Biomechanics Feb 4 - intro and neural adaptations to training Flashcards

1
Q

A group of sarcomeres make up a ___

A

myofibril

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2
Q

A group of myofibrils make up a ____

A

m fibre

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3
Q

At the level of the sarcomere, what are the two basic tenets (force - __ relationships) that make up the BL tenets

A

force v
force L

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4
Q

In addition to the MTU, what is the neuro make up of the muscle look like/mechanisms at play?

A

mo unit which is the mo neuron and the fibres it innervates

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5
Q

Each muscle has ++ motor __

A

units

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6
Q

Constraints based approach of our system - redundancy in degrees of freedom of movement, what does this mean?

A

multiple ways to do a single task

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7
Q

Determinants of strength and power - what factors determine force?

A

m mechanics
neural involvement
morphology
m environemnt

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8
Q

Myosin heavy chain fibres- slow - what type are these and what do they help with?

A

type I
antigravity with w bearing and sustaining movement

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9
Q

Myosin heavy chain fibres- fast oxidative - what type are these and what do they help with?

A

IIa/IIx
sustained locomotor

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10
Q

Myosin heavy chain fibres- fast glycolytic - what type are these and what do they help with?

A

IIb, IIx
burst power

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11
Q

What determines how fast type I, IIa, b, x fibres contract?

A

ATPase - connection and reconnection

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12
Q

Factor 1: muscle mechanics: how does this effect force? (basic terms)

A

F-L relationship
F-v
F-v-power

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13
Q

Fmax depends on ? and ?

A

CSA
vmax/mo unit type

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14
Q

Factor 2 of m force contributor: morphology - what are the two subtypes of interest in this?

A

CSA
pennation angle

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15
Q

Cross sectional area - what causes more force?

A

more contractile pro = more cross bridge = more force

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16
Q

Pennation angle: what is this?

A

angle between tendon and m force

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17
Q

How does pennation angle increase force?

A

increase in angle means m fibres operate at better position but decr shortening velocity potential/vmax

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18
Q

How does increased pennation angle effect vmax?

A

decrease

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19
Q

Factor 3 effecting muscle force: neural inputs - what are the different components of the feedback mechanism?

A

m spindle modulates and at golgi tnedon organ
- intra and inter muscle coordination: how active agonist, antagonist are
-inter m + segments proximal to distal sequencing

20
Q

What two components determine neural activity/drive?

A

number of recruited mo units and firing frequency

21
Q

How is neural activity often measured?

22
Q

In EMG, bigger peak and trough mean?

A

incr neural activation

23
Q

In the short term, what seems to increase the most strength training enhancements when comparing neural drive vs CSA vs pre training

A

neural drive!
> csa
> PT

24
Q

The stronger an athlete is … how does this effect adaptation?

A

less adaptability

25
If untrained, how does agonist antagonist coactivation seem to occur vs in a highly trained athlete?
high level of coactiv in low tr vs highly trained athlete has less of this
26
Why would we expect to see a high level of coactivation of agonist and antagonist in athletes who are untrained?
stability protection and breaking in ballistic movement
27
We tend to see the most adaptation in a short period from untrained individuals via neural adaptation. How does this neural drive change with detraining (Hakkinen 1983)
lost quickly force production ability decrease less, so likely some morphologic changes from initial bit are protective
28
EMG vs torque graph: what changes indicate neural adaptation?
increase y axis
29
EMG vs torque graph: what changes indicate morphologoical change
increase x axis
30
Increases in neural adaptation are often due to firing frequency ___ and or mo unit ___
increase recruitment
31
How does the reflex mechanism work in str training and plyometrics?
increase excit of m spindle (incr facilitation) decr excit and desens golgi tendon organ (to decr inhibition) overall decr inhib for excitatory action
32
Pre-activation (ex pre activ prior to ground contact in plyos) may be hlepful for incr force production why?
may benefit s+C by modulating stiffness may protect against injury through incr stability
33
Explosive strength: on a joint force vs time graph, how is this shown?
inverted U max strength at peak of inverted U rise on first part of U indicates explosive strength
34
Explosive str defn
how much of max strength can generate over short timeframe
35
Rate of force development/RFD defn
specific measure where explosive strength in quality to teast out
36
Common metrics used to describe explosive strength (3)
force at given time point RFD: change in f/change intime - steeper slope = higher rate of force development impulse: area under the curve
37
Common ph used in research used to determine overall explosive strength production: early vs mid vs late
0-50ms 50-100 >100
38
What two main pieces are considered in rate of force development?
Max force time to achieve a given percentage of max force
39
RFD: max force determinants
due to max strength due to CSA
40
RFD: time to achieve a given % of max F - 3 parts?
neural activation insttrinic contractile properties tendon stiffness all determined in some part by contraction speed and type
41
Max force ultimately determined by what "phase" of explosive force development
late >100ms
42
Early ph force development typically determined most by what concept?
neural drive and twitch (force by single stim/instric contraction properties of m fibre)
43
Mid ph force development typically determined most by what concept?
intrinsic - voluntary activation
44
Importance of intent in training: Tillin 2014 paper took isometric training where group 1 trained as hard and fast as possible vs conventional group at 75% max voluntary force (ie greater time under tension at lower intensity). What were the findings of force initially and at 12 weeks pf training?
both incr force but conventional f 2x > than gr 1 0-150ms only explosive gr increased time 0-50ms (neural) at 12 weeks: matched tr type - conventional only incr rate at 150 whereas group 1 increased rate 0-50, 50-100 and >100 CSA changed in conventional group, not group 1
45
Interesting note on training type IIx fibres - can we do this?
not really likely fast enough to train this so ultimately train IIx--> IIb moreso