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Micro Final Exam > Bacterial Skin Infections > Flashcards

Bacterial Skin Infections Flashcards

1
Q

Organism that causes acne

A

Propionibacterium acnes

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2
Q

Open comedos

A

blackheads

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3
Q

Closed comedos

A

whiteheads

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4
Q

G (+) obligate anaerobic, nonmotile, pleomorphic rod. Common in normal flora of the sebaceous glands

A

P. acnes

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5
Q

Folliculitis can be caused by:

A

S. aureus (G+ cocci with coagulase)

P. aeruginosa (G- bacilla w/ VFs pyocyanin/veridn)

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6
Q

Hottub folliculitis is caused by

A

P. aeruginosa

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7
Q

Causes impetigo of Bockhart and Stys

A

S. aureus (G+ cocci w/ coagulase)

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8
Q

Impetigo of Bockhart

A

Beard area, erythema with papules or pustules

Rupture = yellow crust

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9
Q

Folliculitis of the eye

A

Sty

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10
Q

Clinical presentation of P. aeruginosa folliculitis

A

Systemic Sxs: fever, malaise, HA, sore throat, GI due to the LPS/exotixin A

(because it is G-, there is a systemic component)

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11
Q

Hot tub folliculitis is usually developed as a result of

A

poorly chlorinated water

Usu signs in areas occluded by swimwear

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12
Q

Enlarged folliculitis eruptions extended to dermis and SQ layers; Larger abscesses

A

Furnucles

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13
Q

Involves several follicles and extends to dermis and SQ layers

Massive inflammation

A

Carbuncles

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14
Q

Primary cause of non-bullous impetigo (pyoderma)

A

S. aureus

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15
Q

This is most frequently seen in preschool-aged kids, exposed areas of the body, and is increased in warm moist weather

A

Non-bullous impetigo

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16
Q

Non-bullous impetigo is easily transmitted by

A

fomites (towel) self-infecting–can spread to arms, legs, face

17
Q

Intradermal vesicles with exudate- forms thick amber colored adhesive crust

A

Non-bullous impetigo

18
Q

Bullous impetigo requires

A

strain to make exfoliation toxin

19
Q

Large superficial thin-walled blisters. They rupture and crust over and are very contagious! This is not a systemic infection

A

Bullous impetigo

Both the S. aureus and the exfoliation toxin are in the bullae

20
Q

Similar to staph scalded skin syndrome

A

Bullous impetigo

21
Q

Clinical presentation of cellulitis:

A

Heat
Erythema
Edema
Tenderness

22
Q

What should you avoid when treating cellulitis

A

NSAIDS

23
Q

Acute inflammation of SQ connective tissue. There are few microbes present, and it extends to the lower dermis

A

cellulitis

24
Q

The Sxs of cellulitis are due to

A

toxins and inflammatory response

25
Q

Cellulitis is usu caused by

A

S. pyogenes (G+)

26
Q

Does NF response to ABX?

A

NO

27
Q

Type I NF

A

mixed infection- aerobe and anaerobe (AGNR)

4-6 isolates

28
Q

Unexplained and increasing pain–then nerve damage = cutaenous anesthesia

A

NF

29
Q

Putrid odor is a/w which type of NF?

A

Type I

30
Q

Type 2 NF

A

‘flesh eating’

S. pyogenes

  • monomicrobic
  • exposure usu not clear
31
Q

Clinical presentation of NF

A 
Pain out of proportion to PE.
Hard wooden feel
Edema & erythema
Putrid odor (Type I)
Systemic toxicity
32
Q

DX of NF

A

surgical debridement

  • could gram stain tissue biopsies and exudate
  • MRI, CT, xray to determine extent
33
Q

Myonecrosis is caused by what organism

A

C. perfringens

34
Q

G+ bacillus spore former

A

C. perfringens

35
Q

Clinical presentation of myonecrosis

A

Sweet mousy smell

Sudden onset of pain, pale, bronze appearance, skin becomes tense, tender, crepitant (H2 gas), may develop bullae that are clear, red, blue, purple

Fever, tachy, altered mental status

36
Q

Dx Myonecrosis

A

tissue biopsy shows gram + rods with no PMNs and muscle necrosis

imaging for extent

37
Q

Tx of both NF and myonecrosis

A

HBO therapy–kills the anaerobes

Micro Final Exam (3 decks)

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