Arrythmia Flashcards
AV block poem
R is far from P- first degree
Longer longerr longerrr drop- wenkebac
Some Ps dont get through- Mobitz 2
P and Q dont agree- third degree
QRS morphology in AV2
Mobitz 1= normal
Mobitz 2 = usually abnormal
Cause of mobitz 1
DIGOXIN
Cause of Mobitz 2
AVN ischemia
HR in 3°AV block
30-60
WIDE QRS COMPLEXES
AV dissociation
QRS and T in opposite direction
Total arrythmia types (14)
Sinus Bradycardia Sinus arrest 1AVB Mobitz1 Mobitz2 3AVB
Sinus tachycardia Unifocal atrial tachycardia Multifocal atrial tachycardia Atrial flutter Afib PSVT Vtach Vfib
Max conduction rate of AVN
250
Atrial ectopic cause and rate
Hypoxia in COPD
100-250
Atrial flutter presentation and speed
Palpitations
250-350
Afib cause
Stretching of atrial fibres
LAE
CMP
ASD
DOC stable Afib and further mx
Beta blockers
(If not allowed then CCB/Digoxin)
MOA: block avn further so as to stop arrythmia from reaching ventricles
F/b convert afib into sinus rhythm
By either 1) Ibutilide 2)DC shock
Maintenance of sinus rhythm DOC= AMIODARONE
AFib + WPW syndrome C/I and doc
C/I CCB/BB — accentuated bundle of k
DOc- PROCAINAMIDE IBUTILIDE AMIODARONE
Afib most risky factor
Thrombo-embolism
Therefore always do TEE BEFORE CARDIOVERSION
PSVT ecg characteristic
P waves superimposed by QRS
(So usually no P)
If P wave present: in reverse direction (CIRCUS WAVE) d/t retrograde stimulation through bundle
HR regular and fast
Frog sign is positive in
PSVT
PSVT Mx
Carotid massage—6mg adenosine—12mg adenosise—DC shock
Doc maintainence: VERAPAMIL
Doc prophylaxis : class 2 and 4 antiarr
V tach ecg characteristics
> 3 consecutive ectopics
HR: 100-250
Wide QRS
Qrs and T in opposite direction
Rx all Ventricular Tachycardia
Except Vtach<30s = AMIODARONE
IMMEDIATE DC shock 350J
DOC: AMIODARONE»LIGNOCAINE
TDP RX
MgSO4
Causes of 1AVB
Drugs (mc)
K+ / Mg++
Rheumatic Fever
Causes of 3AVB
Degenerative HD (mc)
Post sx
Congenital SLE
Pathology of tachyarrythmia
Defect in impulse production
(Enhanced automaticity of pacemaker cells-IHD/deg/electrolyte)
Defect in impulse propagation
a) Re-entry
b) After depolarisation
Differentiate Atrial and Ventricular tach
QRS normal. QRS wide
Carotid massage. -
- Variab H. Sounds
Doc AFib + HF
DIGOXIN
Only antiarrythmic with inotropic action
Common in elderly in ICU?
MAT
(Sepsis—-cytokines—pacemaker ++)
MAT NEVER CAUSES HYPOTENSION
M.c tachyarrythmia in ICU Pt.
Afib
Causes of AFib
Young: -Valvular HD
Holiday heart syndrome
DCMP
Old: Degenerative HD
Thyrotoxicosis
Capture beats and Fusion beats
Both give evidence of VTACH
CB- Normal beat in VTach (p followed by qrs) - that is impulse from atria has finally reached the ventricle, which was constantly getting depolarised by ectopics
FB- complexes formed due to fusion of atrial impulses and ventricular ectopics
Sustained VT timing
30 seconds
Reperfusion Arrythmia treatment
Atropine & lignocaine
(Aka AIVR- accelerated idioventricular rhythm)
D/t sudden reperfusion of blocked coronary
AF thrombus risk
(CHA2DS2 Vasc)
If > 2 increased risk
CHF HTn Age>75 (2points) 64-75= 1 point DM Stroke H/o (2 points) Sex: F Vascular D/o