (reduce individ. diff, shows cause and effect)
+ real world application (games are good)
- extraneous variables (not lab, other factors effect)

30
Q

Davidson et al 2004

A

Tibetan monks compared to non meditation controls. Both groups meditate for short period - then fitted to electrical sensors to detect activity in brain.
Monks had more gamma activity (which helps coord neuron activity).
+ real life application
+ used a control group
- small sample group
- extraneous variables
- monks are hard to generalise as such a different lifestyle and have been meditating for a long time

31
Q

Kempermann 1998

A

Found that rats who were raised in an enriched environment (more stimulation, more colours) had an increased amount of neurons compared to rats raised in lab cages.

32
Q

Maguire 2000

A

Found that London taxi drivers had a larger posterior hippocampus than non-taxi drivers. This part is responsible for spatial navigation.
To provide more evidence for cause and effect he found that those who had been drivers for longer had a larger hippocampus.

33
Q

Studies for functional recovery of the brain

A

Teuber 1975

Schneider 2014

34
Q

Teuber 1975

Age

A

If you are older, you are less able to recover.

60% under 20s showed significant improvement.
20% over 26s.

35
Q

Schneider 2014

A

More educated - better recovery.

Those who went to college (or equivalent) are seven times more likely to be disability free 1 yer after traumatic brain injury than those who didn’t finish school.

36
Q

Localisation of function evaluation

A

Strengths:
- support from aphasia case studies
- support from brain scanning techniques
- support from phantom limb
Weaknesses:
- case studies specific and rare
- communication not localisation (pathway more damaged than areas)
-individual differences (no two people are the same)

37
Q

Evaluation of lateralisation and split brain research

A

+ scientific cause and effect
+ supported by evolutionary theory (more efficient to have hemispheric lateralisation)
- small sample sizes, low pop and temporal val.
- participants epileptic, normal brains to compare?
- Gazzaniga and Turk both discovered some ability to speak in the right hemisphere as well.
- case studies: individual differences (some people have more lateralised brains) women have less.
- Reductionist explanation

38
Q

Functional recovery after trauma

Neuronal Unmasking

A

Neuronal unmasking: activating dormant synapses after trauma to compensate for the damaged synapses.

1) Axon sprouting: axon is arm of nerve cell - helps connect to other neurons. Sprouting: new axons grow and connect to undamaged areas.
2) Recruitment of homologous areas in opposite hemisphere: functions taken care of by the dormant areas of the other hemisphere.

39
Q

Study for neuronal unmasking

A

Wall 1977

Found brains contain a lot of dormant synapses which are not receiving input but can be still be called upon in need.

40
Q

Example of someone who managed functional recovery after a trauma

A

Ben Underwood

Lost his eyes to cancer and regained sight through echolocation.

41
Q

Functional recovery after trauma

Stem cells

A

Unspecialised cells with the potential to perform any function.
Implanted into the brain to deal with trauma recovery.

1) Direct implants: replace damaged cells
2) Rescue implants: release growth hormones to repair damaged cells.
3) Neural networks: link damaged to undamaged to create functional recovery.

42
Q

Circadian Rhythms

A

Rhythms which work on a daily cycle.

Sleep-wake cycle.
Hormone production:
Melatonin: makes sleepy, increases before bedtime, dec as day progresses as inhibited by light
Cortisol: inc alertness, peaks at 6am ready to wake up, lowest around midnight ready for sleep.

43
Q

Exogenous zeitgebers

A

External cues

44
Q

Endogenous pacemakers

A

Internal cues

45
Q

Study for Circadian Rhythms

Michel Siffre

A

Lived underground in cave for 6 months, Texas.
No external cues , internal body clock influenced behaviour.
No daylight, clocks or radio.
Concluded: natural circadian rhythm settled to just over 24 hours.
+ got into internal rhythm due to endogenous factors (25 hours).
- some dramatic variations -up to 48hours- so external cues needed to regulate cycle.

46
Q

Evaluation of Siffre

A

Case study of 1 man:
+ rich in detail
- individual differences, androcentric
Used dim artificial light to get around:
+ dim light, less impact, ethical as less likely to get injured.
- external factor, cue? (Czeisler)
Cold dark cave 6 months:
+ longitudinal, see change
- unrealistic situation, hard to generalise.
Real world app:
+ Gov. dim public lighting at certain times
+ NASA space exploration
+ therapy for sleep deprivation
- flawed study, society shouldn’t change based on this study

47
Q

Circadian Rhythms

Czeisler

A

1999:

  • Found circadian rhythms of different people vary, from 13-65 hours. So can’t generalise, poor pop validity.
  • Altered participants circadian rhythms down to 22hrs or up to 28hrs by using dim artificial lighting.
48
Q

Circadian Rhythms evaluation

A 
\+ scientific measures
\+ research support, Hughes
\+ real world app (chronotheraputics, timing of drug treatment)
- use of artificial lighting (czeisler)
- could be temperature not lighting
49
Q

Ultradian Rhythms

A

A type of biological rhythm with a frequency of more than one cycle in 24 hours.

Sleeping is made up of cycles.

50
Q

The Sleep Stages

A 
1. Hallucinations 
5-10mins, 4-5%, transition from awake to sleep
2. Brain wave spindles
20mins, 45-55%, HR slows, brain activity 
3. Delta waves
4-6%, transition light to deep sleep
4. Delta Sleep
30mins, 12-15%, deep sleep

3+2 repeat
REM Sleep
20-25%, (dreams here) inc respiration and HR

51
Q

Sleep Stages Order

A

1234 Only go through 1 once

32 REM BRAC (brain rest activity cycle)

234 Cycles last 90 mins and repeat
until awake.
32 REM

234

52
Q

Infradian Rhythms

A

A type of biological rhythm with a frequency of less than one cycle in 24 hours.

E.g. Menstrual cycle

53
Q

Seasonal affective disorder (SAD)

A

Feelings of sadness/depression during winter when there is less light.
Due to too much melatonin in the brain, a hormone inhibited by light (produced in abundance in dark).
Leads to sleepiness and feeling weary in the day.

Treatment is exposure to light.

54
Q

Evaluation of Ultradian Rhythms

A

+ Russell (sweat lip study)
+ Penton-Voak (menstrual cycle and mate choice)
+ effectiveness of phototherapy
- arguments against Russell, cycles vary in length and match up by chance

55
Q

Endogenous pacemaker

A

SCN (suprachiasmatic nucleus): main pacemaker in brain, hypothalamus, regulates circadian rhythm.

Has its own internal clock based on cellular processes.
SCN is endogenous but relies on external info to ensure synchronisation.

56
Q

SCN

A

-cycle resets by use/absence of light
- type of light receptor in retina gauges brightness
With light: receptors send melanopsin to SCN
SCN communicates with pineal gland to inc melatonin in absence of light and inhibit production with light.

57
Q

Exogenous zeitgebers

A

Light: regulates rhythms with melatonin and cortisol

Social cues: regulated by presence of others.

58
Q

Evaluation of exogenous zeitgebers and endogenous pacemakers

A

Endogenous:
Morgans hamsters
DeCourseys chipmunks

Exogenous:
Skene and Arendt
Klein and Wegmann
Burgess et al
Vetter et al
59
Q

Outline the structures and processes involved in synaptic transmission

A

Neurotransmitters in vesicles in pre-synaptic terminal release neurotransmitters into synaptic cleft when stimulated by action potentials/nerve impulses arriving at pre-synaptic terminal.
Neurotransmitters combine with post-synaptic receptors.
Post-synaptic effects whether its going to be either excitatory or inhibitory.

A2 Psychology (11 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions