Biological Flashcards

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1
Q

Brain structure

A

Frontal lobe - high level cognitive functions, such as thinking and planning
Parietal lobes - processes sensory information from the skin
Temporal lobes - deals with auditory information and has a role in understanding language
Occipital lobes - deals with visual information

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2
Q

Hypothalamus

A

Controls motivational behaviours such as hunger and thirst and has a key role in the fight or flight response. Maintains homeostasis and the activity of the endocrine system

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3
Q

Limbic system

A

Consists of several structures such as the amygdala, which plays a large role in regulating emotional response. It also has a role in memory and learning

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4
Q

Cerebellum

A

Coordinates posture, balance and movement.

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5
Q

Structure of a neurone

A

Dendrite - carry impulses from neighbouring neurones to the cell body
Axon - carries impulses away from the cell body and down the neurone
Myelin sheath - fatty insulating cells that speed up the action potential
Nodes of Ranvier - gaps in the myelin sheath where an action potential can be formed

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6
Q

Sensory neurones

A

Carry messages from the sensory receptors (touch etc) to the CNS
Long dendrites, short axon

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7
Q

Motor neurones

A

Carry messages from the CNS to the effector (muscle or gland)
Short dendrites, long axon

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8
Q

Relay neurones

A

Connect sensory and motor neurones in the CNS
Short dendrites, short axon

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9
Q

Functions of a neurone

A

When a stimulus of sufficient size acts on a receptor the rapid depolarisation of the neurone occurs creating an action potential that travels down the neurone conveying information

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10
Q

Synaptic transmission

A

Action potential reaches the axon terminal of the pre-synaptic neurone, triggering the release of neurotransmitters from vesicles. These diffuse across the synapse binding with complementary receptors on the dendrites of the post-synaptic neurone and triggering an action potential along the axon. Any excess neurotransmitters in the synapse are reabsorbed into the pre-synaptic neurone through re-uptake channels

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11
Q

Neurotransmitters

A

Dopamine - excitatory, increases the positive charge of the neurone making it more likely to fire
Serotonin - inhibitory, decreases the charge in the neurone making it less likely to fire

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12
Q

Effect of cocaine on synaptic transmission

A

Cocaine blocks dopamine re-uptake channels, leading to an excess of dopamine in the synapse that will all eventually bind to receptors on the post-synaptic neurone. The dopamine reward pathway then becomes down regulated as it becomes used to that amount of dopamine and normal levels seem small, providing no pleasure.

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13
Q

Effect of heroin on synaptic transmission

A

Binds with specific opioid receptors at the synapse, massively enhancing our natural response. It is an agonist as it mimics the action of another biochemical found in the body. Long term effects lead to down regulation.

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14
Q

Effects of drugs on synaptic transmission - strengths

A

Weinshenker and Schroeder (2007)
Volkow et al (1997)
Application to better treatments for addiction - once heroin was identified as an agonist that bind to opioid receptors, specific drugs were created to reverse the mode of action

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15
Q

Weinshenker and Schroeder (2007)

A

Damaged the dopamine reward pathway in mice brains, leaving the neurones unable to produce the levels of dopamine usually associated with reward, when this occurred mice did not self-administer cocaine. Did not occur when the lesions were in other parts of the brain

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16
Q

Volkow et al (1997)

A

Used PET scans to track the activity of dopamine transporters during a cocaine induced high. Found that as subject experience intensified so did the number of cocaine occupied neurotransmitters

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17
Q

Effects of recreational drugs on synaptic transmission - weaknesses

A

Low validity of non-human studies, mice do not have a pre-frontal cortex, low generalisability to humans

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18
Q

Evolution through natural selection

A

Random mutation occurs which provides an advantage to an organism against a selection pressure. This enables them to live longer and therefore reproduce passing the advantageous characteristic down the generations. Over time this will reach the majority of the population

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19
Q

Sexual selection

A

An evolutionary explanation of partner preference. Attributes or behaviours that increase reproductive success are passed on. This may explain disadvantageous characteristics such as the peacocks large tail

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20
Q

Evolution and aggression

A

Males may be aggressive for two main evolutionary reasons: guarding your mate and guarding your offspring. This aggression enabled their offspring to survive and the characteristic was passed on.

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21
Q

Evolution and aggression - strengths

A

Explains gender differences - males are more likely to be selected when aggressive and females when cooperative to help protect their offspring. Therefore males are more aggressive than females
Application to reproductive behaviour - partners are chosen in order to enhance reproductive success (fertile women and men that could support a child)

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22
Q

Evolution and aggression - weaknesses

A

Impossible to test evolution directly - most research is correlational, meaning that we cannot draw cause and effect conclusions and external variables cannot be ruled out
Cannot explain cultural differences - Wolfgang and Ferracuti (1967)

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23
Q

Wolfgang and Ferracuti (1967)

A

The Kung San people of the Kalahari discourage aggression from childhood and it is therefore rare, whereas the Yanomami view aggression as an accepted behaviour to gain status and therefore is common

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24
Q

Freud - the unconscious

A

The part of the mind that we are unaware of but which continues to direct most of our behaviour. One of its key influences is in the origin of aggression

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25
Q

Freud - the id

A

The id exists from birth and is contained entirely in our unconscious mind. It contains instincts and impulses that are socially unacceptable including aggression. It obeys the pleasure principle, demanding immediate gratification of its desires with no consideration for other factors.

26
Q

Freud - the ego

A

The ego is the logical, rational and mostly conscious part of our personality that begins developing shortly after birth. It is the interface between the unrealistic desires of the id and social realities, not fulfilling the ids urges directly but indirectly or symbolically.

27
Q

Freud - the superego

A

Fulfils the moral role and develops at around 5-6 years old. It represents our conscience and moral sense of right and wrong. It opposes the ids aggressive drive through guilt and shame when we fail to meet our moral standards

28
Q

Freud - catharsis

A

The process of releasing pent up psychic energy, it reduces the aggressive drive and makes further aggression less likely. Preventing catharsis leads to built up energy that will eventually produce a destructive aggressive outburst.

29
Q

Freud - catharsis - displacement

A

An indirect method of catharsis where aggression towards one person is directed towards somebody less powerful and more available

30
Q

Freud - catharsis - observation

A

An indirect method of catharsis where aggression is observed, for example in a violent film

31
Q

Freud - strengths

A

Explains different types of aggression - can explain both ‘hot and cold blooded’ aggression, through the id and the ego
Application to reducing aggression - catharsis can be used to prevent the build up of destructive energy, people can build into their lives harmless ways of releasing aggression

32
Q

Freud - weaknesses

A

Aggression is not cathartic - Bushman (2002)

33
Q

Bushman (2002)

A

Made 600 college students angry by getting a confederate to criticise an essay each had written. One group were allowed to vent their anger by hitting a punchbag whilst thinking of the confederate, they subsequently blasted the confederate with the loudest and longest noises compared to controls that did not vent anger

34
Q

Role of hormones in the body

A

They are biochemical messengers that are transported around the body in the bloodstream, affecting cells with specific receptors

35
Q

Testosterone and aggression

A

Animal studies have demonstrated that experimental increases in testosterone are related to more aggressive behaviour, with the converse also true (Motelica Heino et al 1993). Also human evidence (Dolan et al 2001)

36
Q

Dolan et al (2001)

A

Found a positive correlation between testosterone levels and aggressive behaviours in a sample of 60 male offenders in UK maximum security hospitals.

37
Q

Motelica Heino et al (1993)

A

Castrated male rodents, finding that the castrated ones displayed less aggressive behaviours

38
Q

Dual hormone hypothesis

A

Hormonal causes of aggression are more complex than the action of a single hormone, cortisol may also have an affect (Carre and Mehta 2011)

39
Q

Carre and Mehta (2011)

A

Claim that high levels of testosterone lead to aggressive behaviour only when cortisol levels are low. When cortisol is high testosterones influence on aggression is blocked.

40
Q

Dual hormone hypothesis - strengths

A

Popma et al (2007)

41
Q

Popma et al (2007)

A

Showed that direct physical aggression was only shown in adolescent males when cortisol levels were low

42
Q

Hormones and aggression - strengths

A

Dabbs and Hargrove (1997)
Application to controlling aggression - if aggression is chemically controlled then it should be possible to manufacture a drug that reduces aggressive behaviour

43
Q

Dabbs and Hargrove (1997)

A

Measured testosterone in the saliva of 87 female inmates of a maximum security prison. Finding that the degree of criminal violence used correlated with testosterone

44
Q

Hormones and aggression - weaknesses

A

Studies are correlational - therefore we cannot establish a causal relationship between testosterone and aggression (aggression may lead to increased testosterone)
Measuring testosterone - (Carre et al 2011) psychologists distinguished between baseline and fluctuating levels of testosterone
Ungeneralizable - animals have a different brain structure to humans

45
Q

Carre et al (2011)

A

Argues that baseline testosterone levels play a less important role in human aggression than in animals. Therefore studies may not be valid as it is very hard to measure fluctuating levels unless it is constantly measured, as it changes from one social encounter to another

46
Q

Raine et al (1997) - aims

A

To use brain scanning technology to identify brain impairments in people charged with murder that pleaded not guilty by reasons of insanity (NGRI)

47
Q

Raine et al (1997) - method

A

41 ( 39 men and 2 women) murderers and 41 non-murderers
IV - murderer or non-murderer
Matched pairs design used
DV - glucose metabolism in specific brain areas

48
Q

Raine et al (1997) - procedure

A

Participants were free from medication int he two weeks leading up to their brain scan and were matched for on sex, age and ethnicity with a control group, controls had a physical and a psychiatric interview, none had a history of psychiatric illness apart from six with schizophrenia that were matched for it.
All injected with radioactive glucose tracker, then completed a 32 minute continuous performance task. The scan took ten images through the brain 10mm apart

49
Q

Raine et al (1997) - findings - cortical regions

A

Murderers had significantly lower glucose metabolism than controls in the lateral and medial prefrontal areas, left angular gyrus, and left and right superior parietal areas.
They showed higher metabolism in the occipital lobe

50
Q

Raine et al (1997) - findings - subcortical regions

A

Murderers had lower glucose metabolism than controls in the corpus callosum, left amygdala and left medial temporal lobe.
They had greater activity in the right amygdala, right medial temporal lobe and the right thalamus

51
Q

Raine et al (1997) - conclusions

A

Murderers pleading NGRI had a different brain structure compared to a control group, including impaired functioning in areas previously identified as being involved with violent behaviour. However, dysfunction in a single brain area cannot explain aggression .
We should be careful in interpreting the findings as there are areas of the brain known to be involved in violence that were not scanned

52
Q

Raine et al (1997) - strengths

A

High experimental control - participants were matched, standardised procedures were used, leading to high internal validity
Application to understanding biological evidence - need to be cautious about biological evidence, treatments may not be justified as it is only a partial explanation

53
Q

Raine et al (1997) - weaknesses

A

Lack of controls - participants were not randomly allocated to each group, therefore there may be some confounding variables (history of head injuries in 23 of the murderers)
Inappropriate scanning technique - the canthomeatal line, along which they scanned the participants, varies greatly in individuals. This would make it hard to locate different brain areas in the study

54
Q

Brendgen et al (2005) - aim

A

The extent to which social and physical aggression are explained by genetic and environmental influences
Whether the overlap between social and physical aggression is explained by the direct effect of one type of aggression on the other

55
Q

Brendgen et al (2005) - procedure

A

Used a twin study design, 234 pairs of twins (44 male Mz, 50 female Mz, 41 male Dz and 52 female Dz, as well as 67 mixed Dz that were not used in the final analysis) from the longitudinal Quebec new born twin study, this collected data from the twins regularly from 5-72 months.
Teacher ratings - rated the social and physical aggression of the children on a three point scale (sometimes, never, often) to items such as ‘says bad things’
Peer ratings - each child was given photos and asked to nominate three that best fit a description of a behaviour.

56
Q

Brendgen et al (2005) - findings

A

Only 20% (teacher ratings) and 23% (peer ratings) of social aggression was explained genetically
Physical aggression was mostly explained by genetic factors and social aggression by environmental factors
There was a significant but moderate correlation between physical and social aggression
Higher physical aggression led to higher social aggression but not the other way around

57
Q

Brendgen et al (2005) - conclusions

A

Genetic characteristics predispose some children to aggressive behaviour, however the specific form of that aggression is influenced by the environment

58
Q

Brendgen et al (2005) - strengths

A

Researchers used ratings of aggression from multiple sources, both very similar increasing validity and reliability
Application to reducing aggression - can lead to effective intervention to aggression by reducing physical aggression in early years so that it does not develop into social aggression

58
Q

Brendgen et al (2005) - strengths

A

Researchers used ratings of aggression from multiple sources, both very similar increasing validity and reliability
Application to reducing aggression - can lead to effective intervention to aggression by reducing physical aggression in early years so that it does not develop into social aggression

59
Q

Brendgen et al (2005) - weaknesses

A

Equal environment assumption - genetic influence is overestimated as Mz twins are more likely to be treated similarly than Dz twins, this reduces the validity of the study
Secondary data used - may have been collected unreliably