Arrhythmias Flashcards

1
Q

Name the types of tachyarrhythmia.

A

Afib, SVPC, VT, VPC

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2
Q

Describe the pathogenesis of Afib

A

Atrial stretch (eg CHF, endocarditis, MVDV)

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3
Q

What ECG complexes are seen with ventricular disease?

A

Wide and bizarre QRS

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4
Q

What ECG complexes are seen with supraventricular disease?

A

Thin QRS with no P waves (superimposed on other complex)

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5
Q

What type of changes on an ECG may be seen with a lesion originating near the AV junction?

A

Negative P wave or no P wave, normal QRS complex

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6
Q

What changes would be seen on an ECG with a supraventricular tachycardia?

A

Superimposition of the P wave on the previous complex (t wave, st segment or QRS complex)

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7
Q

What drugs may be used in the treatment of Afib (minus those for primary disease)?

A

Dignoxin (negative chronotrope, vagomimetic - slows av conduction)
Diltiazem (negative inotrope)
Beta-blockers (negative inotrope)

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8
Q

In what situation would you NOT use beta blockers?

A

In cases of uncontrolled CHF

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9
Q

What changes on ECG may be seen with sinus arrhythmia?

A

Alternating periods of fast and slow rhythm (respiration), +/- irregular p wave confirguration

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10
Q

What is a sinus arrhythmia?

A

Irregular sinus rhythm originating in the SAN

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11
Q

What physiological factors may lead to increased vagal tone?

A

Vomiting, intubation, brachycephalic, elevated ICP, hypothermia, hypothyroidism

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12
Q

What events may lead to sinus tachycardia?

A

Stress, exercise, pain

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13
Q

What causes an SVPC?

A

Ectopic nodal tissue in the atria (other than SAN)

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14
Q

What is wolff-parkinson-white syndrome?

A

Accessory nervous pathway creation leading to supraventricular tachycardia

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15
Q

What treatment may be involved in an emergency SVT?

A

Vagal manoeuvres! Ca+ channel blockers - Diltiazem, Vermapil. Negative chronotropes - Digoxin. Beta-blockers - Esmolol

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16
Q

Why do junctional premature complexes generate negative P waves?

A

Retrograde spread of electrical signal to the atria

17
Q

What does a fusion complex suggest?

A

Paroxysmal ventricular tachycardia (none constant VT)

18
Q

Define ventricular tachycardia.

A

3 or more VPCs in a row

19
Q

What underlying causes lead to ventricular ectopic rhythms?

A

Catecholamine release, CHF, myocardial ischemia, acidosis, hypokalaemia, abdominal disease, thoracic trauma

20
Q

What is the treatment of choice for ventricular tachycardia?

A

IV Lidocaine. If persistent use beta blockers such as propranolol, amiodarone, soltalol etc

21
Q

Describe the emergency treatment of ventricular tachycardia.

A

IV access and lidocaine bolus, CRI amiodarone if Lido not affective. Within 24 hours oral mexiltine, propranolol, soltalol and amiodarone

22
Q

MOA amiodarone

A

Potassium channel blockers

23
Q

What mineral imbalance may lead to atrial standstill?

A

Hyperkalaemia

24
Q

Describe the changes associated with atrial standstill seen on ECG.

A

Absent P waves, spikey T waves, mildly prolonged QRS

25
Describe the pathogenesis of atrial standstill
Continued SAN conduction but inability of the atrial myocardium to contract
26
What causes an escape rhythm?
Slowing down or stopping of conduction through the SAN
27
What is sinus sick syndrome?
SAN pathology leading to long pauses between QRS complexes, junctional escape complexes and atrial fibrillation (AV junction disease)
28
What treatment is advised with sinus sick syndrome?
Pacemaker
29
Name four examples of bradyarrhythmias.
Sinus brady, sinus arrest, sinoventricular rhythm, AV block
30
Describe 1st degree AV block
Prolonged p-r interval
31
Describe 2nd degree AV block
P without a QRS every few complexes (mobitz 1) or P without a QRS every other (mobitz 2)
32
Describe 3rd degree AV block
No communication between P waves and QRS complex
33
What does an atropine test of a bradyarrhythmia suggest?
That the murmur is vagally mediated.