ARRYTHMIAS Flashcards

1
Q

What are the most common symptoms experienced by patients with arrythmias?

A
Often no symptoms at all
Palpatations
Light headedness
Syncope
Angina
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2
Q

What is a palpitation?

A

The sensation of ones own heartbeat

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3
Q

What is the mnemonic HIS DEBS and what does each later mean?

A

The causes or precipatating factors for arrythmias.

H - hypoxia (underlying pulmonary disorders)
I - ischemia and irritability
S - sympathetic stimulation

D - drugs
E - electrolyte disturbances
B - bradycardia (presdiposes to arrythmias)
S - stretch (of myocardium)

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4
Q

What is a Holter monitor?

A

A portable ECG machine used to assess arrythmic activity over prolonged periods such as 24 or 48 hours.

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5
Q

What is an event monitor?

A

This is a portable ECG machine which is initiated by the patient when they begin to experience symptoms. The rhythm strip then lasts for the next 3-5 minutes.

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6
Q

What are the five types of rhythm disturbance in the heart?

A
Arrythmias of sinus origin
Ectopic rhythms
Reentrant arrythmias
Conduction blocks
Preexcitation syndromes
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7
Q

What is sinus tachycardia?

A

If the rhythm of the heart speeds up beyond 100 beats per minute

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8
Q

What is sinus bradycardia?

A

If the rhythm of the heart slows down beyond 60 beats per minute

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9
Q

What conditions might be associated with sinus tachycardia?

A
Infections
Hyperthyroidism
Heart failure
Hypoxic related conditions (such as COPD and asthma)
Pain
Shock
Anemia
Pheochromocytoma
PE
Myocardial infarction
Kawasaki disease
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10
Q

What conditions might be associated with sinus bradycardia?

A
Early sign of myocardial infarction
Enhanced vagal tone
Hypothyroidism
Hypothermia
Intrinsic disease of the SA node (sick sinus syndrome)
Increased intracranial pressure
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11
Q

What is a sinus arrythmia?

A

When the ECG reveals a rhythm that is normal in all respects except that it is slightly irregular. It is most often caused by the difference in rate whilst breathing in compared with breathing out.

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12
Q

Why does heart rate increase during inspiration?

A

Negative pressure in the thorax during inspiration leads to decreased filling time and hence increased heart rate.

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13
Q

Why does heart rate decrease during expiration?

A

Positive pressure in the thorax from expiration leads to increased filling time and hence decreased heart rate.

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14
Q

What is an escape beat?

A

A wave of electrical activity that does not originate from the SA node. This might be because the SA has stopped firing.

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15
Q

When the SA node does not fire, which pacemaker most often takes over?

A

The junctional pacemaker cells around the AV node which fire at about 40-60 times per minute.

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16
Q

Which wave is missing from the ECG strip when the junctional pacemaker cells are setting the rhythm of the heart?

A

The p wave

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17
Q

What is a retrograde p wave?

A

When the electrical activity spreads back through the atria having been discharged from the junctional pacemaker cells. The axis of the wave is reversed by 180˚.

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18
Q

What is the theoretical difference between sinus arrest and sinus block?

A

Sinus arrest is a failure of the SA to fire.
Sinus block is a failure of the tissue surrounding the SA node to conduct the electrical activity.
Because the ECG leads cannot pick up SA node depolarization, these two pathologies will present in the same way.

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19
Q

What are ectopic rhythms?

A

Abnormal rhythms that arise from elsewhere in the heart than the sinus node. In this way, they resemble escape beats, but here we are talking about sustained rhythms, not just one or a few beats. They arise from enhanced automaticity of a nonsinus node site, either a single focus or a roving one.

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20
Q

What is the most common cause of ectopic rhythms?

A

Digitalis toxicity (foxglove)

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21
Q

Are re-entrant rhythms a disorder of impulse formation or impulse transmission?

A

Impulse transmission

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22
Q

Describe how a reentry loop works with regard to cardiac electrical impulses?

A

Ischemic or other myocardial damage can mean that two adjacent pathways through the myocardium conduct impulses at different speeds. This is not a problem normally as each impulse cancels each other out when they reach the same point in the loop, as they each find themselves in the refractory period of the other. The problem occurs when a second impulse that comes either prematurely or very soon after the first impulse. The fast conducting pathway will have a relatively long refractory period whereas the slow conducting route has a much faster refractory period. This means that the second impulse cannot go down the fast conducting pathway but is allowed down the slow one. As a result this second impulse that travels initially down the second pathway is not cancelled out by its equivalent impulse coming down the fast pathway and a reentry loop has been established. The impulse can now travel back up the heart in a retrograde fashion which severely limits the hearts capability to pump in unison.

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23
Q

What are the four questions used to assess any ECG strip?

A

Are P waves present and normal?
Are the QRS complexes narrow or wide?
What is the relationship between the P waves and the QRS complexes?
Is the rhythm regular or irregular?

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24
Q

What might you see on an ECG that would indicate an atrial premature beat?

A

A P wave with an abnormal contour.
The timing might also be irregular.
The PR interval may be decreased as well.

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25
Q

What might you see on an ECG that would indicate a junctional premature beat?

A

A narrow QRS complex that is not preceeded by a P wave at all or a retrograde (upside down) P wave.

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26
Q

What is the difference between a junctional premature beat and a junctional escape beat?

A

The junctional premature beat happens too early but the junctional escape happens too late as a result of a lack of atrial impulse.

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27
Q

What are the five main types of sustained supraventricular arrythmias?

A

Paroxysmal supraventricular tachycardia (PSVT)
Atrial flutter
Atrial fibrillation
Multifocal atrial tachycardia (MAT)
Paroxysmal (or ectopic) atrial tachycardia (PAT)

28
Q

Is there always underlying cardiac disease in someone whose heart goes into paroxysmal supraventricular tachycardia?

A

No. Alcohol, coffee and sheer excitement can cause a brief moment of PSVT.

29
Q

Is paroxysmal supraventricular tachycardia regular or irregular?

A

Absolutely regular rhythm

30
Q

What is the usual rate of a heart in paroxysmal supraventricular tachycardia?

A

150-250 beats per minute

31
Q

What is the most common type of paroxysmal supraventricular tachycardia?

A

AV nodal reentrant tachycardia (AVNRT)

32
Q

What might be seen in lead V1 on an ECG of someone in paroxysmal supraventricular tachycardia?

A

Pseudo R waves

33
Q

What is a pseudo R wave?

A

A little blip in the QRS complex that represents the superimposed retrograde P wave from a paroxysmal supraventricular tachycardia.

34
Q

What is a carotid massage and how might it be used therapeutically?

A

A temporary compression of the carotid artery that can be used to diagnose and terminate paroxysmal supraventricular tachycardia and atrial flutter.

35
Q

What is the theory behind why the carotid massage works as a treatment for paroxysmal supraventricular tachycardia?

A

Compression of the carotid leads to increase in blood pressure detected by the baroreceptors in the angle of the jaw. This leads to increase in vagal tone which decreases the rate at which the SA node fires and more importantly slow conduction through the AV node. This can interrupt the reentrant loop.

36
Q

How does one perform a carotid massage?

A
  1. Auscultate for carotid bruits. You do not want to cut off the last remaining trickle of blood to the brain nor dislodge an atherosclerotic plaque. If there is evidence of significant carotid disease, do not perform carotid massage.
  2. With the patient lying flat, extend the neck and rotate the head slightly away from you.
  3. Palpate the carotid artery at the angle of the jaw and apply gentle pressure for 10 to 15 seconds.
37
Q

Which carotid artery seems to be the best option for a carotid massge?

A

The right coratid

38
Q

Why must you always have a rhythm strip running whilst performing the carotid massage?

A

So that you can see what is happening to help diagnose. Also in rare instances, carotid massage may induce sinus arrest. Therefore always have equipment for resuscitation available.

39
Q

What is the underlying pathology of atrial flutter?

A

In its most common form, it is generated by a reentrant circuit that runs largely around the annulus of the tricuspid valve. Atrial depolarization occurs at such a rapid rate that discrete P waves seperated by a flat baseline are no longer seen.

40
Q

What is the most common AV block ratio with atrial flutter?

A

2:1 although 3:1 and 4:1 are also seen

41
Q

How does carotid massage affect atrial flutter?

A

It may increase the amount of block from 2:1 to 3:1 or 4:1 hence slowing the heart rate. It will not terminate the rhythm as the impulses are arising from above the AV node.

42
Q

What is the characteristic ECG changes seen in someone with atrial flutter?

A

Saw toothed pattern.

43
Q

What is the underlying pathology of atrial fibrillation?

A

The normal regular electrical impulses generated by the sinoatrial node in the right atrium of the heart are overwhelmed by disorganized electrical impulses. Multiple reentrant circuits are occurring in totally unpredictable fashion. This leads to irregular conduction of ventricular impulses that generate the heartbeat.

44
Q

Where do the disorganized electrical impulses in atrial fibrillation usually arise from?

A

The roots of the pulmonary vein

45
Q

What is seen on the ECG of someone in atrial fibrillation?

A

No true P waves - instead baseline appears flat or undulated slightly.
Irregularly irregular QRS complexes

46
Q

What are the risk factors and causes of atrial fibrillation?

A
Cardiac causes:
Hypertension
Coronary artery disease
Mitral stenosis
Mitral regurgitation
Tricuspid regurgitation
Hypertrophic cardiomyophathy
Degenerative conduction tissue
disease (sinoatrial node disease)
Myocarditis and pericarditis
Cardiac surgery
Non-cardiac causes:
Hyperthyroidism
Excessive alcohol consumption
Sepsis, esp. chest sepsis
Pulmonary embolism
Chest trauma, including direct trauma on cardiac catheterisation
47
Q

What are the risks associated with prolonged atrial fibrillation?

A

Thromboembolism - stroke/TIA

Increase in size of mitral valve annulus leading to mitral regurgitation

48
Q

Which group of atrial fibrillation patients would you be more likely to choose rhythm controlling medication than rate controlling medication?

A

Young patients or patients where the cause can be identified (such as a chest infection)

49
Q

What rate limiting drugs can be used for someone in atrial fibrillation?

A

Beta blockers

Rate-limiting calcium channel blockers such as verapamil or diltiazem

50
Q

Why might you decide to use digoxin to treat AF?

A

If they also have heart failure, and if they have a sedentary lifestyle (no exercise).

51
Q

What are the options for rhythm control of a patient with AF?

A

Electrical cardioversion

Amiodarone or Sotalol

52
Q

Why would you choose to electrically cardiovert someone in AF rather than pharmacologically?

A

If they have been in AF for 48 hours with no sign of medically intervention taking effect.

53
Q

When would you anti-coagulate someone in atrial fibrillation?

A

Use the CHADS2VASc score tool
Anyone with a score of 2 or above is given anti-coagulation
Consider anti-coagulation is men with a score of 1

54
Q

What are the different components of the CHADS2VASc score?

A
Congestive Heart Failure
Hypertension >140
Age over 65 = 1 point
Diabetes
Stroke history (including TIA) = 2 points
Vascular disease
Age over 75 = another point
Sexual category (female)
55
Q

What are the drugs most commonly used for anti-coagulation of someone in atrial fibrillation?

A
Warfarin
NOAC (dabigatran, rivaroxaban, or apixaban)
56
Q

What is the desired INR for someone diagnosed with AF?

A

2-3

57
Q

What is the HAASBLED score?

A

A tool for assessing likelihood of bleeding, used when deciding whether to anti-coagulate someone in AF.

58
Q

What are the components of the HAASBLED score?

A

Hypertension (uncontrolled, greater than 160 mmHg systolic)
Abnormal liver function
Abnormal renal function
Stroke (previous history, particularly lacunar)
Bleeding (bleeding history or predisposition)
Labile international normalized ratios (INRs, therapeutic time in range less than 60%)
Elderly (aged over 65 years)
Drugs (antiplatelet agents or nonsteroidal anti-inflammatory drugs)

59
Q

If anti-coagulation is not tolerated what other medication can be used to reduce the risk of stroke in AF patients?

A

Dual anti-platelet therapy

60
Q

What are the treatment options for someone in atrial flutter?

A

Atrial flutter is treated in much the same way as atrial fibrillation.
However, atrial flutter is much more sensitive to DC cardioversion and therefore much lower energies can be used.
Ablation of the cavo-tricuspid isthmus to disrupt the re-entrant circuit is also used as the definitive treatment.

61
Q

What are the treatment options for someone in AV nodal reentrant tachycardia?

A

Vagal maneuvres such as Valsava maneuvre

Drugs such as Adenosine, beta-blockers or rate-limiting calcium channel blockers (such as verapamil)

Flecainide and amiodarone may be used if these other drugs are not successful

Electrical cardioversion is used in very rare circumstances where tachycardia is not well tolerated (signs of heart failure or coma)

Catheter ablation of the slow pathway can also be used to treat AVNRT

62
Q

What are the three types of atrial fibrillation according to the Camm classification?

A

Paroxysmal AF
Persistent AF
Permanent AF

63
Q

How is paroxysmal AF defined according the Camm classification?

A

Self-terminating at least once

64
Q

How is persistent AF defined according the Camm classification?

A

Persists at least 48 hours without spontaneous

termination; can be converted to sinus rhythm with drugs or electricity

65
Q

How is permanent AF defined according the Camm classification?

A

Cannot be terminated with drugs or electricity

66
Q

What are the signs that someone might be in atrial fibrillation that one might pick up on examination of the patient?

A

Irregularly irregular pulse
Variable pulse volume
Single waveform JVP (subtle)
Apical-to-radial pulse deficit (if fast AF)
Variable intensity of first heart sound
Signs of complications or of underlying cause

67
Q

Is pulseless atrial fibrillation a shockable rhythm?

A

No