Attention Flashcards

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1
Q

What is the cocktail party problem?

A
  • In a room with many people but trying to focus on a conversion while a lot of people around you are talking
    How do we focus our attention on this conversation?
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2
Q

How do dichotic tasks explain the cocktail party problem?

A
  • Found that unattended auditory information is processed at a lower level than attended information
  • Only ⅓ of participants reported hearing their name in the unattended channel
  • Easier if voices are physically different
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3
Q

What is Broadbent’s theory of how attention is applied?

A
  • Attention as early selection
  • selective filter theory = people do not process unattended stimuli beyond the analysis of basic physical properties.

Sensory register - selective filter - short term memory

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4
Q

What is Deustch and Desutchs theory of how attention is applied?

A

-Selection happens later

  • After processing all the information you pick the stimulus that is most relevant to what you’re trying to do
  • Sensory register - short term memory - selection
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5
Q

What treismans theory of how attention is applied?

A
  • Attention as flexible selection
    Unattended information is weakened after the sensory register
  • Location of bottleneck is flexible because it can happen at an stage
    When we filter depends on the context
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6
Q

What is The posners cueing paradigm?

A

People can pay attention to a part of space they aren’t directly looking at (this is called “covert attention”)

Endogenous cues

Exogenous cues

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7
Q

What are Endogenous cues?

A

You choose to pay attention:
Choosing to pay attention to a particular space makes you react faster to things that happen in that space
- Top down

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8
Q

What are Exogenous cues?

A

It makes you pay attention
Same result as Endogenous cues
- but only if something in that space happens quickly after you shift your attention to it
- Bottom up

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9
Q

What is the feature integration theory?

A
  • Perceptual features are encoded in parallel and prior to attention
  • If an object has unique perceptual feature it may be detected without attention “pop out”
  • But if an object shares features it can’t be detected from a single perceptual feature and spatial attention is needed to search all candidates serially
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10
Q

What are illusionary conjunctions in the feature integration theory?

A

When you think you saw an object with certain features but that object wasn’t there only the features were.

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11
Q

When do illusionary conjunctions occur?

A
  • focused attention is absent
  • relevant stored knowledge is absent
  • spatial attention is diverted
  • display is presented in peripheral vision
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12
Q

Give a strength of the feature integration theory

A
  • explains what happens within the attentional spotlight
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13
Q

Give a weakness of feature integration theory

A

Does Not explain why the similarity of distractors is influential

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14
Q

What is the guided search/ dual path model?

A
  • There is two paths in which attention is allocated
  • In the real world when we search for things we typically have some expectations for where the things are.
  • Prior knowledge can make search more efficient
  • Bottom up & top down
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15
Q

What is visual search?

A
  • Searching cluttered visual environment for task relevant information is difficult when the target shares one or more features with other things in the image (distracters)
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16
Q

What are the two visual search paradigms?

A

Feature search
= Target has a unique feature that is not shared by other items in display (and therefore “pops out”)

Conjunction search
= Target has no unique feature that is not shared by other items in the display (making visual search difficult)

17
Q

What was the dual detection task in the attentional blink?

A
  • included trials in which they were asked firstly, “what letter was written in red” and secondly, “Whether you saw the letter X” (These detections were defined as T1 and T2)
18
Q

What was the single detection task in the attentional blink?

A

participants were asked to “ignore the red letters and just try to detect whether there is an X or not” (only T2).

19
Q

What was the stimulus material in the attentional blink?

A
  • 21 random letters, labelled as masks, presented on a screen at the rate of 10Hz
  • There were 20 trials divided for 10 in each task.
  • Half of each task were long lag and short lag
20
Q

What is hemispatial neglect?

A
  • A lack of awareness of stimuli presented to the opposite side of the brain damage
  • Its an impairment of attentions and conscious access rather than an impairment of perception
21
Q

What lesions underlie hemispatial neglect?

A

In the right of the brain
-Endogenous orienting system is relatively intact to neglect patients but not exogenous attention

22
Q

What is ADHD?

A

hyperactivity, impulsivity and inattention, risk taking
- Begins in childhood

23
Q

What are the 3 ADHD subytypes

A

Predominantly inattentive
= Difficulty in finishing task, following instructions easy distracted

Predominantly hyperactive/ impulsive
= Difficulty in sitting still for long periods; fidgeting; speaks or acts at inappropriate times

Combined inattentive and hyperactive/ impulsive
= Most common subtype

The DSM- V diagnostic criteria requires 6 symptoms from each (excluding the combined)

24
Q

What is the Aetiology of ADHD

A

Heritability
- Parents/ siblings of a child with ADHD are more likely to have it
- Prenatal exposure to alcohol and nicotine
- Premature birth and low birth weight
- Prenatal brain injury
- Environmental toxins

25
Q

What is the structural neural dysfunction of ADHD?

A
  • Reduced overall cortical and especially prefrontal volume
  • Reduced grey matter
  • Reduced cortical connectivity between hemispheres and within fronto- parietal attention networks
26
Q

What is the functional neural dysfunction of ADHD?

A

Hypoactivity in prefrontal cortex

27
Q

What is the molecular neural dysfunction of ADHD?

A
  • Imbalance in dopamine and noradrenaline circuits
  • Impairments to endogenous attention systems
28
Q

What is the treatment for ADHD?

A

Modulation of dopamine and norepinephrine systems