Arrythmias Flashcards

1
Q

What is the most common arrythmia

A

Atrial fibrillation

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2
Q

How can AF be classified

A

first detected episode
paroxysmal
persistent
permanent

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3
Q

What is meant by paroxysmal AF

A

when a patient has 2 or more episodes of AF
and if they terminate spontaneously
episodes last less than 7 days - usually <24hrs

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4
Q

What is meant by persistent AF

A

if 2 or more episodes of AF
not self-terminating
episodes last greater than 7 days

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5
Q

What is meant by permanent AF

A

continuous AF
which cannot be cardioverted

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6
Q

What may someone with AF present with

A

patients may be asymptomatic

palpitations
dyspnoea
chest pain
irregularly irregular pulse
breathlessness

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7
Q

What investigations are required

A

first line ECG
FBC

Echocardiogram

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8
Q

What is AF characterised by

A

very rapid and uncoordinated atrial activity leading to irregularly irregular rhythm

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9
Q

What is the epidemiology of AF

A

commonest cardiac rhythm disorder

ageing is massive risk factor

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10
Q

What are causes of atrial fibrillation

A

ATRIALE-PIBI

alcohol and caffeine
thyrotoxicosis
rheumatic fever and mitral valve disease

ischaemic heart disease
atrial myxoma
lungs
electrolyte disturbances
pharmacological
iatrogenic
blood pressure
infections

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11
Q

How do you manage AF if the patient is hemodynamically unstable

A

cardioversion

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12
Q

If patient with AF is hemodynamically stable the how do you manage them

A

rate or rhythm control

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13
Q

How you manage rate control of a patient with AF

A

Beta blocker

if doesnt work combination of any 2 :
beta blocker
digoxin
diltiazem (CCB)

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14
Q

How do you manage rhythm control in a a patient with AF

A

beta blocker
Amiodarone
Flecainide - if no structural heart disease

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15
Q

What drug should not be offered to patients with AF

A

aspirin monotherapy

  • for stroke prevention
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16
Q

What should patients with AF for less than 48 hours be treated with

A

anticoagulated with heparin

can be cardioverted electrically or pharmacologically
- using flecainide if no SHD or amiodarone if present

17
Q

How should patients with AF for more than 48 hours be treated

A

delay cardioversion until they have been maintained on therapeutic anticoagulation for minimum of 3 weeks

during this period offer rate control

18
Q

What should rate control therapy be offered as

A

as first line strategy
UNLESS
-reversible cause
-new onset AF
-heart failure is present
-rhythm control is more appropriate

19
Q

When is rhythm control for AF usually preferred

A

if patients have concomitant heart failure or in younger patients

20
Q

When may electrophysiological therapy be offered

A

radiofrequency ablation may be offered in intractable cases of AF that do not respond to medical therapy

21
Q

What is the CHA2DS2Vasc score

A

used to assess risk of stroke

C- history of congestive heart failure (1)
H- hypertension (1)
A-age 65-74(1), >75 (2)
D-diabetes
S-stroke, venous thromboembolism (2)
S- female ( 1)
Vasc- vascular disease(1)

22
Q

How do you interpret the CHA2D2Vasc score

A

score of 0 in men or 1 in women , recommend no anticoagulation

score of >1 in men , consider anticoagulation

score of >2 , offer anticoagulation

23
Q

What is the HAS-BLED score for

A

used to assess risk of bleeding if on therapy

H- hypertension SBP>160 (1)
A- abnormal liver/renal function (1 point each )
S- stroke history (1)
B-bleeding history /predisposition (1)
L- labile INR (1)
E-elderly >65
D -drugs ( NSAIDs, antiplatelets) OR alcohol (1)

24
Q

How do you interpret the HAS-BLED score

A

score =>3 does not preclude anticoagulation but caution is warranted , with regular review

25
What DOAC should be considered after CHA2DS2Vasc score has been calculated if needed
Apizaban , dabagatran, edoxaban or rivaroxaban warfarin if DOAC is contraindicated or not tolerated
26
What is supraventricular tachycardia
refers to paroxysmal episodes of tachycardia that are not ventricular in origin
27
What is the aetiology of supraventricular tachycardia
caused by re-entry mechanisms , or impulse initiation disorders producing automatic tachycardias
28
What is the difference between AVNRT AND AVRT and what type of tachycardias are they
Re-entrant tachycardias AVNRT - is caused by a non-anatomical pathway involving dual pathways with fast and slow conduction velocities AVRT- caused by an anatomically defined re-entrant circuit involving one or more accessory pathways
29
What are the 2 types of automatic tachycardias
Junctional tachycardia- secondary to abnormal impulses from junctional region Atrial tachycardia- tachycardia that is initiated and maintained irrespective of SA node, AV junction, accessory pathway or ventricular tissue
30
What is the acute management for SVT
first assess haemodynamic stability - if unstable arrange urgent direct cardioversion Vagal maneuvers- Valsalva - blow into empty plastic syringe carotid sinus massage IV ADENOSINE rapid IV bolus of 6mg - if unsuccessful go up to 12 , then 18
31
What drugs are given to a patient with SVT to prevent further episodes
beta-blockers RFA
32
What is ventricular tachycardia
tachyarrhythmia that originates from the ventricles- producing 3 or more successive broad QRS complexes at rate of greater than 100bpm
33
What is the aetiology of ventricular tachycardias
Ischemic heart disease structural heart disease electrolyte abnormalities medications - digoxin toxicity
34
What is the management for ventricular tachycardia
if patient is hemodynamically unstable - immediate resuscitation emergency direct cardioversion if patient is stable - Amiodarone lidocaine procainamide
35
What drug should not be used in ventricular tachycardia
verapamil
36
What drug to treat ventricular tachycardia should be used with caution
Lidocaine - if patient has severe left ventricular impairment
37
If drug therapy fails how should a patient with ventricular tachycardia be managed
electrophysiological study ICD- implantable cardioverter-defibrillator
38
What is torsades de pointes and how should it be managed
form of polymorphic VT associated with prolonged QT interval IV magnesium sulphate