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Endodontics 1 > Biology and Histology of Pulp and PA Tissues > Flashcards

Biology and Histology of Pulp and PA Tissues Flashcards

1
Q

Central mass of a tooth consisting of soft tissue that is densely innervated by afferent sensory fibers, s

A

Pulpal tissue

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2
Q

_____ secretes type 1 and 3 collagen, repair connective tissue by digesting collagen, and form and repair connective tissue

A

Fibroblasts

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3
Q

______ forms tooth structure, produce collagen fiber and matrix, can’t divide, forms predentin that can become mineralized; in close relation with a-delta nerve

A

Odontoblasts

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4
Q

What is the primary cell in pulpal tissue?

A

Fibroblasts

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5
Q

________:

Anything that causes movement of fluid w/in dentinal tubules that causes pain in pulp

A

Branstromm’s “Hydrodynamic Theory”

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6
Q

______ is cortical bone that surrounds the roots of a tooth

A

Lamina dura

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7
Q

Function of _____ is to create tooth structure

A

Odontoblasts

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8
Q

The odontoblasts produce ______ while the tooth develops

A

Primary dentin

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9
Q

The odontoblasts produce _____ once the tooth is fully developed

A

Secondary dentin

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10
Q

The fibroblasts produce ______ in response to injury or trauma

A

Tertiary dentin

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11
Q

Which forms of dentin lack dentinal tubules?

A

Tertiary dentin

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12
Q

Do younger or older people have less pulp due to secondary dentin causing pulp chambers to shrink?

A

Older people

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13
Q

____ changes shape of pulp chamber: is deposited over time in the immediate area of the injury by fibroblasts in an attempt to repair, protect or insulate the pulp from further injury. General FORM of PULP is altered.

A

Tertiary dentin

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14
Q

•Probably a combination of 2ndary and Tertiary dentin formation in response to extensive and chronic injuries before the pulp became necrotic. (no deposition unless pulp is vital)•This canal is not negotiable by normal means: nerves of dentin are injured: odontoblasts and fibroblasts lay down 2nd and 3o dentin; pulp is chronically inflamed but not dead or necrotic yet

A

Dystrophic calcification

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15
Q

1 reason for dental pulp injury is ________

A

Microbiological

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16
Q

The pulp amazingly resilient if covered by at least _____ mm of healthy dentin. Reasonable trauma can be survived if bacteria are minimized or eliminated

A

.5 mmm

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17
Q

The #1 threat to health of the dental pulp is :

A

DENTAL CARIES

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18
Q

Without _____ & their products, LEOs (Lesions of Endodontic Origin) do NOT occur and dentistry would not exist as the profession we know today.

A

MO

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19
Q

What is the #2 reason for pulpal injury?

A

Fracture/Cracks

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20
Q

How are

A

Caries
Crack
Foramen
Open dentinal tubules

21
Q

____ occurs when old burs are used or inssuficient water used in an operative proceudre causing aspiration of tubules

A

Necrosis

22
Q

-Minimal insult
-Healthy pulp
•Minimal odontoblast damage
•Blood vessels engorge
•Intrapulpal pressure increases
•Exaggerated pain to thermalstimuli
•Pain sharp and short lived
•A delta fibers
•Not lingering*
•Not spontaneous*
•Generally capable of Healing
Use Desensitizing agents GLUMA

A

Reversible pulpitis

23
Q 
•Extensive odontoblast damage
•Blood vessels greatly engorge
•Intrapulpal pressure increases greatly
•Exaggerated pain to thermalstimuli•Pain Intense (Acute Pulpitis)
•C fibers
•Lingering *•Spontaneous *
•Incapable of Healing *
-Throbbing pain
A

Irreversible pulpitis

24
Q

Which 2 types of nerve fibers are present in pulp?

A

A-delta and C fibers

25
Q

____ fibers produce throbbing, lingering pain; associated with dull, diffuse pulpal pain (think CHRONIC damage) IP

A

C fibers

26
Q

_____ fibers: associated with early sharp pain (think ACUTE) maybe RP

A

A delta fibers

27
Q

_______: the pulp is symptom free and positive response to sensibility testing

A

Normal

28
Q

________: subjective or objective findings indicating the inflammation should resolve on its own and the pulp return to normal (A delta fibers)

A

Reversible Pulpitis

29
Q

Is dystrophic calcification SIP, RP, or AiP?

A

AIP

30
Q

Is internal root resorption SIP, RP, or AiP?

A

AIP

31
Q

inflamed pulp is not capable of healing(usually C fibers)–-lingering thermal pain, spontaneous pain, referred pain

A

SymptomaticIP

32
Q

inflamed pulp is not capable of healing(usually C fibers)–-no clinical symptoms but inflammation produced by caries, caries excavation, trauma, etc

A

AIP

33
Q

:(Death of the pulp) No response to pulp tests

A

Pulp Necrosis

34
Q

: Previously obturated

A

Previously Treated

35
Q

:Tooth previously treated by partial endodontic therapy (i.e. pulpotomy, pulpectomy)

A

Previously initiated therapy

36
Q

________: is very richly supplied with collateral circulation as well & has a nearly unlimited source of undifferentiated cells for inflammation and repair. Therefore capable of both exudative (fluid) & formative(cellular) defense mechanisms; can be healed via rich blood supply

A

Peri-Apical Tissue

37
Q

: When the defense mechanism (Pulp or P/A) is overwhelmed, an exudative response results with production of fluids = pain = symptomatic. (Rapid Swelling + Systemic Response – Fever, etc.)

A

Acute Inflamm

38
Q

_____ INflam: When the progress of the disease is of lesser intensity over a longer time, aproliferative or cellular response results (lymphocytes, macrophages & plasma cells. etc.) = a “standoff” between the body’s immune system and infection. usually evolve some type of drainage: Draining Sinus Tract (DST) etc.). As a result, symptoms minimal or none (due to release of pressure/purulence). DST is important in the DX of Chronic Apical Per

A

Chronic inflam

39
Q

What is the term for a reversible inflammation that has gone from chronic to acute?

A

Recrudescent Abscess.

40
Q

An inflammatory reaction to pulpal infection characterized by rapid onset, spontaneous pain, tenderness of the tooth to pressure, pus formation and SWELLING of associated tissues

A

Acute Apical Abscess: (AAA)

41
Q

An inflammatory reaction to pulpal infection and necrosis characterized by gradual onset, little or no discomfort and the intermittent discharge of pusThrough associated DRAINAGE *(DST Draining Sinus Tract or via Sulcus).

A

Chronic Apical Abscess:(CAA)

42
Q

: Diffuse increased trabecular radiopaque lesion representing a localized reaction to a low-grade inflammatory stimulus. Pulp may be vital and inflamed or necrotic. P/A radiolucent inflammatory lesion may or may not be present at apices. Treatment varies depending upon pulpal conditions and symptoms.Older terms: focal sclerosing osteomyelitis & a variant of chronic asymptomatic apical periodontitis)

A

Condensing Osteitis (CO)

43
Q

, (schlerotic bone) which is dense (but normal bone), homogenious, defined radiopacity devoid of pathosis (no treatment req.) Other terms: Enostosis, Osteosclerosis, Periapical Condensing Osseous Dysplasia (PCOD),

A

Ideopathic Osteosclerosis

44
Q

:Teeth w normal periradicular tissues that will not be abnormally sensitive to percussion or palpation. The LD (Lamina Dura) surrounding the root is intact and the PDL space is uniform.

A

Normal Apical Tissues

45
Q

What cells are prevalent in microabscesses in pulp?

A

Neutrophils

46
Q

Inflammation of the apical periodontium, producing clinical symptoms including painful response to biting and percussion.It may or may not be associated with an apical radiolucent area

A

Symptomatic Apical Periodontitis(SAP)

47
Q

Inflammation and destruction of apical periodontium that is of pulpal origin, appears as an apical radiolucent area and does not produce clinical symptoms

A

Asymptomatic Apical Periodontitis: (AAP)

48
Q

:Central mass of a tooth consisting of soft tissue that is densely innervated by afferent (sensory) fibers, sympathetic fibers,vascular structures, lymphatics and specialized cells such as odontoblasts(characteristic cells of pulp) and fibroblasts(most common/prevalent cell type in pulp)
*Primary Function: Formative: odontoblasts – dentin (with dental epithelium) ameloblasts – enamel
Secondary Functions:Supportive Protection, Sensation, Defense

A

Pulpal organ

Endodontics 1 (13 decks)

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